Rebeca Martínez‐Hernández
- Genetics top 2%
- Neurogenetic and Muscular Disorders Research 13
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- RNA modifications and cancer 9
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- T-cell and B-cell Immunology 4
- Immune Cell Function and Interaction 3
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- Congenital Anomalies and Fetal Surgery 8
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- MicroRNA in disease regulation 3
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- Peptidase Inhibition and Analysis 4
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- Neuroendocrine Tumor Research Advances 3
- Co-authors
- Sara BernalEduardo F. TizzanoLaura AlíasMónica MarazuelaAna Serrano-SomavillaEva Also‐RalloMiguel Sampedro‐NúñezAna M. Ramos‐Leví
- Cited by
- GeneticsMolecular BiologyImmunology
In The Last Decade
Rebeca Martínez‐Hernández
33 papers receiving 819 citations
Peers
Comparison fields: 5 of 70
- Genetics 464
- Molecular Biology 541
- Immunology 119
- Surgery 221
- Cancer Research 73
Countries citing papers authored by Rebeca Martínez‐Hernández
This map shows the geographic impact of Rebeca Martínez‐Hernández's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rebeca Martínez‐Hernández with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rebeca Martínez‐Hernández more than expected).
Fields of papers citing papers by Rebeca Martínez‐Hernández
This network shows the impact of papers produced by Rebeca Martínez‐Hernández. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rebeca Martínez‐Hernández. The network helps show where Rebeca Martínez‐Hernández may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Rebeca Martínez‐Hernández, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 1 | |
| 2 | 2024 | 4 | |
| 3 | 2024 | 6 | |
| 4 | 2024 | 1 | |
| 5 | 2024 | 7 | |
| 6 | 2023 | 4 | |
| 7 | 2023 | 14 | |
| 8 | 2020 | 17 | |
| 9 | 2020 | 9 | |
| 10 | 2018 | 43 | |
| 11 | 2017 | 57 | |
| 12 | 2015 | 27 | |
| 13 | 2014 | 30 | |
| 14 | 2012 | 67 | |
| 15 | 2011 | 27 | |
| 16 | 2011 | 25 | |
| 17 | 2011 | 9 | |
| 18 | 2010 | 13 | |
| 19 | 2009 | 76 | |
| 20 | 2008 | 131 |
About Rebeca Martínez‐Hernández
Rebeca Martínez‐Hernández is a scholar working on Genetics, Immunology and Molecular Biology, having authored 36 papers that have together received 827 indexed citations. Recurring topics across this work include Neurogenetic and Muscular Disorders Research (13 papers), RNA modifications and cancer (9 papers), Congenital Anomalies and Fetal Surgery (8 papers), Peptidase Inhibition and Analysis (4 papers), T-cell and B-cell Immunology (4 papers), Neuroendocrine Tumor Research Advances (3 papers), Immune Cell Function and Interaction (3 papers) and MicroRNA in disease regulation (3 papers). The work is most often cited by research in Genetics (464 citations), Molecular Biology (541 citations) and Immunology (119 citations). Rebeca Martínez‐Hernández has collaborated with scholars based in Spain, Mexico and Czechia. Frequent co-authors include Sara Bernal, Eduardo F. Tizzano, Laura Alías, Mónica Marazuela, Ana Serrano-Somavilla, Eva Also‐Rallo, Miguel Sampedro‐Núñez, Ana M. Ramos‐Leví, Roberto González‐Amaro and Montserrat Baiget. Their work appears in journals such as Nature Communications, The Journal of Clinical Endocrinology & Metabolism and Scientific Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.