Peter G. Wells

11.6k total citations · 1 hit paper
203 papers, 8.2k citations indexed

About

Peter G. Wells is a scholar working on Molecular Biology, Pediatrics, Perinatology and Child Health and Health, Toxicology and Mutagenesis. According to data from OpenAlex, Peter G. Wells has authored 203 papers receiving a total of 8.2k indexed citations (citations by other indexed papers that have themselves been cited), including 64 papers in Molecular Biology, 50 papers in Pediatrics, Perinatology and Child Health and 28 papers in Health, Toxicology and Mutagenesis. Recurrent topics in Peter G. Wells's work include Pharmacogenetics and Drug Metabolism (23 papers), Birth, Development, and Health (22 papers) and Prenatal Substance Exposure Effects (19 papers). Peter G. Wells is often cited by papers focused on Pharmacogenetics and Drug Metabolism (23 papers), Birth, Development, and Health (22 papers) and Prenatal Substance Exposure Effects (19 papers). Peter G. Wells collaborates with scholars based in Canada, United States and United Kingdom. Peter G. Wells's co-authors include Louise M. Winn, Michael J. Wiley, Toufan Parman, Gordon P. McCallum, Christopher J.B. Nicol, Rebecca R. Laposa, D. L. Peterson, Damon J. Hamel, Raymond C. Stevens and Gustavo Fenalti and has published in prestigious journals such as Science, Journal of Biological Chemistry and Nature Medicine.

In The Last Decade

Peter G. Wells

199 papers receiving 7.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Structures of the CXCR4 Chemokine GPCR with Small-Molecule and Cyclic Peptide Antagonists

2010 1.5k citations Beili Wu, Ellen Y. T. Chien et al. Science profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Peter G. Wells Canada	42	3.2k	1.5k	1.2k	1.1k	675	203	8.2k
Theo J. Visser Netherlands	84	4.8k 1.5×	3.7k 2.4×	2.7k 2.3×	1.7k 1.6×	919 1.4×	391	24.8k
Bruce Blumberg United States	65	8.0k 2.5×	1.5k 1.0×	5.0k 4.3×	1.3k 1.3×	781 1.2×	148	18.2k
Vasilis Vasiliou United States	65	7.0k 2.2×	463 0.3×	883 0.7×	2.4k 2.2×	481 0.7×	264	14.8k
Robert Barouki France	44	2.3k 0.7×	614 0.4×	2.5k 2.1×	486 0.5×	163 0.2×	189	7.1k
Kathryn Z. Guyton United States	48	3.7k 1.2×	272 0.2×	2.0k 1.7×	1.0k 1.0×	219 0.3×	84	10.5k
Jack A. Taylor United States	59	5.7k 1.8×	1.1k 0.7×	901 0.8×	1.4k 1.3×	80 0.1×	220	11.0k
James R. Olson United States	55	3.0k 0.9×	404 0.3×	3.6k 3.0×	474 0.4×	271 0.4×	322	10.9k
John A. McLachlan United States	67	4.1k 1.3×	910 0.6×	6.3k 5.3×	1.3k 1.2×	233 0.3×	246	15.6k
David F. Smith United States	52	2.8k 0.9×	1.0k 0.7×	1.1k 0.9×	687 0.6×	432 0.6×	194	10.0k
David M. Umbach United States	57	3.0k 1.0×	489 0.3×	1.4k 1.2×	673 0.6×	552 0.8×	213	10.2k

Countries citing papers authored by Peter G. Wells

Since Specialization

Citations

This map shows the geographic impact of Peter G. Wells's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Peter G. Wells with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Peter G. Wells more than expected).

Fields of papers citing papers by Peter G. Wells

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Peter G. Wells. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Peter G. Wells. The network helps show where Peter G. Wells may publish in the future.

Co-authorship network of co-authors of Peter G. Wells

This figure shows the co-authorship network connecting the top 25 collaborators of Peter G. Wells. A scholar is included among the top collaborators of Peter G. Wells based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Peter G. Wells. Peter G. Wells is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Wells, Peter G., et al.. (2021). Novel mechanisms in alcohol neurodevelopmental disorders via BRCA1 depletion and BRCA1-dependent NADPH oxidase regulation. Redox Biology. 48. 102148–102148. 6 indexed citations

Matthews, Jason, et al.. (2019). Characterization of Epigenetic Histone Activation/Repression Marks in Sequences of Genes by Chromatin Immunoprecipitation-Quantitative Polymerase Chain Reaction (ChIP-qPCR). Methods in molecular biology. 1965. 389–403. 8 indexed citations

Wells, Peter G., et al.. (2015). Embryonic catalase protects against ethanol embryopathies in acatalasemic mice and transgenic human catalase-expressing mice in embryo culture. Toxicology and Applied Pharmacology. 287(3). 232–239. 14 indexed citations

