Matthias Löhn

4.0k total citations · 1 hit paper
43 papers, 3.3k citations indexed

About

Matthias Löhn is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cellular and Molecular Neuroscience. According to data from OpenAlex, Matthias Löhn has authored 43 papers receiving a total of 3.3k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Molecular Biology, 16 papers in Cardiology and Cardiovascular Medicine and 10 papers in Cellular and Molecular Neuroscience. Recurrent topics in Matthias Löhn's work include Ion channel regulation and function (12 papers), Cardiac electrophysiology and arrhythmias (7 papers) and Nitric Oxide and Endothelin Effects (5 papers). Matthias Löhn is often cited by papers focused on Ion channel regulation and function (12 papers), Cardiac electrophysiology and arrhythmias (7 papers) and Nitric Oxide and Endothelin Effects (5 papers). Matthias Löhn collaborates with scholars based in Germany, France and United States. Matthias Löhn's co-authors include Friedrich C. Luft, Maik Gollasch, Marlies Elger, Andreas Schedl, Marek Dráb, Teymuras V. Kurzchalia, Carsten Lindschau, Michael Kasper, Paul Verkade and Jan Menne and has published in prestigious journals such as Science, The Journal of Immunology and PLoS ONE.

In The Last Decade

Matthias Löhn

43 papers receiving 3.3k citations

Hit Papers

Loss of Caveolae, Vascular Dysfunction, and Pulmonary Def... 2001 2026 2009 2017 2001 400 800 1.2k

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Matthias Löhn Germany 21 1.7k 1.1k 1.1k 808 464 43 3.3k
Virginia J. Venema United States 32 2.9k 1.7× 829 0.7× 916 0.8× 1.5k 1.9× 263 0.6× 40 4.8k
Jérémy Fauconnier France 34 1.9k 1.1× 330 0.3× 1.0k 0.9× 462 0.6× 220 0.5× 72 2.9k
Minoru Seto Japan 27 1.5k 0.9× 328 0.3× 454 0.4× 666 0.8× 185 0.4× 53 2.8k
Changqing Xu China 36 1.6k 0.9× 251 0.2× 546 0.5× 675 0.8× 392 0.8× 115 3.2k
Antonella Liantonio Italy 31 2.1k 1.2× 421 0.4× 587 0.5× 664 0.8× 186 0.4× 101 2.7k
Catherine Pavoine France 29 1.5k 0.8× 217 0.2× 730 0.7× 527 0.7× 369 0.8× 58 2.9k
Robert G. Tsushima Canada 30 1.6k 0.9× 369 0.3× 607 0.6× 517 0.6× 875 1.9× 59 2.8k
Jun-ichi Kawabe Japan 25 1.9k 1.1× 536 0.5× 608 0.6× 378 0.5× 257 0.6× 68 2.8k
William Kutschke United States 23 1.8k 1.0× 215 0.2× 1.6k 1.5× 297 0.4× 237 0.5× 33 2.8k
Shuh Narumiya Japan 18 1.5k 0.9× 523 0.5× 231 0.2× 533 0.7× 239 0.5× 21 3.5k

Countries citing papers authored by Matthias Löhn

Since Specialization
Citations

This map shows the geographic impact of Matthias Löhn's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthias Löhn with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthias Löhn more than expected).

Fields of papers citing papers by Matthias Löhn

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthias Löhn. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthias Löhn. The network helps show where Matthias Löhn may publish in the future.

