Rainer Heuchel

7.9k total citations · 1 hit paper
78 papers, 6.4k citations indexed

About

Rainer Heuchel is a scholar working on Molecular Biology, Oncology and Surgery. According to data from OpenAlex, Rainer Heuchel has authored 78 papers receiving a total of 6.4k indexed citations (citations by other indexed papers that have themselves been cited), including 43 papers in Molecular Biology, 34 papers in Oncology and 12 papers in Surgery. Recurrent topics in Rainer Heuchel's work include Pancreatic and Hepatic Oncology Research (30 papers), TGF-β signaling in diseases (16 papers) and Cancer Cells and Metastasis (11 papers). Rainer Heuchel is often cited by papers focused on Pancreatic and Hepatic Oncology Research (30 papers), TGF-β signaling in diseases (16 papers) and Cancer Cells and Metastasis (11 papers). Rainer Heuchel collaborates with scholars based in Sweden, United States and Germany. Rainer Heuchel's co-authors include Carl‐Henrik Heldin, Peter ten Dijke, Nils‐Erik Heldin, Takuya Nakayama, Jan L. Christian, Mozhgan Afrakhte, Susumu Itoh, Atsuhito Nakao, Masahiro Kawabata and Freddy Radtke and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Rainer Heuchel

74 papers receiving 6.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Identification of Smad7, a TGFβ-inducible antagonist of TGF-β signalling

1997 1.6k citations Atsuhito Nakao, Mozhgan Afrakhte et al. Nature profile →

Peers

Countries citing papers authored by Rainer Heuchel

Since Specialization

Citations

This map shows the geographic impact of Rainer Heuchel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rainer Heuchel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rainer Heuchel more than expected).

Fields of papers citing papers by Rainer Heuchel

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rainer Heuchel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rainer Heuchel. The network helps show where Rainer Heuchel may publish in the future.

