Robert E. Cashon

1.3k citations
37 papers · 1.2k · h-index 19

Impact in

    • Hemoglobin structure and function
  • Physiology top 10%
    • Nitric Oxide and Endothelin Effects
    • Erythrocyte Function and Pathophysiology

Papers in

Robert E. Cashon

37 papers receiving 1.1k citations

Peers

Robert E. Cashon
Comparison fields: 5 of 103
  • Cell Biology 600
  • Physiology 350
  • Biophysics 61
  • Endocrine and Autonomic Systems 71
  • Genetics 98
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Citations per year

Countries citing papers authored by Robert E. Cashon

Since Specialization
Citations

This map shows the geographic impact of Robert E. Cashon's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert E. Cashon with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert E. Cashon more than expected).

Fields of papers citing papers by Robert E. Cashon

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert E. Cashon. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert E. Cashon. The network helps show where Robert E. Cashon may publish in the future.

Co-authors

The 25 scholars most cited alongside Robert E. Cashon, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Robert E. Cashon Line = papers co-authored together Robert E. Cashon links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown

Showing the 20 most-cited of 37 papers — load more, or switch the sort, to bring in the rest.

#Work
1 2001206
2 199974
3 199874
4 199369
5 199365
6 198065
7 199559
8 198659
9 199550
10 199743
11 199438
12 198133
13 199527
14 200426
15 200222
16 200020
17 200819
18 198619
19 199118
20 200317

About Robert E. Cashon

Robert E. Cashon is a scholar working on Cell Biology, Molecular Biology, Physiology, Pediatrics, Perinatology and Child Health and Genetics, having authored 37 papers that have together received 1.2k indexed citations. Recurring topics across this work include Hemoglobin structure and function (26 papers), Neonatal Health and Biochemistry (9 papers), Hemoglobinopathies and Related Disorders (8 papers), Heme Oxygenase-1 and Carbon Monoxide (7 papers), Erythrocyte Function and Pathophysiology (7 papers), Nitric Oxide and Endothelin Effects (5 papers), Physiological and biochemical adaptations (4 papers) and Protein Structure and Dynamics (4 papers). The work is most often cited by research in Cell Biology (600 citations), Physiology (350 citations), Biophysics (61 citations), Endocrine and Autonomic Systems (71 citations) and Genetics (98 citations). Robert E. Cashon has collaborated with scholars based in United States, Brazil and Slovakia. Frequent co-authors include Abdu I. Alayash, Rakesh P. Patel, Celia Bonaventura, Dennis A. Powers, Marius Brouwer, M. E. Vayda, Joseph Bonaventura, Rebecca J. Van Beneden, Bruce D. Sidell and Joseph C. Fratantoni. Their work appears in journals such as Journal of Biological Chemistry, Archives of Biochemistry and Biophysics, Comparative Biochemistry and Physiology Part B Biochemistry and Molecular Biology, Biochemical Genetics and Canadian Journal of Microbiology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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