Co-authorship network of co-authors of Matthias Löhn

This figure shows the co-authorship network connecting the top 25 collaborators of Matthias Löhn. A scholar is included among the top collaborators of Matthias Löhn based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthias Löhn. Matthias Löhn is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Bielohuby, Maximilian, Kristin Breitschopf, Matthias Löhn, et al.. (2018). Acute and Repeated Treatment with 5-PAHSA or 9-PAHSA Isomers Does Not Improve Glucose Control in Mice. Cell Metabolism. 28(2). 217–227.e13. 50 indexed citations
4.
Löhn, Matthias. (2015). End-organ protection in hypertension by the novel and selective Rho-kinase inhibitor, SAR407899. World Journal of Cardiology. 7(1). 31–31. 9 indexed citations
5.
Bábelová, Andrea, Felix Jansen, Kerstin Sander, et al.. (2013). Activation of Rac-1 and RhoA Contributes to Podocyte Injury in Chronic Kidney Disease. PLoS ONE. 8(11). e80328–e80328. 73 indexed citations
6.
Grisk, Olaf, et al.. (2012). The Rho kinase inhibitor SAR407899 potently inhibits endothelin-1-induced constriction of renal resistance arteries. Journal of Hypertension. 30(5). 980–989. 25 indexed citations
7.
Schmidt, Thorsten, Matthias Löhn, Heinz‐Werner Kleemann, et al.. (2012). Inhibition of Diacylglycerol–Sensitive TRPC Channels by Synthetic and Natural Steroids. PLoS ONE. 7(4). e35393–e35393. 38 indexed citations
8.
Russell, James C., Spencer D. Proctor, Sandra Kelly, et al.. (2005). Insulin-Sensitizing and Cardiovascular Effects of the Sodium-Hydrogen Exchange Inhibitor, Cariporide, in the JCR. Journal of Cardiovascular Pharmacology. 46(6). 746–753. 11 indexed citations
9.
Rütten, Hartmut, et al.. (2005). Long term Rho-kinase inhibition ameliorates endothelial dysfunction in LDL-Receptor deficient mice. European Journal of Pharmacology. 512(2-3). 247–249. 9 indexed citations
10.
Löhn, Matthias, et al.. (2005). Inhibition of Rho-kinase stimulates nitric oxide-independent vasorelaxation. European Journal of Pharmacology. 507(1-3). 179–186. 31 indexed citations
11.
Schindler, Ursula, Hartmut Strobel, Karl Schönafinger, et al.. (2005). Biochemistry and Pharmacology of Novel Anthranilic Acid Derivatives Activating Heme-Oxidized Soluble Guanylyl Cyclase. Molecular Pharmacology. 69(4). 1260–1268. 83 indexed citations
12.
Schäfer, Stefan, et al.. (2003). Chronic vasopeptidase inhibition normalizes diabetic endothelial dysfunction. European Journal of Pharmacology. 484(2-3). 361–362. 18 indexed citations
13.
Löhn, Matthias, Christoph Mueller, & Jürgen Langner. (2002). Cell Cycle Retardation in Monocytoid Cells Induced by Aminopeptidase N (CD13). Leukemia & lymphoma. 43(2). 407–413. 14 indexed citations
14.
Löhn, Matthias, Manuela Klapperstück, Dagmar Riemann, & F Markwárdt. (2001). Sodium block and depolarization diminish P2Z-dependent Ca2+entry in human B lymphocytes. Cell Calcium. 29(6). 395–408. 11 indexed citations
15.
Gollasch, Maik, et al.. (2000). Ca2+ channels, ‘quantized’ Ca2+ release, and differentiation of myocytes in the cardiovascular system. Journal of Hypertension. 18(8). 989–998. 20 indexed citations
16.
Gollasch, Maik, et al.. (2000). Ca 2+ channels, Ca 2+ sparks, and regulation of arterial smooth muscle function. Zeitschrift für Kardiologie. 89(14). S015–S019. 28 indexed citations
17.
Markwárdt, F, et al.. (1999). Chapter 28 Purinoceptors in human B-lymphocytes. Progress in brain research. 120. 345–353. 7 indexed citations
18.
Löhn, Matthias, Christoph Mueller, K. Thiele, et al.. (1997). Aminopeptidase N-Mediated Signal Transduction and Inhibition of Proliferation of Human Myeloid Cells. Advances in experimental medicine and biology. 421. 85–91. 13 indexed citations
19.
Riemann, Dagmar, Astrid Kehlen, K. Thiele, Matthias Löhn, & Jürgen Langner. (1997). Co-Incubation of Lymphocytes with Fibroblast-Like Synoviocytes and other Cell Types Can Induce Lymphocytic Surface Expression of Aminopeptidase N/CD13. Advances in experimental medicine and biology. 421. 75–79. 4 indexed citations
20.
Riemann, Dagmar, Astrid Kehlen, Kathi Thiele, Matthias Löhn, & Jürgen Langner. (1997). Induction of aminopeptidase N/CD13 on human lymphocytes after adhesion to fibroblast-like synoviocytes, endothelial cells, epithelial cells, and monocytes/macrophages. The Journal of Immunology. 158(7). 3425–3432. 39 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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