Co-authorship network of co-authors of Rainer Heuchel

This figure shows the co-authorship network connecting the top 25 collaborators of Rainer Heuchel. A scholar is included among the top collaborators of Rainer Heuchel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rainer Heuchel. Rainer Heuchel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	The Crosstalk Analysis between mPSCs and Panc1 Cells Identifies CCN1 as a Positive Regulator of Gemcitabine Sensitivity in Pancreatic Cancer Cells 2024 ·International Journal of Molecular Sciences ·(unknown), Xinyuan Liu, (unknown), (unknown), Ralf Weiskirchen, Matthias Löhr, Rainer Heuchel	2
2	FPR2 Shapes an Immune-Excluded Pancreatic Tumor Microenvironment and Drives T-cell Exhaustion in a Sex-Dependent Manner 2023 ·Cancer Research ·Fei He, (unknown), (unknown), (unknown), Sayaka Suzuki, Tianjie Liu, Qi Wang, Carlos Fernández Moro, (unknown), Peter Olofsson, Marit M. Melssen, Siu Kwan Sze, Mikael Björnstedt, Matthias Löhr, Mikael C. I. Karlsson, Rainer Heuchel, Dhifaf Sarhan	12
3	Stabilization of the classical phenotype upon integration of pancreatic cancer cells into the duodenal epithelium 2021 ·Neoplasia ·(unknown), Carlos Fernández Moro, Carina Strell, (unknown), Rainer Heuchel, Matthias Löhr, Ingemar Ernberg, László Székely, Marco Gerling, Béla Bozóky	2
4	3D heterospecies spheroids of pancreatic stroma and cancer cells demonstrate key phenotypes of pancreatic ductal adenocarcinoma 2021 ·Translational Oncology ·Xinyuan Liu, (unknown), Xidan Li, Jianping Liu, Anthony P. H. Wright, Matthias Löhr, (unknown), Rainer Heuchel	20
5	Role of c-MET Inhibitors in Overcoming Drug Resistance in Spheroid Models of Primary Human Pancreatic Cancer and Stellate Cells 2019 ·Cancers ·Omidreza Firuzi, Pei Pei, Btissame El Hassouni, Mark J. Buijs, Stefano Coppola, Matthias Löhr, Niccola Funel, Rainer Heuchel, Ilaria Carnevale, Thomas Schmidt, Giulia Mantini, Amir Avan, Luciano Saso, Godefridus J. Peters, Elisa Giovannetti	60
6	Human Cell Encapsulation in Gel Microbeads with Cosynthesized Concentric Nanoporous Solid Shells 2018 ·Advanced Functional Materials ·(unknown), Tommy Haraldsson, (unknown), Federico Ribet, (unknown), Rainer Heuchel, Matthias Löhr, Wouter van der Wijngaart	10
7	RCAN1 is a marker of oxidative stress, induced in acute pancreatitis 2018 ·Pancreatology ·(unknown), (unknown), Xuan Li, Hui Gao, Peter Szatmary, Ralf Segersvärd, Stephan Haas, (unknown), Urban Arnelo, Robert Sutton, Rainer Heuchel, Matthias Löhr	30
8	Pdx1-Cre-driven conditional gene depletion suggests PAK4 as dispensable for mouse pancreas development 2017 ·Scientific Reports ·Miao Zhao, (unknown), (unknown), Ting Zhuang, Audrey Minden, Matthias Löhr, Rainer Heuchel, Staffan Strömblad	4
9	Discrimination of pancreatic cancer and pancreatitis by LC-MS metabolomics 2017 ·Metabolomics ·Anna Lindahl, Rainer Heuchel, Jenny Forshed, Janne Lehtiö, Matthias Löhr, Anders Nordström	38
10	Cerulein-induced pancreatic fibrosis is modulated by Smad7, the major negative regulator of transforming growth factor-β signaling 2016 ·Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease ·Xuan Li, (unknown), (unknown), Carlos Fernández Moro, (unknown), Caroline S. Verbeke, Matthias Löhr, Rainer Heuchel	10
11	Fluorescence labeled microbubbles for multimodal imaging 2015 ·Biochemical and Biophysical Research Communications ·Åsa Barrefelt, Ying Zhao, (unknown), Gabriella Egri, Raoul Kuiper, (unknown), (unknown), Kenneth Caidahl, Torkel B. Brismar, Peter Aspelin, Rainer Heuchel, Mamoun Muhammed, Lars Dähne, Moustapha Hassan	28
12	Nodal/Activin Signaling Drives Self-Renewal and Tumorigenicity of Pancreatic Cancer Stem Cells and Provides a Target for Combined Drug Therapy 2012 ·Cell stem cell ·Enza Lonardo, Patrick Hermann, M Mueller, Stephan M. Huber, Anamaria Balic, Irene Miranda‐Lorenzo, Sladjana Zagorac, Sonia Alcalá, Iker Rodríguez-Arabaolaza, Juan Carlos Ramírez, Raúl Torres, Elena Cano García, (unknown), David Cebrián, Rainer Heuchel, Matthias Löhr, Frank Berger, Peter Bartenstein, Alexandra Aicher, Christopher Heeschen	15
13	Hypoxic Pulmonary Hypertension in Mice with Constitutively Active Platelet‐Derived Growth Factor Receptor‐β 2011 ·Pulmonary Circulation ·Bhola K. Dahal, Rainer Heuchel, Soni Savai Pullamsetti, Jochen Wilhelm, Hossein Ardeschir Ghofrani, Norbert Weißmann, Werner Seeger, Friedrich Grimminger, Ralph T. Schermuly	45
14	Disruption of the Smad7 gene enhances CCI₄‐dependent liver damage and fibrogenesis in mice 2008 ·Journal of Cellular and Molecular Medicine ·Jafar Hamzavi, Sabrina Ehnert, Patrício Godoy, Loredana Ciuclan, Honglei Weng, Peter R. Mertens, Rainer Heuchel, Steven Dooley	49
15	Disruption of the Smad7 gene promotes renal fibrosis and inflammation in unilateral ureteral obstruction (UUO) in mice 2008 ·Nephrology Dialysis Transplantation ·Arthur C.K. Chung, Xingguo Huang, Lin Zhou, Rainer Heuchel, Kar Neng Lai, Hui Y. Lan	159
16	Deletion of Exon I of SMAD7 in Mice Results in Altered B Cell Responses 2006 ·The Journal of Immunology ·Ronggui Li, Alexander Rosendahl, (unknown), Alec M. Cheng, Aive Åhgren, (unknown), Sarang Kulkarni, Tony Pawson, Carl‐Henrik Heldin, Rainer Heuchel	69
17	A Gain of Function Mutation in the Activation Loop of Plateletderived Growth Factor β-Receptor Deregulates Its Kinase Activity 2004 ·Journal of Biological Chemistry ·Federica Chiara, Marie‐José Goumans, Henrik Forsberg, Aive Åhgren, Andrea Rasola, Pontus Aspenström, Christer Wernstedt, Carina Hellberg, Carl‐Henrik Heldin, Rainer Heuchel	23
18	Transforming Growth Factor-β1 (TGF-β)–induced Apoptosis of Prostate Cancer Cells Involves Smad7-dependent Activation of p38 by TGF-β-activated Kinase 1 and Mitogen-activated Protein Kinase Kinase 3 2003 ·Molecular Biology of the Cell ·Sofia Edlund, Shizhong Bu, Norbert Schuster, Pontus Aspenström, Rainer Heuchel, Nils‐Erik Heldin, Peter ten Dijke, Carl‐Henrik Heldin, Maréne Landström	197
19	Retention of PDGFR-β function in mice in the absence of phosphatidylinositol 3′-kinase and phospholipase Cγ signaling pathways 2000 ·Genes & Development ·Michelle D. Tallquist, Richard A. Klinghoffer, Rainer Heuchel, (unknown), Philip Corrin, Carl‐Henrik Heldin, Richard J. Johnson, Philippe Soriano	63
20	Identification of Smad7, a TGFβ-inducible antagonist of TGF-β signalling breakdown → 1997 ·Nature ·Atsuhito Nakao, Mozhgan Afrakhte, (unknown), Takuya Nakayama, Jan L. Christian, Rainer Heuchel, Susumu Itoh, Masahiro Kawabata, Nils‐Erik Heldin, Carl‐Henrik Heldin, Peter ten Dijke	1557

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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