Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies
if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the
same subfield and year (this is the minimum needed to enter the top 1%, not the average
within it), or reaches the top citation threshold in at least one of its specific research
topics.
The Apoptosis Inhibitor Gene API2 and a Novel 18q Gene,MLT, Are Recurrently Rearranged in the t(11;18)(q21;q21) Associated With Mucosa-Associated Lymphoid Tissue Lymphomas
1999530 citationsJudith Dierlamm, Mathijs Baens et al.Bloodprofile →
Author Peers
Peers are selected by citation overlap in the author's most active subfields.
citations ·
hero ref
This map shows the geographic impact of Peter Marynen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peter Marynen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peter Marynen more than expected).
This network shows the impact of papers produced by Peter Marynen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peter Marynen. The network helps show where Peter Marynen may publish in the future.
Co-authorship network of co-authors of Peter Marynen
This figure shows the co-authorship network connecting the top 25 collaborators of Peter Marynen.
A scholar is included among the top collaborators of Peter Marynen based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with Peter Marynen. Peter Marynen is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
Włodarska, Iwona, Thomas Tousseyn, Laurence de Leval, et al.. (2009). Novel t(x;14)(p11.4;q32.33) resulting in upregulation of gpr34 and activation of the nfkb pathway is recurrent in malt lymphomas. Haematologica. 94. 271–272.3 indexed citations
Roosbroeck, Katrien Van, Idoya Lahortiga, Jan Cools, et al.. (2008). A novel t(4;9)(q21;p24) fuses SEC31A to JAK2 in nodular-sclerosis Hodgkin lymphoma. Haematologica. 92. 245–245.2 indexed citations
5.
Keersmaecker, Kim De, Willy Landuyt, Peter Vandenberghe, et al.. (2007). EML1-ABL1 is activated by coiled coil mediated oligomerization and induces T-cell acute lymphoblastic or chronic myeloid leukemia in a mouse bone marrow transplant model. 92. 326–326.
Esch, Hilde Van, Marijke Bauters, Jaakko Ignatius, et al.. (2006). Duplication of the MECP2 region is a frequent cause of severe mental retardation and neurological symptoms in males. 17(1). 119–119.1 indexed citations
8.
Menten, Björn, Cindy Melotte, Bernard Thienpont, et al.. (2005). Molecular karkyotyping detecs structural low grade mosaics in 4 % of patietns with idiopathic mental retardation and multiple congenital aberrations. European Journal of Human Genetics. 13. 63.2 indexed citations
9.
Vermeesch, Joris, Nicole Maas, Cindy Melotte, et al.. (2004). Micro-array CGH analysis of 4p microdeletions refines the genotype-phenotype map of the region and pinpoints low copy repeats as susceptibility sites for terminal chromosomal deletions. European Journal of Human Genetics. 12. 145.
10.
Martini, Alessandra Cadete, et al.. (2002). The TET RNA-binding proteins, EWSR1 and TAF15, are involved in acute lymphoblastic leukemia, through fusion with a new transcription factor, CIZ/NMP4. Blood. 100(11).2 indexed citations
11.
Poppe, Bruce, Nurten Yigit, Peter Marynen, et al.. (2001). Molecular characterization of a t(2;3)(p23;q26): A recurrent translocation involving the EVI1 gene. Ghent University Academic Bibliography (Ghent University).2 indexed citations
12.
Ma, Zhong, Jan Cools, Peter Marynen, et al.. (1999). Inv(2)(p23q35) in anaplastic large-cell lymphoma induces constitutive ALK tyrosine kinase activation by fusion to ATIC, an enzyme involved in purine nucleotide biosynthesis. Blood.2 indexed citations
13.
Dierlamm, Judith, M Stefanova, Kristina Hinz, et al.. (1999). The t(11;18)(q21;q21) characterizes a subgroup of extranodal MALT lymphomas: a two-color fluor-escence in situ hybridization study using AP12 and MLT specific probes. Blood.1 indexed citations
14.
Bekaert, Sofie, Joris Vermeesch, Peter Marynen, & Patric Van Oostveldt. (1998). Telomere length regulation in telomerase positive murine cell line. Ghent University Academic Bibliography (Ghent University).1 indexed citations
Cools, Jan, et al.. (1998). Identification and characterisation of BTL, a new fusion partner of ETV6 in 1 T-ALL case and 2 AML cases with a translocation t(4;12) (q11;p13). Blood. 92(10).1 indexed citations
17.
Dierlamm, Judith, Iwona Włodarska, M Stefanova, et al.. (1998). Molecular cytogenetic characterization of the chromosome 11 and 18 breakpoints in the t(11;18)(q21;q21) associated with malt lymphomas. Blood. 92(10).1 indexed citations
18.
Soekarman, D, et al.. (1996). A form of non-specific mental retardation is probably caused by a microdeletion in a Belgian family. American Journal of Medical Genetics Part A. 64(1). 16.12 indexed citations
19.
Włodarska, Iwona, C Mecucci, Ana Aventín, et al.. (1994). FISH analysis of typical t(5;12) (q32;p12) and variant t(10;12)(q23;p12) translocation in four patients with chronic myeloid disorders. British Journal of Haematology. 87. 203.4 indexed citations
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.