Joseph A. Madri

25.5k total citations · 4 hit papers
241 papers, 21.0k citations indexed

About

Joseph A. Madri is a scholar working on Immunology and Allergy, Molecular Biology and Cancer Research. According to data from OpenAlex, Joseph A. Madri has authored 241 papers receiving a total of 21.0k indexed citations (citations by other indexed papers that have themselves been cited), including 107 papers in Immunology and Allergy, 106 papers in Molecular Biology and 58 papers in Cancer Research. Recurrent topics in Joseph A. Madri's work include Cell Adhesion Molecules Research (107 papers), Angiogenesis and VEGF in Cancer (60 papers) and Protease and Inhibitor Mechanisms (44 papers). Joseph A. Madri is often cited by papers focused on Cell Adhesion Molecules Research (107 papers), Angiogenesis and VEGF in Cancer (60 papers) and Protease and Inhibitor Mechanisms (44 papers). Joseph A. Madri collaborates with scholars based in United States, Germany and Switzerland. Joseph A. Madri's co-authors include Bruce M. Pratt, Heinz Furthmayr, William C. Sessa, Guillermo García‐Cardeña, Tara L. Haas, Andreas Papapetropoulos, Stuart K. Williams, Adeline Tucker, Sandra Davis and Donald E. Ingber and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Joseph A. Madri

241 papers receiving 20.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Nitric oxide production contributes to the angiogenic properties of vascular endothelial growth factor in human endothelial cells.

1997 993 citations Joseph A. Madri et al. Journal of Clinical Investigation profile →
Surface expression of alpha 4 integrin by CD4 T cells is required for their entry into brain parenchyma.

1993 704 citations Joseph A. Madri, Nancy H. Ruddle et al. The Journal of Experimental Medicine profile →
Shapes, domain organizations and flexibility of laminin and fibronectin, two multifunctional proteins of the extracellular matrix

1981 556 citations Joseph A. Madri, Heinz Furthmayr et al. profile →
Capillary endothelial cell cultures: phenotypic modulation by matrix components.

1983 511 citations Joseph A. Madri et al. The Journal of Cell Biology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Joseph A. Madri United States	85	8.6k	5.5k	3.6k	3.3k	2.9k	241	21.0k
Elaine W. Raines United States	72	10.4k 1.2×	3.6k 0.6×	3.4k 1.0×	5.2k 1.6×	2.5k 0.9×	142	23.3k
Deane F. Mosher United States	79	7.9k 0.9×	6.9k 1.2×	4.7k 1.3×	2.2k 0.7×	4.4k 1.5×	314	20.3k
Ann M. Dvořàk United States	87	13.7k 1.6×	4.8k 0.9×	4.4k 1.2×	8.4k 2.6×	3.2k 1.1×	330	30.5k
Paul Börnstein United States	93	13.8k 1.6×	4.8k 0.9×	5.4k 1.5×	1.8k 0.6×	4.2k 1.5×	277	27.2k
Jean‐Michel Foidart Belgium	68	5.7k 0.7×	3.5k 0.6×	3.8k 1.1×	2.8k 0.9×	2.0k 0.7×	345	17.4k
Ismo Virtanen Finland	75	8.3k 1.0×	4.7k 0.9×	2.0k 0.6×	2.3k 0.7×	5.1k 1.8×	480	20.7k
M. Luisa Iruela‐Arispe United States	82	12.5k 1.5×	2.2k 0.4×	5.0k 1.4×	2.8k 0.8×	3.2k 1.1×	202	21.4k
Jack Lawler United States	77	11.8k 1.4×	2.6k 0.5×	4.7k 1.3×	2.3k 0.7×	2.6k 0.9×	206	20.2k
Karl Tryggvason Sweden	96	15.1k 1.8×	9.8k 1.8×	6.8k 1.9×	4.1k 1.2×	4.9k 1.7×	384	34.3k
Bjørn R. Olsen United States	80	14.7k 1.7×	4.0k 0.7×	4.9k 1.4×	1.3k 0.4×	2.7k 1.0×	277	26.7k

Countries citing papers authored by Joseph A. Madri

Since Specialization

Citations

This map shows the geographic impact of Joseph A. Madri's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joseph A. Madri with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joseph A. Madri more than expected).

Fields of papers citing papers by Joseph A. Madri

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joseph A. Madri. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joseph A. Madri. The network helps show where Joseph A. Madri may publish in the future.

Co-authorship network of co-authors of Joseph A. Madri

This figure shows the co-authorship network connecting the top 25 collaborators of Joseph A. Madri. A scholar is included among the top collaborators of Joseph A. Madri based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joseph A. Madri. Joseph A. Madri is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

1.

Means, Robert E., et al.. (2020). A Static Self-Directed Method for Generating Brain Organoids from Human Embryonic Stem Cells. Journal of Visualized Experiments. 3 indexed citations

2.

Wu, Yue, Michael Hannigan, Lijun Zhan, Joseph A. Madri, & Chi‐Kuang Huang. (2016). NOD Mice Having a Lyn Tyrosine Kinase Mutation Exhibit Abnormal Neutrophil Chemotaxis. Journal of Cellular Physiology. 232(7). 1689–1695. 2 indexed citations

3.

