Rainer Heuchel

7.9k total citations · 1 hit paper
78 papers, 6.4k citations indexed

About

Rainer Heuchel is a scholar working on Molecular Biology, Oncology and Surgery. According to data from OpenAlex, Rainer Heuchel has authored 78 papers receiving a total of 6.4k indexed citations (citations by other indexed papers that have themselves been cited), including 43 papers in Molecular Biology, 34 papers in Oncology and 12 papers in Surgery. Recurrent topics in Rainer Heuchel's work include Pancreatic and Hepatic Oncology Research (30 papers), TGF-β signaling in diseases (16 papers) and Cancer Cells and Metastasis (11 papers). Rainer Heuchel is often cited by papers focused on Pancreatic and Hepatic Oncology Research (30 papers), TGF-β signaling in diseases (16 papers) and Cancer Cells and Metastasis (11 papers). Rainer Heuchel collaborates with scholars based in Sweden, United States and Germany. Rainer Heuchel's co-authors include Carl‐Henrik Heldin, Peter ten Dijke, Nils‐Erik Heldin, Takuya Nakayama, Jan L. Christian, Mozhgan Afrakhte, Susumu Itoh, Atsuhito Nakao, Masahiro Kawabata and Freddy Radtke and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Rainer Heuchel

74 papers receiving 6.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Identification of Smad7, a TGFβ-inducible antagonist of TGF-β signalling

1997 1.6k citations Atsuhito Nakao, Mozhgan Afrakhte et al. Nature profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Rainer Heuchel Sweden	35	3.6k	1.8k	983	801	660	78	6.4k
Naoyuki Miura Japan	42	3.8k 1.1×	2.9k 1.6×	905 0.9×	817 1.0×	477 0.7×	96	6.7k
Masayoshi Kobune Japan	45	2.9k 0.8×	1.4k 0.8×	448 0.5×	684 0.9×	104 0.2×	173	7.0k
Junji Kato Japan	38	1.7k 0.5×	967 0.5×	494 0.5×	416 0.5×	107 0.2×	197	5.5k
Jack L. Pinkus United States	35	1.6k 0.4×	1.0k 0.6×	1.4k 1.4×	299 0.4×	179 0.3×	79	6.9k
Ning Mao China	21	2.6k 0.7×	719 0.4×	224 0.2×	1.6k 2.0×	78 0.1×	85	6.0k
Herbert M. Kagan United States	49	5.3k 1.5×	408 0.2×	238 0.2×	737 0.9×	116 0.2×	126	8.0k
Lin Feng China	44	3.2k 0.9×	1.0k 0.6×	66 0.1×	1.1k 1.3×	463 0.7×	184	5.5k
Paavo Pääkkö Finland	47	2.5k 0.7×	1.8k 1.0×	147 0.1×	1.1k 1.4×	130 0.2×	148	6.2k
Alan O. Perantoni United States	38	2.8k 0.8×	573 0.3×	151 0.2×	467 0.6×	161 0.2×	91	4.0k
Feng Cao China	19	2.2k 0.6×	655 0.4×	207 0.2×	1.5k 1.9×	77 0.1×	69	4.1k

Countries citing papers authored by Rainer Heuchel

Since Specialization

Citations

This map shows the geographic impact of Rainer Heuchel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rainer Heuchel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rainer Heuchel more than expected).

Fields of papers citing papers by Rainer Heuchel

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rainer Heuchel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rainer Heuchel. The network helps show where Rainer Heuchel may publish in the future.

Co-authorship network of co-authors of Rainer Heuchel

This figure shows the co-authorship network connecting the top 25 collaborators of Rainer Heuchel. A scholar is included among the top collaborators of Rainer Heuchel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rainer Heuchel. Rainer Heuchel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Liu, Xinyuan, et al.. (2024). The Crosstalk Analysis between mPSCs and Panc1 Cells Identifies CCN1 as a Positive Regulator of Gemcitabine Sensitivity in Pancreatic Cancer Cells. International Journal of Molecular Sciences. 25(17). 9369–9369. 2 indexed citations

He, Fei, Sayaka Suzuki, Tianjie Liu, et al.. (2023). FPR2 Shapes an Immune-Excluded Pancreatic Tumor Microenvironment and Drives T-cell Exhaustion in a Sex-Dependent Manner. Cancer Research. 83(10). 1628–1645. 12 indexed citations

Moro, Carlos Fernández, Carina Strell, Rainer Heuchel, et al.. (2021). Stabilization of the classical phenotype upon integration of pancreatic cancer cells into the duodenal epithelium. Neoplasia. 23(12). 1300–1306. 2 indexed citations

