Hans-Henning Arnold

8.0k total citations · 2 hit papers
67 papers, 6.3k citations indexed

About

Hans-Henning Arnold is a scholar working on Molecular Biology, Genetics and Genetics. According to data from OpenAlex, Hans-Henning Arnold has authored 67 papers receiving a total of 6.3k indexed citations (citations by other indexed papers that have themselves been cited), including 64 papers in Molecular Biology, 19 papers in Genetics and 4 papers in Genetics. Recurrent topics in Hans-Henning Arnold's work include Developmental Biology and Gene Regulation (29 papers), Congenital heart defects research (28 papers) and Muscle Physiology and Disorders (23 papers). Hans-Henning Arnold is often cited by papers focused on Developmental Biology and Gene Regulation (29 papers), Congenital heart defects research (28 papers) and Muscle Physiology and Disorders (23 papers). Hans-Henning Arnold collaborates with scholars based in Germany, United States and United Kingdom. Hans-Henning Arnold's co-authors include Thomas Braun, Michael A. Rudnicki, Rudolf Jaenisch, Eva Bober, R. H. Stead, Thomas Brand, Oliver Pabst, Birgit Andrée, Thomas Floß and Astrid Buchberger and has published in prestigious journals such as Cell, Nucleic Acids Research and Journal of Biological Chemistry.

In The Last Decade

Hans-Henning Arnold

67 papers receiving 6.2k citations

Hit Papers

MyoD or Myf-5 is required for the formation of skeletal m... 1992 2026 2003 2014 1993 1992 400 800 1.2k

Peers

Hans-Henning Arnold
Eva Bober Germany
Tom Strachan United Kingdom
Peter Cserjesi United States
Lauren Snider United States
Kenro Kusumi United States
Eva Bober Germany
Hans-Henning Arnold
Citations per year, relative to Hans-Henning Arnold Hans-Henning Arnold (= 1×) peers Eva Bober

Countries citing papers authored by Hans-Henning Arnold

Since Specialization
Citations

This map shows the geographic impact of Hans-Henning Arnold's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Hans-Henning Arnold with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Hans-Henning Arnold more than expected).

Fields of papers citing papers by Hans-Henning Arnold

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Hans-Henning Arnold. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Hans-Henning Arnold. The network helps show where Hans-Henning Arnold may publish in the future.

Co-authorship network of co-authors of Hans-Henning Arnold

This figure shows the co-authorship network connecting the top 25 collaborators of Hans-Henning Arnold. A scholar is included among the top collaborators of Hans-Henning Arnold based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Hans-Henning Arnold. Hans-Henning Arnold is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Papalazarou, Vassilis, Karthic Swaminathan, Farah Jaber‐Hijazi, et al.. (2020). The Arp2/3 complex is critical for colonisation of the mouse skin by melanoblasts. Development. 147(22). 10 indexed citations
2.
Goel, Aviva, et al.. (2017). Niche Cadherins Control the Quiescence-to-Activation Transition in Muscle Stem Cells. Cell Reports. 21(8). 2236–2250. 95 indexed citations
3.
Vauti, Franz, et al.. (2015). Generation of a Nkx2.2Cre knock-in mouse line: Analysis of cell lineages in the central nervous system. Differentiation. 89(3-4). 70–76. 5 indexed citations
4.
Thalheimer, Frederic B., Katharina Gerlach, Stefanie Böhm, et al.. (2015). Single-Stranded DNA-Binding Transcriptional Regulator FUBP1 Is Essential for Fetal and Adult Hematopoietic Stem Cell Self-Renewal. Cell Reports. 11(12). 1847–1855. 30 indexed citations
5.
Vauti, Franz, et al.. (2007). Arp3 is required during preimplantation development of the mouse embryo. FEBS Letters. 581(29). 5691–5697. 25 indexed citations
6.
Schlange, Thomas, Birgit Andrée, Andreas D. Ebert, et al.. (2001). Chick CFC Controls Lefty1 Expression in the Embryonic Midline and Nodal Expression in the Lateral Plate. Developmental Biology. 234(2). 376–389. 35 indexed citations
7.
Schlange, Thomas, Birgit Andrée, Hans-Henning Arnold, & Thomas Brand. (2000). BMP2 is required for early heart development during a distinct time period. Mechanisms of Development. 91(1-2). 259–270. 160 indexed citations
8.
9.
Andrée, Birgit, Tina Hillemann, Gania Kessler‐Icekson, et al.. (2000). Isolation and Characterization of the Novel Popeye Gene Family Expressed in Skeletal Muscle and Heart. Developmental Biology. 223(2). 371–382. 98 indexed citations
10.
Buchberger, Astrid, et al.. (2000). Expression of the novel basic-helix-loop-helix transcription factor cMespo in presomitic mesoderm of chicken embryos. Mechanisms of Development. 97(1-2). 223–226. 17 indexed citations
11.
Buchberger, Astrid & Hans-Henning Arnold. (1999). The MADS domain containing transcription factor cMef2a is expressed in heart and skeletal muscle during embryonic chick development. Development Genes and Evolution. 209(6). 376–381. 13 indexed citations
12.
Schneider, André, et al.. (1999). The homeobox gene it NKX3.2 is a target of left–right signalling and is expressed on opposite sides in chick and mouse embryos. Current Biology. 9(16). 911–S1. 79 indexed citations
13.
Buchberger, Astrid, et al.. (1998). cMeso-1, a Novel bHLH Transcription Factor, Is Involved in Somite Formation in Chicken Embryos. Developmental Biology. 199(2). 201–215. 57 indexed citations
14.
Pabst, Oliver, Heike Herbrand, & Hans-Henning Arnold. (1998). Nkx2-9 is a novel homeobox transcription factor which demarcates ventral domains in the developing mouse CNS. Mechanisms of Development. 73(1). 85–93. 63 indexed citations
15.
Floß, Thomas, Hans-Henning Arnold, & Thomas Braun. (1997). A role for FGF-6 in skeletal muscle regeneration. Genes & Development. 11(16). 2040–2051. 258 indexed citations
16.
Arnold, Hans-Henning & Thomas Braun. (1996). Targeted inactivation of myogenic factor genes reveals their role during mouse myogenesis: a review. The International Journal of Developmental Biology. 40(1). 345–353. 97 indexed citations
17.
Arnold, Hans-Henning, et al.. (1996). Regulation and Function of SF/HGF during Migration of Limb Muscle Precursor Cells in Chicken. Developmental Biology. 180(2). 566–578. 76 indexed citations
18.
Buchberger, Astrid, Oliver Pabst, Thomas Brand, Klaus Seidl, & Hans-Henning Arnold. (1996). Chick NKx-2.3 represents a novel family member of vertebrate homologues to the Drosophila homeo☐ gene tinman: differential expression of cNKx-2.3 and cNKx-2.5 during heart and gut development. Mechanisms of Development. 56(1-2). 151–163. 69 indexed citations
19.
Floß, Thomas, Hans-Henning Arnold, & Thomas Braun. (1996). Myf-5m1/Myf-6m1Compound Heterozygous Mouse Mutants Down-regulate Myf-5 Expression and Exert Rib Defects: Evidence for Long-RangecisEffects on Myf-5 Transcription. Developmental Biology. 174(1). 140–147. 21 indexed citations
20.
Braun, Thomas, Michael A. Rudnicki, Hans-Henning Arnold, & Rudolf Jaenisch. (1992). Targeted inactivation of the muscle regulatory gene Myf-5 results in abnormal rib development and perinatal death. Cell. 71(3). 369–382. 587 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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