Pierre Launay

7.4k total citations · 3 hit papers
71 papers, 5.8k citations indexed

About

Pierre Launay is a scholar working on Immunology, Molecular Biology and Sensory Systems. According to data from OpenAlex, Pierre Launay has authored 71 papers receiving a total of 5.8k indexed citations (citations by other indexed papers that have themselves been cited), including 31 papers in Immunology, 20 papers in Molecular Biology and 16 papers in Sensory Systems. Recurrent topics in Pierre Launay's work include Ion Channels and Receptors (15 papers), Mast cells and histamine (14 papers) and Monoclonal and Polyclonal Antibodies Research (13 papers). Pierre Launay is often cited by papers focused on Ion Channels and Receptors (15 papers), Mast cells and histamine (14 papers) and Monoclonal and Polyclonal Antibodies Research (13 papers). Pierre Launay collaborates with scholars based in France, United States and Brazil. Pierre Launay's co-authors include Renato C. Monteiro, Andrea Fleig, Reinhold Penner, Andrew M. Scharenberg, Jean-Pierre Kinet, Marc Benhamou, Ulrich Blank, Anne-Laure Perraud, Agnès Lehuen and Jean‐Pierre Kinet and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Pierre Launay

71 papers receiving 5.7k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

ADP-ribose gating of the calcium-permeable LTRPC2 channel revealed by Nudix motif homology

2001 750 citations Andrea Fleig, Pierre Launay et al. profile →
TRPM4 Is a Ca2+-Activated Nonselective Cation Channel Mediating Cell Membrane Depolarization

2002 583 citations Pierre Launay, Andrea Fleig et al. Cell profile →
Immune sensing of food allergens promotes avoidance behaviour

2023 86 citations Pierre Launay et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Pierre Launay France	42	1.9k	1.9k	1.8k	877	662	71	5.8k
Shang‐Zhong Xu China	35	1.9k 1.0×	1.8k 1.0×	219 0.1×	484 0.6×	405 0.6×	98	4.0k
Zheng Zhang United States	38	382 0.2×	1.4k 0.7×	286 0.2×	457 0.5×	379 0.6×	96	3.7k
Attila Braun Germany	37	1.0k 0.5×	1.7k 0.9×	572 0.3×	277 0.3×	255 0.4×	75	4.4k
Ana M. Gómez France	47	333 0.2×	4.1k 2.2×	346 0.2×	316 0.4×	505 0.8×	158	6.8k
Nora Rozengurt United States	27	477 0.3×	2.2k 1.2×	396 0.2×	645 0.7×	223 0.3×	50	4.4k
Maria F. Gomez Sweden	34	456 0.2×	1.6k 0.9×	515 0.3×	129 0.1×	598 0.9×	112	3.6k
Yassine Amrani United States	43	437 0.2×	1.6k 0.8×	1.7k 0.9×	127 0.1×	418 0.6×	108	5.1k
Alasdair M. Gilfillan United States	46	264 0.1×	2.0k 1.1×	4.7k 2.6×	185 0.2×	323 0.5×	111	6.7k
Clark Distelhorst United States	43	218 0.1×	3.8k 2.0×	759 0.4×	184 0.2×	241 0.4×	82	5.3k
Marcelle Bens France	38	160 0.1×	3.2k 1.7×	739 0.4×	341 0.4×	572 0.9×	93	5.1k

Countries citing papers authored by Pierre Launay

Since Specialization

Citations

This map shows the geographic impact of Pierre Launay's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pierre Launay with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pierre Launay more than expected).

Fields of papers citing papers by Pierre Launay

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pierre Launay. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pierre Launay. The network helps show where Pierre Launay may publish in the future.

Co-authorship network of co-authors of Pierre Launay

This figure shows the co-authorship network connecting the top 25 collaborators of Pierre Launay. A scholar is included among the top collaborators of Pierre Launay based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pierre Launay. Pierre Launay is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Launay, Pierre. (2025). 11P Preclinical evaluation of an anti-transferrin receptor antibody drug conjugate targeting triple-negative breast cancer. ESMO Open. 10. 104167–104167. 2 indexed citations

Kolodych, Sergii, Rémy Castellano, Emmanuelle Josselin, et al.. (2024). INA03: A Potent Transferrin-Competitive Antibody–Drug Conjugate against CD71 for Safer Acute Leukemia Treatment. Molecular Cancer Therapeutics. 23(8). 1159–1175. 1 indexed citations

Garciaz, Sylvain, Pierre Bories, Jean‐Marie Boher, et al.. (2023). Preliminary results of a phase 1, first-in-human study of INA03, an anti-CD71 antibody-drug conjugate, in patients with relapsed or refractory (R/R) acute leukemias.. Journal of Clinical Oncology. 41(16_suppl). 7045–7045. 3 indexed citations

