Rishika Kundra

507 citations
5 papers · 364 · h-index 5

Impact in

Papers in

    • Ubiquitin and proteasome pathways 1
    • Mitochondrial Function and Pathology 1
    • Prion Diseases and Protein Misfolding 1
    • Bioinformatics and Genomic Networks 1
    • Endoplasmic Reticulum Stress and Disease 4

Rishika Kundra

5 papers receiving 364 citations

Peers

Rishika Kundra
Comparison fields: 5 of 57
  • Aging 23
  • Physiology 131
  • Cell Biology 87
  • Molecular Biology 267
  • Neurology 28
Replace Christof Hiebel with:
Christof Hiebel Germany
Rachel E. Lackie Canada
Kyoko Chiba Japan
Alexandria Ingram United States
YongTian Liang Germany
Roeland Vanhauwaert Belgium
Adriano Gonnelli Italy
Marcell Szabo United States
Lorena Fernández-Mosquera Germany
Wenjuan Wu Portugal
Rishika Kundra relative to Christof Hiebel Germany Christof Hiebel's profile →
Citations per field
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Christof Hiebel · 1×
Citations per year

Countries citing papers authored by Rishika Kundra

Since Specialization
Citations

This map shows the geographic impact of Rishika Kundra's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rishika Kundra with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rishika Kundra more than expected).

Fields of papers citing papers by Rishika Kundra

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rishika Kundra. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rishika Kundra. The network helps show where Rishika Kundra may publish in the future.

Co-authors

The 13 scholars most cited alongside Rishika Kundra, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Rishika Kundra Line = papers co-authored together Rishika Kundra links everyone, so they are left out of the graph.

All Works

About Rishika Kundra

Rishika Kundra is a scholar working on Molecular Biology, Cell Biology, Physiology, Cellular and Molecular Neuroscience and Spectroscopy, having authored 5 papers that have together received 364 indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (4 papers), Endoplasmic Reticulum Stress and Disease (4 papers), Advanced Proteomics Techniques and Applications (1 paper), Ubiquitin and proteasome pathways (1 paper), Mitochondrial Function and Pathology (1 paper), Prion Diseases and Protein Misfolding (1 paper), Bioinformatics and Genomic Networks (1 paper) and Genetic Neurodegenerative Diseases (1 paper). The work is most often cited by research in Aging (23 citations), Physiology (131 citations), Cell Biology (87 citations), Molecular Biology (267 citations) and Neurology (28 citations). Rishika Kundra has collaborated with scholars based in United Kingdom, United States and Poland. Frequent co-authors include Michele Vendruscolo, Christopher M. Dobson, Richard I. Morimoto, Prajwal Ciryam, Rosie Freer, Michele Perni, Tomasz Góral, Barbara Uszczyńska-Ratajczak, Agnieszka Chacińska and Urszula Nowicka. Their work appears in journals such as Proceedings of the National Academy of Sciences, Trends in Pharmacological Sciences, iScience and eLife.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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