Phillip F. Pratt

4.2k total citations · 1 hit paper
56 papers, 3.6k citations indexed

About

Phillip F. Pratt is a scholar working on Physiology, Pathology and Forensic Medicine and Molecular Biology. According to data from OpenAlex, Phillip F. Pratt has authored 56 papers receiving a total of 3.6k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Physiology, 19 papers in Pathology and Forensic Medicine and 16 papers in Molecular Biology. Recurrent topics in Phillip F. Pratt's work include Nitric Oxide and Endothelin Effects (23 papers), Cardiac Ischemia and Reperfusion (17 papers) and Eicosanoids and Hypertension Pharmacology (9 papers). Phillip F. Pratt is often cited by papers focused on Nitric Oxide and Endothelin Effects (23 papers), Cardiac Ischemia and Reperfusion (17 papers) and Eicosanoids and Hypertension Pharmacology (9 papers). Phillip F. Pratt collaborates with scholars based in United States, Czechia and France. Phillip F. Pratt's co-authors include William B. Campbell, David R. Harder, Debebe Gebremedhin, David C. Warltier, Judy R. Kersten, Paul S. Pagel, Dorothée Weihrauch, John G. Krolikowski, Garrett J. Gross and Cecilia J. Hillard and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and Journal of Clinical Investigation.

In The Last Decade

Phillip F. Pratt

56 papers receiving 3.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Identification of Epoxyeicosatrienoic Acids as Endothelium-Derived Hyperpolarizing Factors

1996 1.1k citations William B. Campbell, Debebe Gebremedhin et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Phillip F. Pratt United States	31	1.2k	1.1k	998	851	751	56	3.6k
Galen M. Pieper United States	39	472 0.4×	2.3k 2.0×	981 1.0×	995 1.2×	1.5k 2.0×	134	4.9k
S. Jamal Mustafa United States	38	398 0.3×	1.5k 1.3×	1.2k 1.2×	514 0.6×	1.1k 1.4×	170	4.5k
Ai-Ping Zou United States	41	671 0.5×	1.4k 1.2×	1.4k 1.4×	331 0.4×	953 1.3×	57	3.8k
Susheel Gundewar United States	16	811 0.7×	902 0.8×	1.1k 1.1×	512 0.6×	640 0.9×	23	2.7k
Salvatore Pepe Australia	35	352 0.3×	751 0.7×	2.2k 2.2×	1.2k 1.4×	1.3k 1.7×	115	4.4k
František Kolář Czechia	31	218 0.2×	785 0.7×	1.4k 1.4×	1.1k 1.3×	1.1k 1.4×	183	3.3k
Prasad V. G. Katakam United States	35	294 0.2×	970 0.8×	1.1k 1.1×	443 0.5×	499 0.7×	96	2.8k
Xinliang Ma United States	46	299 0.2×	1.9k 1.7×	2.6k 2.6×	1.2k 1.5×	2.0k 2.7×	182	7.0k
Earl F. Ellis United States	34	570 0.5×	599 0.5×	1.3k 1.3×	294 0.3×	351 0.5×	62	3.8k
Masanori Takaoka Japan	36	347 0.3×	1.8k 1.6×	1.2k 1.2×	385 0.5×	1.2k 1.6×	144	3.8k

Countries citing papers authored by Phillip F. Pratt

Since Specialization

Citations

This map shows the geographic impact of Phillip F. Pratt's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Phillip F. Pratt with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Phillip F. Pratt more than expected).

Fields of papers citing papers by Phillip F. Pratt

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Phillip F. Pratt. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Phillip F. Pratt. The network helps show where Phillip F. Pratt may publish in the future.

Co-authorship network of co-authors of Phillip F. Pratt

This figure shows the co-authorship network connecting the top 25 collaborators of Phillip F. Pratt. A scholar is included among the top collaborators of Phillip F. Pratt based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Phillip F. Pratt. Phillip F. Pratt is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Doody, Kevin J., K.M. Doody, Phillip F. Pratt, & Konstance Knox. (2019). Prospective evaluation of human herpesvirus 6 (HHV-6) in endometrial tissues of women with repeat implantation failure. Fertility and Sterility. 112(3). e334–e334. 1 indexed citations

Leucker, Thorsten M., Martin Bienengraeber, Maria Muravyeva, et al.. (2011). Endothelial–cardiomyocyte crosstalk enhances pharmacological cardioprotection. Journal of Molecular and Cellular Cardiology. 51(5). 803–811. 59 indexed citations

Ge, Zhi‐Dong, Danijel Pravdić, Martin Bienengraeber, et al.. (2009). Isoflurane Postconditioning Protects against Reperfusion Injury by Preventing Mitochondrial Permeability Transition by an Endothelial Nitric Oxide Synthase–dependent Mechanism. Anesthesiology. 112(1). 73–85. 69 indexed citations

