Nicolas Serafini

3.7k total citations
26 papers, 2.1k citations indexed

About

Nicolas Serafini is a scholar working on Immunology, Surgery and Molecular Biology. According to data from OpenAlex, Nicolas Serafini has authored 26 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Immunology, 10 papers in Surgery and 5 papers in Molecular Biology. Recurrent topics in Nicolas Serafini's work include IL-33, ST2, and ILC Pathways (15 papers), Immune Cell Function and Interaction (15 papers) and Eosinophilic Esophagitis (10 papers). Nicolas Serafini is often cited by papers focused on IL-33, ST2, and ILC Pathways (15 papers), Immune Cell Function and Interaction (15 papers) and Eosinophilic Esophagitis (10 papers). Nicolas Serafini collaborates with scholars based in France, United States and United Kingdom. Nicolas Serafini's co-authors include James P. Di Santo, Christian A. J. Vosshenrich, Rudi W. Hendriks, Irma Tindemans, Naoko Satoh‐Takayama, Roel G. J. Klein Wolterink, Pierre Launay, Wei Xu, Gad Frankel and Marie Demion and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Nicolas Serafini

26 papers receiving 2.1k citations

Peers

Nicolas Serafini
Sydney Lavoie United States
Petra Voland Germany
J L Madara United States
Shresh Pathak United States
Dave Boucher Australia
Sydney Lavoie United States
Nicolas Serafini
Citations per year, relative to Nicolas Serafini Nicolas Serafini (= 1×) peers Sydney Lavoie

Countries citing papers authored by Nicolas Serafini

Since Specialization
Citations

This map shows the geographic impact of Nicolas Serafini's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nicolas Serafini with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nicolas Serafini more than expected).

Fields of papers citing papers by Nicolas Serafini

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nicolas Serafini. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nicolas Serafini. The network helps show where Nicolas Serafini may publish in the future.

Co-authorship network of co-authors of Nicolas Serafini

This figure shows the co-authorship network connecting the top 25 collaborators of Nicolas Serafini. A scholar is included among the top collaborators of Nicolas Serafini based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nicolas Serafini. Nicolas Serafini is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Serafini, Nicolas & James P. Di Santo. (2024). Group 3 innate lymphoid cells: A trained Gutkeeper. Immunological Reviews. 323(1). 126–137. 5 indexed citations
2.
Garcia, Zacarias, Solenne Marie, Immo Prinz, et al.. (2022). Inflammation triggers ILC3 patrolling of the intestinal barrier. Nature Immunology. 23(9). 1317–1323. 52 indexed citations
3.
Guillemot, Vincent, Pascal Campagne, Nicolas Serafini, et al.. (2021). Host genetic control of natural killer cell diversity revealed in the Collaborative Cross. Proceedings of the National Academy of Sciences. 118(10). 8 indexed citations
4.
Gonçalves, Pedro, Sary El Daker, Florence Vasseur, et al.. (2020). Microbiota stimulation generates LCMV-specific memory CD8+ T cells in SPF mice and determines their TCR repertoire during LCMV infection. Molecular Immunology. 124. 125–141. 3 indexed citations
5.
Xu, Wei, Sylvestre Chea, Christian A. J. Vosshenrich, et al.. (2019). An Id2RFP-Reporter Mouse Redefines Innate Lymphoid Cell Precursor Potentials. Immunity. 50(4). 1054–1068.e3. 120 indexed citations
6.
Rodrigues, Pedro M., et al.. (2018). Intrathymic Deletion of IL-7 Reveals a Contribution of the Bone Marrow to Thymic Rebound Induced by Androgen Blockade. The Journal of Immunology. 200(4). 1389–1398. 10 indexed citations
7.
Li, Yan, Guillemette Masse‐Ranson, Zacarias Garcia, et al.. (2018). A human immune system mouse model with robust lymph node development. Nature Methods. 15(8). 623–630. 76 indexed citations
8.
Berger, Cédric N., Valérie F. Crepin, Theodoros I. Roumeliotis, et al.. (2018). The Citrobacter rodentium type III secretion system effector EspO affects mucosal damage repair and antimicrobial responses. PLoS Pathogens. 14(10). e1007406–e1007406. 29 indexed citations
9.
Serafini, Nicolas, et al.. (2018). Innate Lymphoid Cell Development: A T Cell Perspective. Immunity. 48(6). 1091–1103. 141 indexed citations
10.
Serafini, Nicolas, Richard H. Wheeler, Muriel Derrien, et al.. (2017). Lactobacillus paracasei feeding improves immune control of influenza infection in mice. PLoS ONE. 12(9). e0184976–e0184976. 74 indexed citations
11.
Pallett, Mitchell A., Valérie F. Crepin, Nicolas Serafini, et al.. (2016). Bacterial virulence factor inhibits caspase-4/11 activation in intestinal epithelial cells. Mucosal Immunology. 10(3). 602–612. 66 indexed citations
12.
Serafini, Nicolas, Christian A. J. Vosshenrich, & James P. Di Santo. (2015). Transcriptional regulation of innate lymphoid cell fate. Nature reviews. Immunology. 15(7). 415–428. 219 indexed citations
13.
Serafini, Nicolas, Roel G. J. Klein Wolterink, Naoko Satoh‐Takayama, et al.. (2014). Gata3 drives development of RORγt+ group 3 innate lymphoid cells. The Journal of Experimental Medicine. 211(2). 199–208. 167 indexed citations
14.
Tindemans, Irma, Nicolas Serafini, James P. Di Santo, & Rudi W. Hendriks. (2014). GATA-3 Function in Innate and Adaptive Immunity. Immunity. 41(2). 191–206. 203 indexed citations
15.
Demion, Marie, Jérôme Thireau, Amanda Finan, et al.. (2014). Trpm4 Gene Invalidation Leads to Cardiac Hypertrophy and Electrophysiological Alterations. PLoS ONE. 9(12). e115256–e115256. 66 indexed citations
16.
Satoh‐Takayama, Naoko, Nicolas Serafini, Abdessalem Rekiki, et al.. (2014). The Chemokine Receptor CXCR6 Controls the Functional Topography of Interleukin-22 Producing Intestinal Innate Lymphoid Cells. Immunity. 41(5). 776–788. 133 indexed citations
17.
Wolterink, Roel G. J. Klein, Nicolas Serafini, Menno van Nimwegen, et al.. (2013). Essential, dose-dependent role for the transcription factor Gata3 in the development of IL-5 + and IL-13 + type 2 innate lymphoid cells. Proceedings of the National Academy of Sciences. 110(25). 10240–10245. 185 indexed citations
18.
Demion, Marie, Jérôme Thireau, Cécile Cassan, et al.. (2011). Abstract 14918: Invalidation Of Trpm4 Channel In Mouse Induces Slowed Cardiac Conduction, Arrhythmias And Hyperplasia. Circulation. 124. 2 indexed citations
19.
Barbet, Gaëtan, Marie Demion, Ivan Cruz Moura, et al.. (2008). The calcium-activated nonselective cation channel TRPM4 is essential for the migration but not the maturation of dendritic cells. Nature Immunology. 9(10). 1148–1156. 174 indexed citations
20.
Kolísek, Martin, Pierre Launay, Andreas Beck, et al.. (2008). SLC41A1 Is a Novel Mammalian Mg2+ Carrier. Journal of Biological Chemistry. 283(23). 16235–16247. 96 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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