Show‐Ling Shyng

8.4k total citations · 1 hit paper
87 papers, 6.3k citations indexed

About

Show‐Ling Shyng is a scholar working on Pathology and Forensic Medicine, Molecular Biology and Surgery. According to data from OpenAlex, Show‐Ling Shyng has authored 87 papers receiving a total of 6.3k indexed citations (citations by other indexed papers that have themselves been cited), including 56 papers in Pathology and Forensic Medicine, 39 papers in Molecular Biology and 29 papers in Surgery. Recurrent topics in Show‐Ling Shyng's work include Cardiac Ischemia and Reperfusion (56 papers), Ion channel regulation and function (28 papers) and Pancreatic function and diabetes (28 papers). Show‐Ling Shyng is often cited by papers focused on Cardiac Ischemia and Reperfusion (56 papers), Ion channel regulation and function (28 papers) and Pancreatic function and diabetes (28 papers). Show‐Ling Shyng collaborates with scholars based in United States, Egypt and Canada. Show‐Ling Shyng's co-authors include Colin G. Nichols, David A. Harris, Krista L. Moulder, Sylvain Lehmann, Charles A. Stanley, Benjamin Gläser, Arupa Ganguly, Courtney MacMullen, Mary T. Huber and Ann Nestorowicz and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Show‐Ling Shyng

84 papers receiving 6.2k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Sulfated Glycans Stimulate Endocytosis of the Cellular Isoform of the Prion Protein, PrPC, in Cultured Cells

1995 607 citations Show‐Ling Shyng, Krista L. Moulder et al. Journal of Biological Chemistry profile →

Peers

Countries citing papers authored by Show‐Ling Shyng

Since Specialization

Citations

This map shows the geographic impact of Show‐Ling Shyng's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Show‐Ling Shyng with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Show‐Ling Shyng more than expected).

Fields of papers citing papers by Show‐Ling Shyng

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Show‐Ling Shyng. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Show‐Ling Shyng. The network helps show where Show‐Ling Shyng may publish in the future.

Co-authorship network of co-authors of Show‐Ling Shyng

This figure shows the co-authorship network connecting the top 25 collaborators of Show‐Ling Shyng. A scholar is included among the top collaborators of Show‐Ling Shyng based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Show‐Ling Shyng. Show‐Ling Shyng is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Lipid probes to study ion channels 2025 ·Current Opinion in Chemical Biology ·Helene Jahn, Show‐Ling Shyng, Carsten Schultz	0
2	Dynamic duo: Kir6 and SUR in K _ATP channel structure and function 2024 ·Channels ·Bruce L. Patton, (unknown), (unknown), Camden Driggers, Show‐Ling Shyng	7
3	Non-radioactive Rb+ Efflux Assay for Screening KATP Channel Modulators 2024 ·Methods in molecular biology ·(unknown), Camden Driggers, Show‐Ling Shyng	1
4	Structure of an open KATP channel reveals tandem PIP2 binding sites mediating the Kir6.2 and SUR1 regulatory interface 2024 ·Nature Communications ·Camden Driggers, (unknown), (unknown), (unknown), Show‐Ling Shyng	15
5	KATP channel mutations in congenital hyperinsulinism: Progress and challenges towards mechanism-based therapies 2023 ·Frontiers in Endocrinology ·(unknown), Show‐Ling Shyng	7
6	KATP channels in focus: Progress toward a structural understanding of ligand regulation 2023 ·Current Opinion in Structural Biology ·Gregory M. Martin, Bruce L. Patton, Show‐Ling Shyng	13
7	KATP Channel Function: More than Meets the Eye 2022 ·Function ·Show‐Ling Shyng	6
8	Vascular K _ATP channel structural dynamics reveal regulatory mechanism by Mg-nucleotides 2021 ·Proceedings of the National Academy of Sciences ·Min Woo Sung, Zhongying Yang, Camden Driggers, Bruce L. Patton, Barmak Mostofian, (unknown), Daniel M. Zuckerman, Show‐Ling Shyng	32
9	AKAP79/150 coordinates leptin-induced PKA signaling to regulate KATP channel trafficking in pancreatic β-cells 2021 ·Journal of Biological Chemistry ·(unknown), Zhongying Yang, Mark L. Dell’Acqua, Show‐Ling Shyng	11
10	Production and purification of ATP-sensitive potassium channel particles for cryo-electron microscopy 2021 ·Methods in enzymology on CD-ROM/Methods in enzymology ·Camden Driggers, Show‐Ling Shyng	6
11	Ion Channels of the Islets in Type 2 Diabetes 2019 ·Journal of Molecular Biology ·David A. Jacobson, Show‐Ling Shyng	43
12	Concerted Trafficking Regulation of Kv2.1 and KATP Channels by Leptin in Pancreatic β-Cells 2015 ·Journal of Biological Chemistry ·Yi Wu, Show‐Ling Shyng, Pei‐Chun Chen	31
13	Leptin Regulates KATP Channel Trafficking in Pancreatic β-Cells by a Signaling Mechanism Involving AMP-activated Protein Kinase (AMPK) and cAMP-dependent Protein Kinase (PKA) 2013 ·Journal of Biological Chemistry ·Pei‐Chun Chen, Yelena Kryukova, Show‐Ling Shyng	67
14	ATP activates ATP-sensitive potassium channels composed of mutant sulfonylurea receptor 1 and Kir6.2 with diminished PIP2 sensitivity 2011 ·Channels ·Emily B. Pratt, Show‐Ling Shyng	2
15	Role of Hsp90 in Biogenesis of the β-Cell ATP-sensitive Potassium Channel Complex 2010 ·Molecular Biology of the Cell ·(unknown), Emily B. Pratt, Pei‐Chun Chen, Fang Wang, William R. Skach, Larry L. David, Show‐Ling Shyng	37
16	Characterization and Functional Restoration of a Potassium Channel Kir6.2 Pore Mutation Identified in Congenital Hyperinsulinism 2009 ·Journal of Biological Chemistry ·Jeremy D. Bushman, (unknown), Paul Tewson, Charles A. Stanley, Show‐Ling Shyng	4
17	Identification of a Familial Hyperinsulinism-causing Mutation in the Sulfonylurea Receptor 1 That Prevents Normal Trafficking and Function of KATP Channels 2002 ·Journal of Biological Chemistry ·Grit Taschenberger, (unknown), (unknown), Linda B. Lester, Stephen LaFranchi, Show‐Ling Shyng	99
18	Structural Determinants of Pip2 Regulation of Inward Rectifier KATP Channels 2000 ·The Journal of General Physiology ·Show‐Ling Shyng, Catherine A. Cukras, J. A. C. Harwood, Colin G. Nichols	169
19	ATP inhibition of K_ATP channels: control of nucleotide sensitivity by the N‐terminal domain of the Kir6.2 subunit 1999 ·The Journal of Physiology ·Joseph C. Koster, Qun Sha, Show‐Ling Shyng, Colin G. Nichols	82
20	The N-terminal Domain of a Glycolipid-anchored Prion Protein Is Essential for Its Endocytosis via Clathrin-coated Pits 1995 ·Journal of Biological Chemistry ·Show‐Ling Shyng, Krista L. Moulder, (unknown), David A. Harris	147

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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