Chan, Norman, Mohsin Ali, Gordon P. McCallum, et al.. (2014). Hypoxia Provokes Base Excision Repair Changes and a Repair-Deficient, Mutator Phenotype in Colorectal Cancer Cells. Molecular Cancer Research. 12(10). 1407–1415. 41 indexed citations

Tamblyn, Laura, et al.. (2012). Oxoguanine Glycosylase 1 (OGG1) Protects Cells from DNA Double-Strand Break Damage Following Methylmercury (MeHg) Exposure. Toxicological Sciences. 128(1). 272–283. 16 indexed citations

Wu, Beili, Ellen Y. T. Chien, Clifford D. Mol, et al.. (2010). Structures of the CXCR4 Chemokine GPCR with Small-Molecule and Cyclic Peptide Antagonists. Science. 330(6007). 1066–1071. 1462 indexed citations breakdown →

Jeng, Winnie, Annmarie Ramkissoon, & Peter G. Wells. (2010). Reduced DNA oxidation in aged prostaglandin H synthase-1 knockout mice. Free Radical Biology and Medicine. 50(4). 550–556. 5 indexed citations

Wells, Peter G., Gordon P. McCallum, Jeffrey T. Henderson, et al.. (2009). Oxidative Stress in Developmental Origins of Disease: Teratogenesis, Neurodevelopmental Deficits, and Cancer. Toxicological Sciences. 108(1). 4–18. 336 indexed citations

Jeng, Winnie, Andrea W. Wong, Ryan Ting‐A‐Kee, & Peter G. Wells. (2005). Methamphetamine-enhanced embryonic oxidative DNA damage and neurodevelopmental deficits. Free Radical Biology and Medicine. 39(3). 317–326. 62 indexed citations

10.

Wells, Peter G., Chao Chen, Winnie Jeng, et al.. (2005). Molecular and biochemical mechanisms in teratogenesis involving reactive oxygen species. Toxicology and Applied Pharmacology. 207(2). 354–366. 180 indexed citations

11.

Kennedy, Julia, Sylvie Mémet, & Peter G. Wells. (2004). Antisense Evidence for Nuclear Factor-κB–Dependent Embryopathies Initiated by Phenytoin-Enhanced Oxidative Stress. Molecular Pharmacology. 66(3). 404–412. 12 indexed citations

12.

Jones, Stephen H., et al.. (2001). Monitoring for toxic contaminants in Mytilus edulis from New Hampshire and the Gulf of Maine. Journal of Shellfish Research. 9 indexed citations

13.

Wiley, Michael J., et al.. (1995). In vitro murine embryotoxicity of cyclophosphamide in embryos co-cultured with maternal hepatocytes: development and application of a murine embryo-hepatocyte co-culture model. Toxicology. 102(3). 259–274. 14 indexed citations

14.

Liu, Li & Peter G. Wells. (1995). Potential Molecular Targets Mediating Chemical Teratogenesis: In Vitro Peroxidase-Catalyzed Phenytoin Metabolism and Oxidative Damage to Proteins and Lipids in Murine Maternal Hepatic Microsomes and Embryonic 9000g Supernatant. Toxicology and Applied Pharmacology. 134(1). 71–80. 15 indexed citations

15.

Hú, Zhìhóng & Peter G. Wells. (1994). Modulation of Benzo[a]pyrene Bioactivation by Glucuronidation in Lymphocytes and Hepatic Microsomes from Rats with a Hereditary Deficiency in Bilirubin UDP-Glucuronosyltransferase. Toxicology and Applied Pharmacology. 127(2). 306–313. 32 indexed citations

16.

Wells, Peter G. & B. Faragher. (1992). Referral rates to a regional adolescent psychiatric service: a comparison of visited and unvisited general practitioners.. PubMed. 24(2). 53–6. 3 indexed citations

17.

Côté, Raymond P., et al.. (1991). Controlling chemical hazards: Fundamentals of the management of toxic chemicals. Environment International. 17(6). 616–616. 1 indexed citations

18.

Wells, Peter G., et al.. (1989). Modulation of phenytoin teratogenicity and embryonic covalent binding by acetylsalicylic acid, caffeic acid, and α-phenyl-N-t-butylnitrone: Implications for bioactivation by prostaglandin synthetase. Toxicology and Applied Pharmacology. 97(2). 192–202. 55 indexed citations

19.

Turner, Christopher J., et al.. (1988). A high-performance liquid chromatographie assay for reduced and oxidised glutathione in embryonic, neonatal, and adult tissues using a porous graphite electrochemical detector. Journal of Pharmacological Methods. 19(1). 75–83. 33 indexed citations

20.

Wells, Peter G., et al.. (1988). Metabolic evidence for the involvement of enzymatic bioactivation in the cataractogenicity of acetaminophen in genetically susceptible (C57BL6) and resistant (DBA2) murine strains. Toxicology and Applied Pharmacology. 94(3). 487–495. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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