Philip H. Jones

13.2k total citations · 6 hit papers
96 papers, 8.6k citations indexed

About

Philip H. Jones is a scholar working on Molecular Biology, Oncology and Cancer Research. According to data from OpenAlex, Philip H. Jones has authored 96 papers receiving a total of 8.6k indexed citations (citations by other indexed papers that have themselves been cited), including 50 papers in Molecular Biology, 25 papers in Oncology and 25 papers in Cancer Research. Recurrent topics in Philip H. Jones's work include Cancer Cells and Metastasis (16 papers), Cancer Genomics and Diagnostics (15 papers) and Epigenetics and DNA Methylation (13 papers). Philip H. Jones is often cited by papers focused on Cancer Cells and Metastasis (16 papers), Cancer Genomics and Diagnostics (15 papers) and Epigenetics and DNA Methylation (13 papers). Philip H. Jones collaborates with scholars based in United Kingdom, United States and Spain. Philip H. Jones's co-authors include Fiona M. Watt, Benjamin D. Simons, David P. Doupé, Allon M. Klein, Steven J. Harper, Michael R. Stratton, Peter J. Campbell, Ludmil B. Alexandrov, David C. Wedge and Maria P. Alcolea and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

Philip H. Jones

93 papers receiving 8.5k citations

Hit Papers

5 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

High burden and pervasive positive selection of somatic mutations in normal human skin

2015 1.1k citations Iñigo Martincorena, Amit Roshan et al. Science profile →
Separation of human epidermal stem cells from transit amplifying cells on the basis of differences in integrin function and expression

1993 941 citations Philip H. Jones, Fiona M. Watt profile →
Stem cell patterning and fate in human epidermis

1995 660 citations Philip H. Jones, Fiona M. Watt et al. profile →
Somatic mutant clones colonize the human esophagus with age

2018 651 citations Iñigo Martincorena, Joanna C. Fowler et al. Science profile →
A single type of progenitor cell maintains normal epidermis

2007 649 citations Benjamin D. Simons, Philip H. Jones et al. Nature profile →
Clock-like mutational processes in human somatic cells

2015 569 citations Ludmil B. Alexandrov, Philip H. Jones et al. Nature Genetics profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Philip H. Jones United Kingdom	38	4.4k	2.0k	1.9k	1.9k	910	96	8.6k
Jyrki Heino Finland	56	4.3k 1.0×	1.9k 1.0×	1.2k 0.6×	2.6k 1.4×	878 1.0×	196	10.7k
Cornelia Mauch Germany	49	2.7k 0.6×	1.4k 0.7×	3.0k 1.6×	1.3k 0.7×	532 0.6×	208	7.9k
Katsutoshi Yoshizato Japan	56	3.6k 0.8×	843 0.4×	896 0.5×	1.7k 0.9×	715 0.8×	269	9.8k
Irene M. Leigh United Kingdom	54	2.7k 0.6×	615 0.3×	2.2k 1.1×	1.9k 1.0×	776 0.9×	184	9.1k
Thomas Bugge United States	62	4.5k 1.0×	2.4k 1.2×	1.9k 1.0×	1.2k 0.6×	1.4k 1.5×	165	11.2k
Paul A. Khavari United States	68	12.2k 2.8×	4.2k 2.1×	2.4k 1.2×	2.1k 1.1×	1.8k 2.0×	151	16.9k
Guerrino Meneguzzi France	46	3.3k 0.7×	918 0.5×	1.2k 0.6×	3.3k 1.8×	1.0k 1.1×	155	7.7k
John Couchman United States	72	7.5k 1.7×	2.3k 1.2×	1.3k 0.7×	9.2k 4.9×	1.6k 1.7×	204	15.6k
Sylvie Ricard‐Blum France	45	3.3k 0.8×	1.1k 0.5×	736 0.4×	1.9k 1.0×	763 0.8×	139	8.2k
John W. Tobias United States	46	6.1k 1.4×	1.8k 0.9×	1.5k 0.8×	946 0.5×	1.1k 1.2×	111	9.3k

Countries citing papers authored by Philip H. Jones

Since Specialization

Citations

This map shows the geographic impact of Philip H. Jones's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Philip H. Jones with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Philip H. Jones more than expected).

Fields of papers citing papers by Philip H. Jones

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Philip H. Jones. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Philip H. Jones. The network helps show where Philip H. Jones may publish in the future.

