Qin Yang

3.0k total citations
70 papers, 1.7k citations indexed

About

Qin Yang is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Qin Yang has authored 70 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 38 papers in Molecular Biology, 17 papers in Oncology and 15 papers in Cancer Research. Recurrent topics in Qin Yang's work include RNA modifications and cancer (10 papers), Cancer Genomics and Diagnostics (5 papers) and Cancer-related molecular mechanisms research (5 papers). Qin Yang is often cited by papers focused on RNA modifications and cancer (10 papers), Cancer Genomics and Diagnostics (5 papers) and Cancer-related molecular mechanisms research (5 papers). Qin Yang collaborates with scholars based in China, United States and South Korea. Qin Yang's co-authors include Zhigang Zhang, Jun Li, Yahui Wang, Shu‐Heng Jiang, Li-Peng Hu, Yanli Zhang, Qing Li, Lei Zhu, Xueli Zhang and Hui Nie and has published in prestigious journals such as Cell, Nucleic Acids Research and Nature Communications.

In The Last Decade

Qin Yang

68 papers receiving 1.7k citations

Peers

Qin Yang

ONCOL

IMMUN

PRM

Miguel Reina‐Campos United States

Zhiyong Ding China

Amy Lam Hong Kong

M. Kay Washington United States

Ying‐Hui Zhu China

Roble Bedolla United States

Kouhei Yamamoto Japan

Hengjun Gao China

Huamei Tang China

Miguel Reina‐Campos United States

Qin Yang

1.2k ×1.4

672 ×1.3

628 ×1.6

ONCOL

550 ×1.7

IMMUN

272 ×0.9

PRM

Citations per year, relative to Qin Yang Qin Yang (= 1×) peers Miguel Reina‐Campos

Countries citing papers authored by Qin Yang

Since Specialization

Citations

This map shows the geographic impact of Qin Yang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Qin Yang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Qin Yang more than expected).

Fields of papers citing papers by Qin Yang

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Qin Yang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Qin Yang. The network helps show where Qin Yang may publish in the future.

Co-authorship network of co-authors of Qin Yang

This figure shows the co-authorship network connecting the top 25 collaborators of Qin Yang. A scholar is included among the top collaborators of Qin Yang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Qin Yang. Qin Yang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Li, Nannan, Qin Yang, Fuqiang Li, et al.. (2025). Identification and Evaluation of the Urinary Microbiota Associated With Bladder Cancer. PubMed. 4(4). e70012–e70012. 2 indexed citations

Wang, Hongyu, et al.. (2025). 2.5D Numerical Formulation for Analysing Long‐Term Settlement of Tunnel‐Soil System Induced by Cyclic Train Loading in Soft Soil Area. International Journal for Numerical and Analytical Methods in Geomechanics. 49(8). 1986–2002.

Wang, Jibing, et al.. (2024). Effects of nano-Rhodiola rosea combined with treadmill exercise on anti-exercise fatigue in rats. Frontiers in Nutrition. 11. 1446944–1446944. 5 indexed citations

Wang, Hui, Guolei Wang, Li Zou, et al.. (2024). Fused in sarcoma (FUS) inhibits milk production efficiency in mammals. Nature Communications. 15(1). 3953–3953.

Wang, Yangyang, et al.. (2021). Norepinephrine Enhances Aerobic Glycolysis and May Act as a Predictive Factor for Immunotherapy in Gastric Cancer. Journal of Immunology Research. 2021. 1–13. 12 indexed citations

Wang, Xu, Li-Peng Hu, Weiting Qin, et al.. (2021). Identification of a subset of immunosuppressive P2RX1-negative neutrophils in pancreatic cancer liver metastasis. Nature Communications. 12(1). 174–174. 87 indexed citations

Zhang, Xueli, Li-Peng Hu, Qin Yang, et al.. (2021). CTHRC1 promotes liver metastasis by reshaping infiltrated macrophages through physical interactions with TGF-β receptors in colorectal cancer. Oncogene. 40(23). 3959–3973. 61 indexed citations

Hu, Li-Peng, Yan‐Miao Huo, Dejun Liu, et al.. (2021). Single-cell RNA sequencing reveals that targeting HSP90 suppresses PDAC progression by restraining mitochondrial bioenergetics. Oncogenesis. 10(3). 22–22. 8 indexed citations

Yang, Qin, et al.. (2021). Targeting the complex I and III of mitochondrial electron transport chain as a potentially viable option in liver cancer management. Cell Death Discovery. 7(1). 293–293. 15 indexed citations

10.

Liu, Zhenqiu, Xianhua Mao, Jiaqi Wu, et al.. (2019). World-wide Prevalence of Substitutions in HCV Genome Associated With Resistance to Direct-Acting Antiviral Agents. Clinical Gastroenterology and Hepatology. 19(9). 1906–1914.e25. 8 indexed citations

11.

Xie, Fei, et al.. (2019). MiR-345-5p functions as a tumor suppressor in pancreatic cancer by directly targeting CCL8. Biomedicine & Pharmacotherapy. 111. 891–900. 49 indexed citations

12.

Zhu, Chunchao, Qin Yang, Jia Xu, et al.. (2019). Somatic mutation of DNAH genes implicated higher chemotherapy response rate in gastric adenocarcinoma patients. Journal of Translational Medicine. 17(1). 109–109. 20 indexed citations

13.

Zhang, Yanli, Qing Li, Fang Fang, et al.. (2018). SPON2 Promotes M1-like Macrophage Recruitment and Inhibits Hepatocellular Carcinoma Metastasis by Distinct Integrin–Rho GTPase–Hippo Pathways. Cancer Research. 78(9). 2305–2317. 119 indexed citations

14.

Hu, Li-Peng, Xiaoxin Zhang, Shu-Heng Jiang, et al.. (2018). Targeting Purinergic Receptor P2Y2 Prevents the Growth of Pancreatic Ductal Adenocarcinoma by Inhibiting Cancer Cell Glycolysis. Clinical Cancer Research. 25(4). 1318–1330. 93 indexed citations

15.

Zhang, Yanli, Xin Xing, Lei Zhu, et al.. (2018). Integrin α9 Suppresses Hepatocellular Carcinoma Metastasis by Rho GTPase Signaling. Journal of Immunology Research. 2018. 1–11. 27 indexed citations

16.

Yang, Xiaomei, Xiaoyan Cao, Ping He, et al.. (2018). Overexpression of Rac GTPase Activating Protein 1 Contributes to Proliferation of Cancer Cells by Reducing Hippo Signaling to Promote Cytokinesis. Gastroenterology. 155(4). 1233–1249.e22. 90 indexed citations

17.

Jie, Zhijun, et al.. (2018). Aberrant CpG‐methylation affects genes expression predicting survival in lung adenocarcinoma. Cancer Medicine. 7(11). 5716–5726. 19 indexed citations

18.

Yang, Qin, Zhi‐Yong Wu, Suan Sun, et al.. (2018). PD-L1 Expression Predicts a Distinct Prognosis in Krukenberg Tumor with Corresponding Origins. Journal of Immunology Research. 2018. 1–10. 14 indexed citations

19.

Wang, Yahui, Rongkun Li, Ying Fu, et al.. (2017). Exemestane Attenuates Hepatic Fibrosis in Rats by Inhibiting Activation of Hepatic Stellate Cells and Promoting the Secretion of Interleukin 10. Journal of Immunology Research. 2017. 1–9. 6 indexed citations

20.

Sah, Birendra Kumar, et al.. (2009). Post-operative complications of gastric cancer surgery: female gender at high risk. European Journal of Cancer Care. 18(2). 202–208. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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