Rebekka Mindnich
- Molecular Biology
- Endocrinology, Diabetes and Metabolism top 5%
- Genetics top 5%
- Cell Biology top 5%
- Physiology top 2%
- Co-authors
- Jerzy AdamskiT.M. PenningGabriele MöllerMartin Hrabě de AngelisDominga DelucaEdmund MaserYvonne KallbergStephen L. Dellaporta
- Topics
- Aldose Reductase and Taurine (11 papers)Hormonal Regulation and Hypertension (10 papers)Hormonal and reproductive studies (4 papers)
- Journals
- Journal of Biological ChemistryJournal of Molecular BiologyMolecular and Cellular Endocrinology
- Partner nations
- GermanyUnited StatesAustralia
In The Last Decade
Rebekka Mindnich
17 papers receiving 1.2k citations
Peers
Comparison fields: 5 of 99
- Molecular Biology 447
- Endocrinology, Diabetes and Metabolism 380
- Genetics 369
- Cell Biology 268
- Physiology 172
Countries citing papers authored by Rebekka Mindnich
This map shows the geographic impact of Rebekka Mindnich's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rebekka Mindnich with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rebekka Mindnich more than expected).
Fields of papers citing papers by Rebekka Mindnich
This network shows the impact of papers produced by Rebekka Mindnich. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rebekka Mindnich. The network helps show where Rebekka Mindnich may publish in the future.
Co-authorship network of co-authors of Rebekka Mindnich
This figure shows the co-authorship network connecting the top 25 collaborators of Rebekka Mindnich. A scholar is included among the top collaborators of Rebekka Mindnich based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rebekka Mindnich. Rebekka Mindnich is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 8 | |
| 2 | 50 | |
| 3 | 16 | |
| 4 | 43 | |
| 5 | 14 | |
| 6 | 38 | |
| 7 | 13 | |
| 8 | 127 | |
| 9 | 323 | |
| 10 | 50 | |
| 11 | 14 | |
| 12 | 8 | |
| 13 | 13 | |
| 14 | 4 | |
| 15 | 92 | |
| 16 | 297 | |
| 17 | 75 |
About Rebekka Mindnich
Rebekka Mindnich is a scholar working on Cell Biology, Endocrinology, Diabetes and Metabolism and Physiology, having authored 17 papers that have together received 1.2k indexed citations. Recurring topics across this work include Aldose Reductase and Taurine (11 papers), Hormonal Regulation and Hypertension (10 papers) and Hormonal and reproductive studies (4 papers). The work is most often cited by research in Physiology (172 citations), Endocrinology, Diabetes and Metabolism (380 citations) and Cell Biology (268 citations). Rebekka Mindnich has collaborated with scholars based in Germany, United States and Australia. Frequent co-authors include Jerzy Adamski, T.M. Penning, Gabriele Möller, Martin Hrabě de Angelis, Dominga Deluca, Edmund Maser, Yvonne Kallberg, Stephen L. Dellaporta, Angelo D. Favia and James E. Bray. Their work appears in journals such as Journal of Biological Chemistry, Journal of Molecular Biology and Molecular and Cellular Endocrinology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.