R. H. Stead

2.2k total citations · 1 hit paper
17 papers, 1.8k citations indexed

About

R. H. Stead is a scholar working on Physiology, Immunology and Molecular Biology. According to data from OpenAlex, R. H. Stead has authored 17 papers receiving a total of 1.8k indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Physiology, 5 papers in Immunology and 4 papers in Molecular Biology. Recurrent topics in R. H. Stead's work include Mast cells and histamine (4 papers), Pain Mechanisms and Treatments (3 papers) and Asthma and respiratory diseases (3 papers). R. H. Stead is often cited by papers focused on Mast cells and histamine (4 papers), Pain Mechanisms and Treatments (3 papers) and Asthma and respiratory diseases (3 papers). R. H. Stead collaborates with scholars based in Canada, Germany and United Kingdom. R. H. Stead's co-authors include Michael A. Rudnicki, Rudolf Jaenisch, Thomas Braun, Hans-Henning Arnold, Andrzej Stanisz, John Bienenstock, Kirk Hillsley, David Grundy, Jeroen Aerssens and Bernard Coulie and has published in prestigious journals such as Cell, The Journal of Immunology and Gastroenterology.

In The Last Decade

R. H. Stead

17 papers receiving 1.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

MyoD or Myf-5 is required for the formation of skeletal muscle

1993 1.4k citations Michael A. Rudnicki, R. H. Stead et al. Cell profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
R. H. Stead Canada	13	1.3k	264	248	232	193	17	1.8k
Josiane Fontaine-Pérus France	23	993 0.8×	222 0.8×	108 0.4×	347 1.5×	281 1.5×	48	1.4k
Arnold Munnich France	25	1.6k 1.3×	580 2.2×	96 0.4×	221 1.0×	341 1.8×	55	2.3k
Baljinder S. Mankoo United Kingdom	19	1.4k 1.1×	590 2.2×	80 0.3×	413 1.8×	283 1.5×	33	2.1k
Lixin Kan United States	29	1.3k 1.0×	562 2.1×	162 0.7×	375 1.6×	288 1.5×	44	2.6k
Tsuyoshi Takagi Japan	25	1.1k 0.8×	230 0.9×	174 0.7×	236 1.0×	170 0.9×	78	2.1k
Hiroki Kokubo Japan	26	1.7k 1.3×	397 1.5×	78 0.3×	229 1.0×	248 1.3×	46	2.2k
Barbara S. Mallon United States	23	1.3k 1.0×	194 0.7×	205 0.8×	279 1.2×	561 2.9×	38	2.3k
Junzo Desaki Japan	21	507 0.4×	173 0.7×	214 0.9×	133 0.6×	307 1.6×	65	1.4k
Maria‐Simonetta Faussone‐Pellegrini Italy	26	692 0.5×	146 0.6×	405 1.6×	722 3.1×	166 0.9×	43	2.0k
Emma Lou Cardell United States	10	1.4k 1.1×	290 1.1×	94 0.4×	202 0.9×	161 0.8×	15	1.9k

Countries citing papers authored by R. H. Stead

Since Specialization

Citations

This map shows the geographic impact of R. H. Stead's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by R. H. Stead with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites R. H. Stead more than expected).

Fields of papers citing papers by R. H. Stead

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by R. H. Stead. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by R. H. Stead. The network helps show where R. H. Stead may publish in the future.

Co-authorship network of co-authors of R. H. Stead

This figure shows the co-authorship network connecting the top 25 collaborators of R. H. Stead. A scholar is included among the top collaborators of R. H. Stead based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with R. H. Stead. R. H. Stead is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Stead, R. H., et al.. (2014). Comparison of Dako fluorescencein situhybridization assays (FISH and IQFISH) in the assessment of HER2 amplification in breast cancer. Journal of Histotechnology. 37(3). 90–94. 1 indexed citations

