Qiu‐Lan Ma

2.9k citations
26 papers · 2.3k indexed · h-index 19
Topics
Alzheimer's disease research and treatments (19 papers)Nuclear Receptors and Signaling (7 papers)Fatty Acid Research and Health (5 papers)
Partner nations
United StatesChinaJapan

In The Last Decade

Qiu‐Lan Ma

26 papers receiving 2.2k citations

Peers

Qiu‐Lan Ma
Comparison fields: 5 of 105
  • Physiology 1.2k
  • Molecular Biology 728
  • Cellular and Molecular Neuroscience 580
  • Pharmacology 310
  • Neurology 304
Replace Daijiro Yanagisawa with:
Daijiro Yanagisawa Japan
Panchanan Maiti United States
Changiz Taghibiglou Canada
Balmiki Ray United States
Karen H. Gylys United States
Erika Gyengési Australia
Rui‐tian Liu China
Surendra S. Ambegaokar United States
Yasuhiko Izumi Japan
Maria‐Grazia Martinoli Canada
Qiu‐Lan Ma relative to Daijiro Yanagisawa Japan Daijiro Yanagisawa's profile →
Citations per field
00.5×1.7×
Daijiro Yanagisawa · 1×
Citations per year

Countries citing papers authored by Qiu‐Lan Ma

Since Specialization
Citations

This map shows the geographic impact of Qiu‐Lan Ma's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Qiu‐Lan Ma with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Qiu‐Lan Ma more than expected).

Fields of papers citing papers by Qiu‐Lan Ma

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Qiu‐Lan Ma. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Qiu‐Lan Ma. The network helps show where Qiu‐Lan Ma may publish in the future.

Co-authorship network of co-authors of Qiu‐Lan Ma

This figure shows the co-authorship network connecting the top 25 collaborators of Qiu‐Lan Ma. A scholar is included among the top collaborators of Qiu‐Lan Ma based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Qiu‐Lan Ma. Qiu‐Lan Ma is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
#WorkIndexed citations
1 7
2 29
3 26
4 2
5 106
6 159
7 65
8 15
9 65
10 179
11 70
12 102
13 139
14 86
15 96
16 269
17 113
18 173
19 13
20 60

About Qiu‐Lan Ma

Qiu‐Lan Ma is a scholar working on Molecular Medicine, Physiology and Cellular and Molecular Neuroscience, having authored 26 papers that have together received 2.3k indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (19 papers), Nuclear Receptors and Signaling (7 papers) and Fatty Acid Research and Health (5 papers). The work is most often cited by research in Physiology (1.2k citations), Molecular Medicine (204 citations) and Neurology (287 citations). Qiu‐Lan Ma has collaborated with scholars based in United States, China and Japan. Frequent co-authors include Sally A. Frautschy, Greg M. Cole, Fusheng Yang, Oliver J. Ubeda, Bruce Teter, Giselle P. Lim, Marni E. Harris‐White, Shuxin Hu, Xiaohong Zuo and Mychica Jones. Their work appears in journals such as Journal of Biological Chemistry, Journal of Neuroscience and Nature Neuroscience.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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