Phyllis S. Frisa
- Co-authors
- James W. JacobbergerJanice G. DouglasPhilip G. WoostDavid E. OroszUlrich HopferR. Michael SramkoskiJerry SilverGeorge M. Smith
- Topics
- Eosinophilic Disorders and Syndromes (2 papers)Ion Transport and Channel Regulation (2 papers)Chronic Myeloid Leukemia Treatments (2 papers)
- Partner nations
- United StatesCanadaHong Kong
In The Last Decade
Phyllis S. Frisa
13 papers receiving 392 citations
Peers
Comparison fields: 5 of 67
- Molecular Biology 260
- Oncology 62
- Cell Biology 39
- Physiology 37
- Hematology 36
Countries citing papers authored by Phyllis S. Frisa
This map shows the geographic impact of Phyllis S. Frisa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Phyllis S. Frisa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Phyllis S. Frisa more than expected).
Fields of papers citing papers by Phyllis S. Frisa
This network shows the impact of papers produced by Phyllis S. Frisa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Phyllis S. Frisa. The network helps show where Phyllis S. Frisa may publish in the future.
Co-authorship network of co-authors of Phyllis S. Frisa
This figure shows the co-authorship network connecting the top 25 collaborators of Phyllis S. Frisa. A scholar is included among the top collaborators of Phyllis S. Frisa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Phyllis S. Frisa. Phyllis S. Frisa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 13 | |
| 2 | 27 | |
| 3 | 32 | |
| 4 | 42 | |
| 5 | 42 | |
| 6 | Phase I and correlative study of combination bryostatin 1 and vincristine in relapsed B-cell malignancies. | 38 |
| 7 | Original Articles Immunoreactivity of Stat5 Phosphorylated on Tyrosine as a Cell-Based Measure of Bcr/Abl Kinase Activity | 1 |
| 8 | 16 | |
| 9 | 10 | |
| 10 | 115 | |
| 11 | Stepwise transformation of astrocytes by simian virus 40 large T antigen and epidermal growth factor receptor overexpression. | 10 |
| 12 | 41 | |
| 13 | 10 |
About Phyllis S. Frisa
Phyllis S. Frisa is a scholar working on Hematology, Developmental Neuroscience and Physiology, having authored 13 papers that have together received 397 indexed citations. Recurring topics across this work include Eosinophilic Disorders and Syndromes (2 papers), Ion Transport and Channel Regulation (2 papers) and Chronic Myeloid Leukemia Treatments (2 papers). The work is most often cited by research in Biophysics (31 citations), Molecular Biology (260 citations) and Developmental Neuroscience (14 citations). Phyllis S. Frisa has collaborated with scholars based in United States, Canada and Hong Kong. Frequent co-authors include James W. Jacobberger, Janice G. Douglas, Philip G. Woost, David E. Orosz, Ulrich Hopfer, R. Michael Sramkoski, Jerry Silver, George M. Smith, Tammy Stefan and D. R. Sonneborn. Their work appears in journals such as Proceedings of the National Academy of Sciences, PLoS ONE and Kidney International.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.