Pasquale Nigro
- Physiology top 10%
- Molecular Biology
- Epidemiology
- Cardiology and Cardiovascular Medicine
- Rehabilitation top 10%
- Co-authors
- Angelo CignarelliFrancesco GiorginoSebastio PerriniLaurie J. GoodyearRomina FicarellaRoeland J.W. MiddelbeekLuigi LaviolaAnnalisa Natalicchio
- Topics
- Adipose Tissue and Metabolism (16 papers)Adipokines, Inflammation, and Metabolic Diseases (6 papers)Exercise and Physiological Responses (4 papers)
- Journals
- PLoS ONECell MetabolismDiabetes
- Partner nations
- United StatesItalyFinland
In The Last Decade
Pasquale Nigro
15 papers receiving 364 citations
Peers
Comparison fields: 5 of 63
- Physiology 219
- Molecular Biology 106
- Epidemiology 104
- Cardiology and Cardiovascular Medicine 57
- Rehabilitation 42
Countries citing papers authored by Pasquale Nigro
This map shows the geographic impact of Pasquale Nigro's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pasquale Nigro with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pasquale Nigro more than expected).
Fields of papers citing papers by Pasquale Nigro
This network shows the impact of papers produced by Pasquale Nigro. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pasquale Nigro. The network helps show where Pasquale Nigro may publish in the future.
Co-authorship network of co-authors of Pasquale Nigro
This figure shows the co-authorship network connecting the top 25 collaborators of Pasquale Nigro. A scholar is included among the top collaborators of Pasquale Nigro based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pasquale Nigro. Pasquale Nigro is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 4 | |
| 3 | 2 | |
| 4 | 24 | |
| 5 | 65 | |
| 6 | 7 | |
| 7 | 0 | |
| 8 | 22 | |
| 9 | 38 | |
| 10 | 9 | |
| 11 | 46 | |
| 12 | 5 | |
| 13 | 52 | |
| 14 | 14 | |
| 15 | 48 | |
| 16 | 31 | |
| 17 | Insulin Detemir Induced less Adipogenesis and less Lipid Droplets Accumulation than Human Insulin in Human Adipose Stem Cells | 1 |
About Pasquale Nigro
Pasquale Nigro is a scholar working on Physiology, Rehabilitation and Geriatrics and Gerontology, having authored 17 papers that have together received 368 indexed citations. Recurring topics across this work include Adipose Tissue and Metabolism (16 papers), Adipokines, Inflammation, and Metabolic Diseases (6 papers) and Exercise and Physiological Responses (4 papers). The work is most often cited by research in Physiology (219 citations), Geriatrics and Gerontology (26 citations) and Rehabilitation (42 citations). Pasquale Nigro has collaborated with scholars based in United States, Italy and Finland. Frequent co-authors include Angelo Cignarelli, Francesco Giorgino, Sebastio Perrini, Laurie J. Goodyear, Romina Ficarella, Roeland J.W. Middelbeek, Luigi Laviola, Annalisa Natalicchio, Alessandro Peschechera and Michael F. Hirshman. Their work appears in journals such as PLoS ONE, Cell Metabolism and Diabetes.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.