Tchérakian, Colas, Paul‐Louis Woerther, Aurélie Guguin, et al.. (2019). Overexpression of GILZ in macrophages limits systemic inflammation while increasing bacterial clearance in sepsis in mice. European Journal of Immunology. 50(4). 589–602. 24 indexed citations

Carrasco, Kévin, Amir Boufenzer, Lucie Jolly, et al.. (2018). TREM-1 multimerization is essential for its activation on monocytes and neutrophils. Cellular and Molecular Immunology. 16(5). 460–472. 76 indexed citations

Mkaddem, Sanae Ben, Héloïse Flament, Dimitri Titeca‐Beauport, et al.. (2017). Lyn and Fyn function as molecular switches that control immunoreceptors to direct homeostasis or inflammation. Nature Communications. 8(1). 246–246. 82 indexed citations

Lagraauw, H. Maxime, Anouk Wezel, Pierre Launay, et al.. (2016). Mast cell depletion in the preclinical phase of collagen-induced arthritis reduces clinical outcome by lowering the inflammatory cytokine profile. Arthritis Research & Therapy. 18(1). 138–138. 28 indexed citations

Solanes, Paola, Mélina L. Heuzé, Mathieu Maurin, et al.. (2015). Space exploration by dendritic cells requires maintenance of myosin II activity by IP ₃ receptor 1. The EMBO Journal. 34(6). 798–810. 27 indexed citations

Mkaddem, Sanae Ben, Gilles Hayem, Friederike Jönsson, et al.. (2014). Shifting FcγRIIA-ITAM from activation to inhibitory configuration ameliorates arthritis. Journal of Clinical Investigation. 124(9). 3945–3959. 78 indexed citations

10.

Demion, Marie, Jérôme Thireau, Cécile Cassan, et al.. (2011). Abstract 14918: Invalidation Of Trpm4 Channel In Mouse Induces Slowed Cardiac Conduction, Arrhythmias And Hyperplasia. Circulation. 124. 2 indexed citations

11.

Gaujoux, Sébastien, Sophie Grabar, Martin Faßnacht, et al.. (2010). β-Catenin Activation Is Associated with Specific Clinical and Pathologic Characteristics and a Poor Outcome in Adrenocortical Carcinoma. Clinical Cancer Research. 17(2). 328–336. 114 indexed citations

12.

Ragazzon, Bruno, Rossella Libé, Sébastien Gaujoux, et al.. (2010). Transcriptome Analysis Reveals that p53 and β-Catenin Alterations Occur in a Group of Aggressive Adrenocortical Cancers. Cancer Research. 70(21). 8276–8281. 117 indexed citations

13.

Barbet, Gaëtan, Marie Demion, Ivan Cruz Moura, et al.. (2008). The calcium-activated nonselective cation channel TRPM4 is essential for the migration but not the maturation of dendritic cells. Nature Immunology. 9(10). 1148–1156. 174 indexed citations

14.

Alexia, Catherine, et al.. (2008). Phospholipid Scramblase 1 Modulates a Selected Set of IgE Receptor-mediated Mast Cell Responses through LAT-dependent Pathway. Journal of Biological Chemistry. 283(37). 25514–25523. 30 indexed citations

15.

Kanamaru, Yutaka, Michelle Arcos‐Fajardo, Ivan Cruz Moura, et al.. (2007). Fcα receptor I activation induces leukocyte recruitment and promotes aggravation of glomerulonephritis through the FcRγ adaptor. European Journal of Immunology. 37(4). 1116–1128. 44 indexed citations

16.

Silva, Fabiano Pinheiro da, Meryem Aloulou, David Skurnik, et al.. (2007). CD16 promotes Escherichia coli sepsis through an FcRγ inhibitory pathway that prevents phagocytosis and facilitates inflammation. Nature Medicine. 13(11). 1368–1374. 118 indexed citations

17.

Pasquier, Benoit, Pierre Launay, Yutaka Kanamaru, et al.. (2005). Identification of FcαRI as an Inhibitory Receptor that Controls Inflammation. Immunity. 22(1). 31–42. 279 indexed citations

18.

Launay, Pierre, Henrique Cheng, Subhashini Srivatsan, et al.. (2004). TRPM4 Regulates Calcium Oscillations After T Cell Activation. Science. 306(5700). 1374–1377. 270 indexed citations

19.

Launay, Pierre, Andrea Fleig, Anne-Laure Perraud, et al.. (2002). TRPM4 Is a Ca2+-Activated Nonselective Cation Channel Mediating Cell Membrane Depolarization. Cell. 109(3). 397–407. 583 indexed citations breakdown →

20.

Geissmann, Frédéric, Pierre Launay, Benoit Pasquier, et al.. (2001). A Subset of Human Dendritic Cells Expresses IgA Fc Receptor (CD89), Which Mediates Internalization and Activation Upon Cross-Linking by IgA Complexes. The Journal of Immunology. 166(1). 346–352. 111 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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