Paul R. Stabach
- Molecular Biology top 10%
- Cell Biology top 2%
- Physiology top 5%
- Cellular and Molecular Neuroscience top 5%
- Immunology top 10%
- Co-authors
- Jon S. MorrowPrasad DevarajanMichael C. StankewichMaria Antonietta De MatteisThomas ArditoMichael KashgarianAndrea MannCarol D. Cianci
- Topics
- Erythrocyte Function and Pathophysiology (14 papers)Cellular transport and secretion (6 papers)Dermatological and Skeletal Disorders (5 papers)
- Cited by
- Cell BiologyPhysiology
- Journals
- Proceedings of the National Academy of SciencesJournal of Biological ChemistryThe Journal of Cell Biology
- Partner nations
- United StatesItalyGermany
In The Last Decade
Paul R. Stabach
37 papers receiving 1.6k citations
Peers
Comparison fields: 5 of 95
- Molecular Biology 843
- Cell Biology 516
- Physiology 507
- Cellular and Molecular Neuroscience 274
- Immunology 227
Countries citing papers authored by Paul R. Stabach
This map shows the geographic impact of Paul R. Stabach's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Paul R. Stabach with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Paul R. Stabach more than expected).
Fields of papers citing papers by Paul R. Stabach
This network shows the impact of papers produced by Paul R. Stabach. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Paul R. Stabach. The network helps show where Paul R. Stabach may publish in the future.
Co-authorship network of co-authors of Paul R. Stabach
This figure shows the co-authorship network connecting the top 25 collaborators of Paul R. Stabach. A scholar is included among the top collaborators of Paul R. Stabach based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Paul R. Stabach. Paul R. Stabach is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 3 | |
| 3 | 4 | |
| 4 | 41 | |
| 5 | 14 | |
| 6 | 13 | |
| 7 | 17 | |
| 8 | 35 | |
| 9 | 61 | |
| 10 | 120 | |
| 11 | 14 | |
| 12 | 33 | |
| 13 | 19 | |
| 14 | 34 | |
| 15 | 11 | |
| 16 | 28 | |
| 17 | 61 | |
| 18 | 72 | |
| 19 | 1 | |
| 20 | 8 |
About Paul R. Stabach
Paul R. Stabach is a scholar working on Cell Biology, Physiology and Immunology, having authored 38 papers that have together received 1.6k indexed citations. Recurring topics across this work include Erythrocyte Function and Pathophysiology (14 papers), Cellular transport and secretion (6 papers) and Dermatological and Skeletal Disorders (5 papers). The work is most often cited by research in Cell Biology (516 citations), Physiology (507 citations) and Physiology (74 citations). Paul R. Stabach has collaborated with scholars based in United States, Italy and Germany. Frequent co-authors include Jon S. Morrow, Prasad Devarajan, Michael C. Stankewich, Maria Antonietta De Matteis, Thomas Ardito, Michael Kashgarian, Andrea Mann, Carol D. Cianci, Demetrios T. Braddock and William M. Philbrick. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The Journal of Cell Biology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.