Patrick M. Navolanic

4.4k total citations · 3 hit papers
23 papers, 3.5k citations indexed

About

Patrick M. Navolanic is a scholar working on Molecular Biology, Oncology and Organic Chemistry. According to data from OpenAlex, Patrick M. Navolanic has authored 23 papers receiving a total of 3.5k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 12 papers in Oncology and 6 papers in Organic Chemistry. Recurrent topics in Patrick M. Navolanic's work include Synthesis and biological activity (6 papers), PI3K/AKT/mTOR signaling in cancer (6 papers) and Melanoma and MAPK Pathways (5 papers). Patrick M. Navolanic is often cited by papers focused on Synthesis and biological activity (6 papers), PI3K/AKT/mTOR signaling in cancer (6 papers) and Melanoma and MAPK Pathways (5 papers). Patrick M. Navolanic collaborates with scholars based in United States, Italy and Ukraine. Patrick M. Navolanic's co-authors include James A. McCubrey, Linda S. Steelman, Richard A. Franklin, William L. Blalock, J G Shelton, Fuju Chang, Massimo Libra, John Tayu Lee, Fumin Chang and Franca Stivala and has published in prestigious journals such as Oncogene, Biology of Reproduction and Journal of Clinical Pathology.

In The Last Decade

Patrick M. Navolanic

23 papers receiving 3.5k citations

Hit Papers

Involvement of PI3K/Akt pathway in cell cycle progression... 2003 2026 2010 2018 2003 2003 2006 250 500 750

Peers

Patrick M. Navolanic
Comparison fields: 5 of 114
  • Molecular Biology 2.3k
  • Oncology 1.0k
  • Cancer Research 562
  • Pathology and Forensic Medicine 403
  • Pulmonary and Respiratory Medicine 371
Replace Fumin Chang with:
Fumin Chang United States
Jörg Bäsecke Germany
William H. Chappell United States
Narayan Shivapurkar United States
Suzanne Hagan United Kingdom
William L. Blalock Italy
Olivier Coqueret France
John Brognard United States
Jingxuan Pan China
Juan Fernando Martínez-Leal Spain
Fumin Chang United States View profile →
Citations per field, relative to Patrick M. Navolanic
Patrick M. Navolanic · 1×
Citations per year, relative to Patrick M. Navolanic
Patrick M. Navolanic · 1×

Countries citing papers authored by Patrick M. Navolanic

Since Specialization
Citations

This map shows the geographic impact of Patrick M. Navolanic's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Patrick M. Navolanic with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Patrick M. Navolanic more than expected).

Fields of papers citing papers by Patrick M. Navolanic

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Patrick M. Navolanic. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Patrick M. Navolanic. The network helps show where Patrick M. Navolanic may publish in the future.

Co-authorship network of co-authors of Patrick M. Navolanic

This figure shows the co-authorship network connecting the top 25 collaborators of Patrick M. Navolanic. A scholar is included among the top collaborators of Patrick M. Navolanic based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Patrick M. Navolanic. Patrick M. Navolanic is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 70
2 130
3 5
4 67
5
Roles of the RAF/MEK/ERK and PI3K/PTEN/AKT pathways in malignant transformation and drug resistance breakdown →
550
6 21
7 83
8 8
9 104
10 13
11 25
12 50
13
Signal transduction mediated by the Ras/Raf/MEK/ERK pathway from cytokine receptors to transcription factors: potential targeting for therapeutic intervention breakdown →
577
14
Involvement of PI3K/Akt pathway in cell cycle progression, apoptosis, and neoplastic transformation: a target for cancer chemotherapy breakdown →
999
15 69
16
Raf-1 and Bcl-2 induce distinct and common pathways that contribute to breast cancer drug resistance.
96
17 150
18
Regulation of cell cycle progression and apoptosis by the Ras/Raf/MEK/ERK pathway (Review).
332
19
Role of the Raf signal transduction cascade in the in vitro resistance to the anticancer drug doxorubicin.
72
20 52

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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