Lee, Chunsik, Anguo Liu, Alba Miranda-Ribera, et al.. (2014). NEU1 Sialidase Regulates the Sialylation State of CD31 and Disrupts CD31-driven Capillary-like Tube Formation in Human Lung Microvascular Endothelia. Journal of Biological Chemistry. 289(13). 9121–9135. 59 indexed citations

4.

Li, Qi, Michael Michaud, Sandra Canosa, Andrew Kuo, & Joseph A. Madri. (2011). GSK-3β: a signaling pathway node modulating neural stem cell and endothelial cell interactions. Angiogenesis. 14(2). 173–185. 27 indexed citations

5.

Naito, Yuji, Daniel R. Duncan, Spencer N. Church, et al.. (2011). Characterization of the Natural History of Extracellular Matrix Production in Tissue-Engineered Vascular Grafts during Neovessel Formation. Cells Tissues Organs. 195(1-2). 60–72. 56 indexed citations

6.

Wu, Yue, et al.. (2009). Bone Marrow Monocyte PECAM-1 Deficiency Elicits Increased Osteoclastogenesis Resulting in Trabecular Bone Loss. The Journal of Immunology. 182(5). 2672–2679. 16 indexed citations

7.

Wang, Penghua, Jianfeng Dai, Fengwei Bai, et al.. (2008). Matrix Metalloproteinase 9 Facilitates West Nile Virus Entry into the Brain. Journal of Virology. 82(18). 8978–8985. 149 indexed citations

8.

Kim, Ji‐il, et al.. (2008). Differential Effects of Shear Stress and Cyclic Strain on Sp1 Phosphorylation by Protein Kinase Cζ Modulates Membrane Type 1–Matrix Metalloproteinase in Endothelial Cells. Endothelium. 15(1-2). 33–42. 14 indexed citations

9.

Chyou, Susan, Eric H. Ekland, April C. Carpenter, et al.. (2008). Fibroblast-Type Reticular Stromal Cells Regulate the Lymph Node Vasculature. The Journal of Immunology. 181(6). 3887–3896. 105 indexed citations

10.

Wu, Yue, Paul R. Stabach, Michael Michaud, & Joseph A. Madri. (2005). Neutrophils Lacking Platelet-Endothelial Cell Adhesion Molecule-1 Exhibit Loss of Directionality and Motility in CXCR2-Mediated Chemotaxis. The Journal of Immunology. 175(6). 3484–3491. 34 indexed citations

11.

Hallaq, Haifa, Emese Pinter, Josephine Enciso, et al.. (2004). A null mutation of Hhex results in abnormal cardiac development,defective vasculogenesis and elevated Vegfa levels. Development. 131(20). 5197–5209. 82 indexed citations

12.

Biswas, Purba, Sandra Canosa, Jonathan D. Schoenfeld, et al.. (2003). PECAM-1 promotes β-catenin accumulation and stimulates endothelial cell proliferation. Biochemical and Biophysical Research Communications. 303(1). 212–218. 36 indexed citations

13.

Graesser, Donnasue, Emily K. Osterweil, Amy E. Juedes, et al.. (2002). Altered vascular permeability and early onset of experimental autoimmune encephalomyelitis in PECAM-1–deficient mice. Journal of Clinical Investigation. 109(3). 383–392. 256 indexed citations

14.

Graesser, Donnasue, Emily K. Osterweil, Amy E. Juedes, et al.. (2002). Altered vascular permeability and early onset of experimental autoimmune encephalomyelitis in PECAM-1–deficient mice. Journal of Clinical Investigation. 109(3). 383–392. 242 indexed citations

15.

Ilan, Neta & Joseph A. Madri. (1999). New paradigms of signaling in the vasculature: ephrins and metalloproteases. Current Opinion in Biotechnology. 10(6). 536–540. 13 indexed citations

16.

Madri, Joseph A., et al.. (1992). Endothelial cells interact with the core protein of basement membrane perlecan through beta 1 and beta 3 integrins: an adhesion modulated by glycosaminoglycan.. The Journal of Cell Biology. 119(4). 945–959. 153 indexed citations

17.

Merwin, June Rae, Anita B. Roberts, Paturu Kondaiah, Adeline Tucker, & Joseph A. Madri. (1991). Vascular Cell Responses to TGF-β ₃ Mimic Those of TGF-β ₁ in vitro. Growth Factors. 5(2). 149–158. 29 indexed citations

18.

Murray, Joseph S., Joseph A. Madri, J P Tite, Simon R. Carding, & Kim Bottomly. (1989). MHC control of CD4+ T cell subset activation.. The Journal of Experimental Medicine. 170(6). 2135–2140. 103 indexed citations

19.

Keller, R., J E Silbert, Heinz Furthmayr, & Joseph A. Madri. (1987). Aortic endothelial cell proteoheparan sulfate. I. Isolation and characterization of plasmamembrane-associated and extracellular species.. PubMed. 128(2). 286–98. 28 indexed citations

20.

Keller, R., Bruce M. Pratt, Heinz Furthmayr, & Joseph A. Madri. (1987). Aortic endothelial cell proteoheparan sulfate. II. Modulation by extracellular matrix.. PubMed. 128(2). 299–306. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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