Liu, Xinyuan, Xidan Li, Jianping Liu, et al.. (2021). 3D heterospecies spheroids of pancreatic stroma and cancer cells demonstrate key phenotypes of pancreatic ductal adenocarcinoma. Translational Oncology. 14(7). 101107–101107. 20 indexed citations

Firuzi, Omidreza, Pei Pei, Btissame El Hassouni, et al.. (2019). Role of c-MET Inhibitors in Overcoming Drug Resistance in Spheroid Models of Primary Human Pancreatic Cancer and Stellate Cells. Cancers. 11(5). 638–638. 60 indexed citations

Haraldsson, Tommy, et al.. (2018). Human Cell Encapsulation in Gel Microbeads with Cosynthesized Concentric Nanoporous Solid Shells. Advanced Functional Materials. 28(21). 10 indexed citations

Li, Xuan, Hui Gao, Peter Szatmary, et al.. (2018). RCAN1 is a marker of oxidative stress, induced in acute pancreatitis. Pancreatology. 18(7). 734–741. 30 indexed citations

Zhao, Miao, Ting Zhuang, Audrey Minden, et al.. (2017). Pdx1-Cre-driven conditional gene depletion suggests PAK4 as dispensable for mouse pancreas development. Scientific Reports. 7(1). 7031–7031. 4 indexed citations

Lindahl, Anna, Rainer Heuchel, Jenny Forshed, et al.. (2017). Discrimination of pancreatic cancer and pancreatitis by LC-MS metabolomics. Metabolomics. 13(5). 61–61. 38 indexed citations

10.

Li, Xuan, et al.. (2016). Cerulein-induced pancreatic fibrosis is modulated by Smad7, the major negative regulator of transforming growth factor-β signaling. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1862(9). 1839–1846. 10 indexed citations

11.

Barrefelt, Åsa, Ying Zhao, Gabriella Egri, et al.. (2015). Fluorescence labeled microbubbles for multimodal imaging. Biochemical and Biophysical Research Communications. 464(3). 737–742. 28 indexed citations

12.

Lonardo, Enza, Patrick Hermann, M Mueller, et al.. (2012). Nodal/Activin Signaling Drives Self-Renewal and Tumorigenicity of Pancreatic Cancer Stem Cells and Provides a Target for Combined Drug Therapy. Cell stem cell. 10(1). 104–104. 15 indexed citations

13.

Dahal, Bhola K., Rainer Heuchel, Soni Savai Pullamsetti, et al.. (2011). Hypoxic Pulmonary Hypertension in Mice with Constitutively Active Platelet‐Derived Growth Factor Receptor‐β. Pulmonary Circulation. 1(2). 259–268. 45 indexed citations

14.

Hamzavi, Jafar, Sabrina Ehnert, Patrício Godoy, et al.. (2008). Disruption of the Smad7 gene enhances CCI₄‐dependent liver damage and fibrogenesis in mice. Journal of Cellular and Molecular Medicine. 12(5b). 2130–2144. 49 indexed citations

15.

Chung, Arthur C.K., Xingguo Huang, Lin Zhou, et al.. (2008). Disruption of the Smad7 gene promotes renal fibrosis and inflammation in unilateral ureteral obstruction (UUO) in mice. Nephrology Dialysis Transplantation. 24(5). 1443–1454. 159 indexed citations

16.

Li, Ronggui, Alexander Rosendahl, Alec M. Cheng, et al.. (2006). Deletion of Exon I of SMAD7 in Mice Results in Altered B Cell Responses. The Journal of Immunology. 176(11). 6777–6784. 69 indexed citations

17.

Chiara, Federica, Marie‐José Goumans, Henrik Forsberg, et al.. (2004). A Gain of Function Mutation in the Activation Loop of Plateletderived Growth Factor β-Receptor Deregulates Its Kinase Activity. Journal of Biological Chemistry. 279(41). 42516–42527. 23 indexed citations

18.

Edlund, Sofia, Shizhong Bu, Norbert Schuster, et al.. (2003). Transforming Growth Factor-β1 (TGF-β)–induced Apoptosis of Prostate Cancer Cells Involves Smad7-dependent Activation of p38 by TGF-β-activated Kinase 1 and Mitogen-activated Protein Kinase Kinase 3. Molecular Biology of the Cell. 14(2). 529–544. 197 indexed citations

19.

Tallquist, Michelle D., Richard A. Klinghoffer, Rainer Heuchel, et al.. (2000). Retention of PDGFR-β function in mice in the absence of phosphatidylinositol 3′-kinase and phospholipase Cγ signaling pathways. Genes & Development. 14(24). 3179–3190. 63 indexed citations

20.

Nakao, Atsuhito, Mozhgan Afrakhte, Takuya Nakayama, et al.. (1997). Identification of Smad7, a TGFβ-inducible antagonist of TGF-β signalling. Nature. 389(6651). 631–635. 1557 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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