Pagel, Paul S., John G. Krolikowski, Phillip F. Pratt, et al.. (2008). Reactive Oxygen Species and Mitochondrial Adenosine Triphosphate–Regulated Potassium Channels Mediate Helium-Induced Preconditioning Against Myocardial Infarction In Vivo. Journal of Cardiothoracic and Vascular Anesthesia. 22(4). 554–559. 32 indexed citations

Pratt, Phillip F., Martin Bienengraeber, Dorothée Weihrauch, Judy R. Kersten, & David C. Warltier. (2008). Abstract 1904: Myocardial Postconditioning Against Ischemia and Reperfusion Injury by Near Infrared Electromagnetic Radiation. Circulation. 118(suppl_18). 1 indexed citations

Pagel, Paul S., John G. Krolikowski, Phillip F. Pratt, et al.. (2008). The Mechanism of Helium-Induced Preconditioning: A Direct Role for Nitric Oxide in Rabbits. Anesthesia & Analgesia. 107(3). 762–768. 32 indexed citations

Wang, Chen, Dorothée Weihrauch, Martin Bienengraeber, et al.. (2006). Extracellular Signal-Regulated Kinases Trigger Isoflurane Preconditioning Concomitant with Upregulation of Hypoxia-Inducible Factor-1α and Vascular Endothelial Growth Factor Expression in Rats. Anesthesia & Analgesia. 103(2). 281–288. 68 indexed citations

Pratt, Phillip F., Chen Wang, Dorothée Weihrauch, et al.. (2006). Cardioprotection by volatile anesthetics: new applications for old drugs?. Current Opinion in Anaesthesiology. 19(4). 397–403. 28 indexed citations

Terashvili, Maia, Phillip F. Pratt, Debebe Gebremedhin, Jayashree Narayanan, & David R. Harder. (2006). Reactive Oxygen Species Cerebral Autoregulation in Health and Disease. Pediatric Clinics of North America. 53(5). 1029–1037. 21 indexed citations

10.

Pratt, Phillip F., et al.. (2003). Functional expression of L-type calcium channels in cultured rat renal smooth muscle cells. The FASEB Journal. 17. 6546. 1 indexed citations

11.

Prosser, Simon, Елена А. Сорокина, Phillip F. Pratt, & Andrey Sorokin. (2003). CrkIII: a novel and biologically distinct member of the Crk family of adaptor proteins. Oncogene. 22(31). 4799–4806. 27 indexed citations

12.

Albarwani, Sulayma, et al.. (2003). Voltage-gated K+ channels in rat small cerebral arteries: molecular identity of the functional channels. The Journal of Physiology. 551(3). 751–763. 105 indexed citations

13.

Pratt, Phillip F., Dirk Bokemeyer, Marco Foschi, Andrey Sorokin, & Michael J. Dünn. (2003). Alterations in Subcellular Localization of p38 MAPK Potentiates Endothelin-stimulated COX-2 Expression in Glomerular Mesangial Cells. Journal of Biological Chemistry. 278(51). 51928–51936. 37 indexed citations

14.

Pratt, Phillip F., Mark Rosolowsky, & William B. Campbell. (2002). Effects of epoxyeicosatrienoic acids on polymorphonuclear leukocyte function. Life Sciences. 70(21). 2521–2533. 27 indexed citations

15.

Pratt, Phillip F., Sébastien Bonnet, Lynda M. Ludwig, Pierre Bonnet, & Nancy J. Rusch. (2002). Upregulation of L-Type Ca ²⁺ Channels in Mesenteric and Skeletal Arteries of SHR. Hypertension. 40(2). 214–219. 118 indexed citations

16.

Fryer, Ryan M., Phillip F. Pratt, Anna K. Hsu, & Garrett J. Gross. (2001). Differential Activation of Extracellular Signal Regulated Kinase Isoforms in Preconditioning and Opioid-Induced Cardioprotection. Journal of Pharmacology and Experimental Therapeutics. 296(2). 642–649. 129 indexed citations

17.

Nithipatikom, Kasem, Phillip F. Pratt, & William B. Campbell. (1996). Nitro-L-Arginine Interferes with the Cadmium Reduction of Nitrate/GriessReaction Method of Measuring Nitric Oxide Production. Clinical Chemistry and Laboratory Medicine (CCLM). 34(2). 133–138. 10 indexed citations

18.

Dundore, Ronald L., et al.. (1992). Hemodynamic and Renal Effects of the Protein Kinase Inhibitor Staurosporine in Conscious Rats. Journal of Cardiovascular Pharmacology. 20(4). 525–532. 4 indexed citations

19.

Dundore, Ronald L., et al.. (1991). Nω-Nitro-L-arginine attenuates the accumulation of aortic cyclic GMP and the hypotension produced by zaprinast. European Journal of Pharmacology. 200(1). 83–87. 14 indexed citations

20.

Silver, Paul J., et al.. (1991). Reversal or nitroglycerin tolerance by the cGMP phosphodiesterase inhibitor zaprinast. European Journal of Pharmacology. 199(1). 141–142. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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