Co-authorship network of co-authors of Philip H. Jones

This figure shows the co-authorship network connecting the top 25 collaborators of Philip H. Jones. A scholar is included among the top collaborators of Philip H. Jones based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Philip H. Jones. Philip H. Jones is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Ólafsson, Sigurgeir, Elke Rodríguez, Andrew Lawson, et al.. (2023). Effects of psoriasis and psoralen exposure on the somatic mutation landscape of the skin. Nature Genetics. 55(11). 1892–1900. 4 indexed citations

Hall, Michael, et al.. (2023). Mutations observed in somatic evolution reveal underlying gene mechanisms. Communications Biology. 6(1). 753–753. 4 indexed citations

Abby, Emilie, Stefan C. Dentro, Michael Hall, et al.. (2023). Notch1 mutations drive clonal expansion in normal esophageal epithelium but impair tumor growth. Nature Genetics. 55(2). 232–245. 50 indexed citations

King, Charlotte, Joanna C. Fowler, Irina Abnizova, et al.. (2023). Somatic mutations in facial skin from countries of contrasting skin cancer risk. Nature Genetics. 55(9). 1440–1447. 15 indexed citations

Fowler, Joanna C. & Philip H. Jones. (2022). Somatic Mutation: What Shapes the Mutational Landscape of Normal Epithelia?. Cancer Discovery. 12(7). 1642–1655. 19 indexed citations

Murai, Kasumi, Stefan C. Dentro, Swee Hoe Ong, et al.. (2022). p53 mutation in normal esophagus promotes multiple stages of carcinogenesis but is constrained by clonal competition. Nature Communications. 13(1). 6206–6206. 25 indexed citations

Kostiou, Vasiliki, Michael Hall, Philip H. Jones, & Benjamin A. Hall. (2021). Simulations reveal that different responses to cell crowding determine the expansion of p53 and Notch mutant clones in squamous epithelia. Journal of The Royal Society Interface. 18(183). 20210607–20210607. 4 indexed citations

Colom, Bartomeu, Albert Herms, Michael Hall, et al.. (2021). Mutant clones in normal epithelium outcompete and eliminate emerging tumours. Nature. 598(7881). 510–514. 105 indexed citations

Colom, Bartomeu, Maria P. Alcolea, Gabriel Piedrafita, et al.. (2020). Spatial competition shapes the dynamic mutational landscape of normal esophageal epithelium. Nature Genetics. 52(6). 604–614. 106 indexed citations

10.

Belly, Henry De, Aki Stubb, Ayaka Yanagida, et al.. (2020). Membrane Tension Gates ERK-Mediated Regulation of Pluripotent Cell Fate. Cell stem cell. 28(2). 273–284.e6. 111 indexed citations

11.

Martincorena, Iñigo, Joanna C. Fowler, Agnieszka Wabik, et al.. (2018). Somatic mutant clones colonize the human esophagus with age. Science. 362(6417). 911–917. 651 indexed citations breakdown →

12.

Alexandrov, Ludmil B., Philip H. Jones, David C. Wedge, et al.. (2015). Clock-like mutational processes in human somatic cells. Nature Genetics. 47(12). 1402–1407. 569 indexed citations breakdown →

13.

Martincorena, Iñigo, Amit Roshan, Moritz Gerstung, et al.. (2015). High burden and pervasive positive selection of somatic mutations in normal human skin. Science. 348(6237). 880–886. 1123 indexed citations breakdown →

14.

Roshan, Amit, Philip H. Jones, & Chris Greenman. (2014). Exact, time-independent estimation of clone size distributions in normal and mutated cells. Journal of The Royal Society Interface. 11(99). 20140654–20140654. 6 indexed citations

15.

Barbera, Mariagnese, Massimiliano di Pietro, Elaine Walker, et al.. (2014). The human squamous oesophagus has widespread capacity for clonal expansion from cells at diverse stages of differentiation. Gut. 64(1). 11–19. 38 indexed citations

16.

Alcolea, Maria P., Philip Greulich, Agnieszka Wabik, et al.. (2014). Differentiation imbalance in single oesophageal progenitor cells causes clonal immortalization and field change. Nature Cell Biology. 16(6). 612–619. 124 indexed citations

17.

Albertella, Mark R., Paul M. Loadman, Philip H. Jones, et al.. (2008). Hypoxia-Selective Targeting by the Bioreductive Prodrug AQ4N in Patients with Solid Tumors: Results of a Phase I Study. Clinical Cancer Research. 14(4). 1096–1104. 98 indexed citations

18.

Williamson, Daniel, Joanna Selfe, Tony Gordon, et al.. (2007). Role for Amplification and Expression of Glypican-5 in Rhabdomyosarcoma. Cancer Research. 67(1). 57–65. 77 indexed citations

19.

Jones, Philip H., Benjamin D. Simons, & Fiona M. Watt. (2007). Sic Transit Gloria: Farewell to the Epidermal Transit Amplifying Cell?. Cell stem cell. 1(4). 371–381. 135 indexed citations

20.

Bailey, Mick, K. Haverson, Charlotte Inman, et al.. (2005). The influence of environment on development of the mucosal immune system. Veterinary Immunology and Immunopathology. 108(1-2). 189–198. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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