Grundy, David, Peter McLean, & R. H. Stead. (2007). Impact of 5‐HT3 receptor blockade on colonic haemodynamic responses to ischaemia and reperfusion in the rat. Neurogastroenterology & Motility. 19(7). 607–616. 12 indexed citations

Aerssens, Jeroen, Kirk Hillsley, Pieter J. Peeters, et al.. (2007). Alterations in the Brain–Gut Axis Underlying Visceral Chemosensitivity in Nippostrongylus brasiliensis-Infected Mice. Gastroenterology. 132(4). 1375–1387. 30 indexed citations

Hillsley, Kirk, Jiahui Lin, David Grundy, et al.. (2006). Dissecting the role of sodium currents in visceral sensory neurons in a model of chronic hyperexcitability using Na_v1.8 and Na_v1.9 null mice. The Journal of Physiology. 576(1). 257–267. 60 indexed citations

Stead, R. H., et al.. (2006). Vagal influences over mast cells. Autonomic Neuroscience. 125(1-2). 53–61. 44 indexed citations

Peeters, Pieter J., Jeroen Aerssens, Ronald De Hoogt, et al.. (2005). Molecular profiling of murine sensory neurons in the nodose and dorsal root ganglia labeled from the peritoneal cavity. Physiological Genomics. 24(3). 252–263. 45 indexed citations

Gottwald, Thomas, et al.. (1998). The mast cell‐nerve axis in wound healing: a hypothesis. Wound Repair and Regeneration. 6(1). 8–20. 28 indexed citations

Ratcliffe, Elyanne M., Derek J. deSa, Michael F. Dixon, & R. H. Stead. (1998). Choline Acetyltransferase (ChAT) Immunoreactivity in Paraffin Sections of Normal and Diseased Intestines. Journal of Histochemistry & Cytochemistry. 46(11). 1223–1231. 23 indexed citations

Ali, Shaikh Nisar, et al.. (1997). Mast cell Fc epsilonRI expression in the rat intestinal mucosa and tongue is enhanced during Nippostrongylus brasiliensis infection and can be up-regulated by in vivo administration of IgE. The Journal of Immunology. 158(8). 3805–3812. 33 indexed citations

10.

Kannan, Yukiko, et al.. (1996). Nerve growth factor and cytokines mediate lymphoid tissue-induced neurite outgrowth from mouse superior cervical ganglia in vitro. The Journal of Immunology. 157(1). 313–320. 44 indexed citations

11.

Kannan, Yukiko, R. H. Stead, C. H. Goldsmith, & John Bienenstock. (1994). Lymphoid tissues induce NGF‐dependent and NGF‐independent neurite outgrowth from rat superior cervical ganglia explants in culture. Journal of Neuroscience Research. 37(3). 374–383. 18 indexed citations

12.

Rudnicki, Michael A., et al.. (1993). MyoD or Myf-5 is required for the formation of skeletal muscle. Cell. 75(7). 1351–1359. 1369 indexed citations breakdown →

13.

Stead, R. H., et al.. (1992). Nerve remodeling during intestinal inflammation. Presentation at the Conference on Neuro-immuno-physiology of Gastrointestinal Mucosa.. NSUWorks (Nova Southeastern University). 1 indexed citations

14.

Stanisz, Andrzej, et al.. (1992). Expression of nerve growth factor receptor immunoreactivity on follicular dendritic cells from human mucosa associated lymphoid tissues.. PubMed. 76(3). 485–90. 28 indexed citations

15.

Bienenstock, John, JA Denburg, R Scicchitano, et al.. (1988). Role of neuropeptides, nerves and mast cells in intestinal immunity and physiology.. PubMed. 24. 134–43. 18 indexed citations

16.

Swieter, Mark, et al.. (1987). Mast Cell Pleomorphism: Properties of Intestinal Mast Cells. Advances in experimental medicine and biology. 216A. 613–623. 10 indexed citations

17.

Bundza, A. & R. H. Stead. (1986). Medullary Thyroid Carcinoma in Two Cows. Veterinary Pathology. 23(1). 90–92. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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