Fiona J. Hemming

2.6k total citations
33 papers, 2.1k citations indexed

About

Fiona J. Hemming is a scholar working on Molecular Biology, Cell Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Fiona J. Hemming has authored 33 papers receiving a total of 2.1k indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 15 papers in Cell Biology and 7 papers in Cellular and Molecular Neuroscience. Recurrent topics in Fiona J. Hemming's work include Extracellular vesicles in disease (7 papers), Proteoglycans and glycosaminoglycans research (7 papers) and Cellular transport and secretion (6 papers). Fiona J. Hemming is often cited by papers focused on Extracellular vesicles in disease (7 papers), Proteoglycans and glycosaminoglycans research (7 papers) and Cellular transport and secretion (6 papers). Fiona J. Hemming collaborates with scholars based in France, Switzerland and Belgium. Fiona J. Hemming's co-authors include Rémy Sadoul, Mathilde Chivet, Béatrice Blot, Karin Pernet‐Gallay, Charlotte Javalet, Paul Dubois, Sandrine Fraboulet, Karine Laulagnier, Yves Goldberg and Agnès Belly and has published in prestigious journals such as Science, Journal of Neuroscience and Scientific Reports.

In The Last Decade

Fiona J. Hemming

33 papers receiving 2.0k citations

Peers

Fiona J. Hemming
Cees B.M. Oudejans Netherlands
Lawrence C. Kenyon United States
Toni R. Prezant United States
Masao Kishikawa Japan
Daniel Bilbao United States
Jin He United States
Siu‐Pok Yee United States
Lisbeth S. Laursen Denmark
Mordechai Shohat Israel
Koichi Miyake Japan
Cees B.M. Oudejans Netherlands
Fiona J. Hemming
Citations per year, relative to Fiona J. Hemming Fiona J. Hemming (= 1×) peers Cees B.M. Oudejans

Countries citing papers authored by Fiona J. Hemming

Since Specialization
Citations

This map shows the geographic impact of Fiona J. Hemming's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Fiona J. Hemming with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Fiona J. Hemming more than expected).

Fields of papers citing papers by Fiona J. Hemming

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Fiona J. Hemming. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Fiona J. Hemming. The network helps show where Fiona J. Hemming may publish in the future.

Co-authorship network of co-authors of Fiona J. Hemming

This figure shows the co-authorship network connecting the top 25 collaborators of Fiona J. Hemming. A scholar is included among the top collaborators of Fiona J. Hemming based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Fiona J. Hemming. Fiona J. Hemming is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Guo, Hui‐Shan, Charlotte Javalet, Fiona J. Hemming, et al.. (2017). Atg5 Disassociates the V1V0-ATPase to Promote Exosome Production and Tumor Metastasis Independent of Canonical Macroautophagy. Developmental Cell. 43(6). 716–730.e7. 227 indexed citations
2.
Sadoul, Rémy, Marine H. Laporte, Romain Chassefeyre, et al.. (2017). The role of ESCRT during development and functioning of the nervous system. Seminars in Cell and Developmental Biology. 74. 40–49. 26 indexed citations
3.
Laporte, Marine H., Fiona J. Hemming, Jean‐Christophe Deloulme, et al.. (2017). Alix is required during development for normal growth of the mouse brain. Scientific Reports. 7(1). 44767–44767. 26 indexed citations
4.
Laulagnier, Karine, Charlotte Javalet, Fiona J. Hemming, et al.. (2017). Amyloid precursor protein products concentrate in a subset of exosomes specifically endocytosed by neurons. Cellular and Molecular Life Sciences. 75(4). 757–773. 135 indexed citations
5.
Laulagnier, Karine, Charlotte Javalet, Fiona J. Hemming, & Rémy Sadoul. (2016). Purification and Analysis of Exosomes Released by Mature Cortical Neurons Following Synaptic Activation. Methods in molecular biology. 1545. 129–138. 19 indexed citations
6.
Chivet, Mathilde, Charlotte Javalet, Fiona J. Hemming, et al.. (2013). Exosomes as a novel way of interneuronal communication. Biochemical Society Transactions. 41(1). 241–244. 107 indexed citations
7.
Chivet, Mathilde, Fiona J. Hemming, Karin Pernet‐Gallay, Sandrine Fraboulet, & Rémy Sadoul. (2012). Emerging Role of Neuronal Exosomes in the Central Nervous System. Frontiers in Physiology. 3. 145–145. 178 indexed citations
8.
Pernet‐Gallay, Karin, Mathilde Chivet, Fiona J. Hemming, et al.. (2010). Release of exosomes from differentiated neurons and its regulation by synaptic glutamatergic activity. Molecular and Cellular Neuroscience. 46(2). 409–418. 470 indexed citations
9.
Mahul‐Mellier, Anne‐Laure, Fiona J. Hemming, Béatrice Blot, Sandrine Fraboulet, & Rémy Sadoul. (2006). Alix, Making a Link between Apoptosis-Linked Gene-2, the Endosomal Sorting Complexes Required for Transport, and Neuronal DeathIn Vivo. Journal of Neuroscience. 26(2). 542–549. 75 indexed citations
10.
Hemming, Fiona J., Sandrine Fraboulet, Béatrice Blot, & Rémy Sadoul. (2004). Early increase of apoptosis-linked gene-2 interacting protein X in areas of kainate-induced neurodegeneration. Neuroscience. 123(4). 887–895. 24 indexed citations
11.
Blum, David, Fiona J. Hemming, Marie‐Christine Galas, et al.. (2004). Increased Alix (apoptosis-linked gene-2 interacting protein X) immunoreactivity in the degenerating striatum of rats chronically treated by 3-nitropropionic acid. Neuroscience Letters. 368(3). 309–313. 21 indexed citations
12.
Fraboulet, Sandrine, et al.. (2003). Expression of Alix protein during early avian development. Gene Expression Patterns. 3(2). 139–142. 6 indexed citations
13.
Saxod, Raymond, et al.. (2002). Chondroitin and keratan sulfates have opposing effects on attachment and outgrowth of ventral mesencephalic explants in culture. Journal of Neuroscience Research. 70(1). 46–56. 21 indexed citations
14.
15.
Hemming, Fiona J. & Raymond Saxod. (1998). Regulated expression of keratan sulphate and peanut agglutinin binding sites during organogenesis in the developing chick. Histochemistry and Cell Biology. 110(2). 189–200. 3 indexed citations
16.
Hemming, Fiona J., et al.. (1998). Transient compartmental expression of neurocan in the developing striatum of the rat. Journal of Neuroscience Research. 51(5). 612–618. 15 indexed citations
17.
18.
Hemming, Fiona J., et al.. (1994). Development of sensory innervation in chick skin: comparison of nerve fibre and chondroitin sulphate distributions in vivo and in vitro. Cell and Tissue Research. 277(3). 519–529. 10 indexed citations
19.
Dubois, Paul & Fiona J. Hemming. (1991). Fetal development and regulation of pituitary cell types. Journal of Electron Microscopy Technique. 19(1). 2–20. 28 indexed citations
20.
Hemming, Fiona J., Michel L. Aubert, & Paul Dubois. (1988). Differentiation of Fetal Rat Somatotropesin Vitro: Effects of Cortisol, 3,5,3′-Triiodothyronine, and Glucagon, a Light Microscopic and Radioimmunological Study*. Endocrinology. 123(3). 1230–1236. 42 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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