Laurie J. Goodyear

42.3k total citations · 11 hit papers
245 papers, 31.8k citations indexed

About

Laurie J. Goodyear is a scholar working on Molecular Biology, Physiology and Surgery. According to data from OpenAlex, Laurie J. Goodyear has authored 245 papers receiving a total of 31.8k indexed citations (citations by other indexed papers that have themselves been cited), including 183 papers in Molecular Biology, 163 papers in Physiology and 73 papers in Surgery. Recurrent topics in Laurie J. Goodyear's work include Adipose Tissue and Metabolism (151 papers), Metabolism, Diabetes, and Cancer (140 papers) and Pancreatic function and diabetes (72 papers). Laurie J. Goodyear is often cited by papers focused on Adipose Tissue and Metabolism (151 papers), Metabolism, Diabetes, and Cancer (140 papers) and Pancreatic function and diabetes (72 papers). Laurie J. Goodyear collaborates with scholars based in United States, Denmark and Japan. Laurie J. Goodyear's co-authors include Michael F. Hirshman, Nicolas Musi, Nobuharu Fujii, Tatsuya Hayashi, Kristin I. Stanford, David E. Moller, Barbara B. Kahn, Gaochao Zhou, Jørgen F. P. Wojtaszewski and Kei Sakamoto and has published in prestigious journals such as Science, New England Journal of Medicine and Cell.

In The Last Decade

Laurie J. Goodyear

236 papers receiving 31.3k citations

Hit Papers

5 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Role of AMP-activated protein kinase in mechanism of metformin action

2001 4.5k citations Gaochao Zhou, Robert P. Myers et al. Journal of Clinical Investigation profile →
Free fatty acid-induced insulin resistance is associated with activation of protein kinase C theta and alterations in the insulin signaling cascade.

1999 955 citations Laurie J. Goodyear et al. Diabetes profile →
A Muscle-Specific Insulin Receptor Knockout Exhibits Features of the Metabolic Syndrome of NIDDM without Altering Glucose Tolerance

1998 939 citations Domenico Accili, Laurie J. Goodyear et al. profile →
Brown adipose tissue regulates glucose homeostasis and insulin sensitivity

2012 918 citations Kristin I. Stanford, Roeland J.W. Middelbeek et al. Journal of Clinical Investigation profile →
Exercise, Glucose Transport, and Insulin Sensitivity

1998 805 citations Laurie J. Goodyear et al. profile →
Metformin Increases AMP-Activated Protein Kinase Activity in Skeletal Muscle of Subjects With Type 2 Diabetes

2002 672 citations Nicolas Musi, Michael F. Hirshman et al. Diabetes profile →
Evidence for 5′AMP-Activated Protein Kinase Mediation of the Effect of Muscle Contraction on Glucose Transport

1998 638 citations Michael F. Hirshman, Laurie J. Goodyear et al. Diabetes profile →
Restoring Systemic GDF11 Levels Reverses Age-Related Dysfunction in Mouse Skeletal Muscle

2014 621 citations Manisha Sinha, Young C. Jang et al. Science profile →
Targeted disruption of the glucose transporter 4 selectively in muscle causes insulin resistance and glucose intolerance

2000 612 citations Franck Mauvais‐Jarvis, Jørgen F. P. Wojtaszewski et al. profile →
5' AMP-activated protein kinase activation causes GLUT4 translocation in skeletal muscle.

1999 589 citations Michael F. Hirshman, Laurie J. Goodyear et al. Diabetes profile →
Ampk phosphorylation of Ulk1 is required for targeting of mitochondria to lysosomes in exercise-induced mitophagy

2017 384 citations Laurie J. Goodyear et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Laurie J. Goodyear United States	90	20.0k	14.8k	7.6k	4.9k	4.5k	245	31.8k
Juleen R. Zierath Sweden	94	16.7k 0.8×	14.4k 1.0×	4.4k 0.6×	3.4k 0.7×	2.9k 0.6×	366	29.9k
Michael F. Hirshman United States	73	14.3k 0.7×	9.3k 0.6×	6.0k 0.8×	3.1k 0.6×	3.3k 0.7×	155	21.3k
Neil B. Ruderman United States	97	14.1k 0.7×	13.6k 0.9×	4.9k 0.6×	6.6k 1.3×	5.1k 1.1×	248	29.6k
Pere Puigserver United States	72	25.8k 1.3×	22.3k 1.5×	3.5k 0.5×	8.6k 1.8×	2.4k 0.5×	121	43.8k
Gregory R. Steinberg Canada	78	12.3k 0.6×	8.8k 0.6×	4.7k 0.6×	6.2k 1.3×	3.3k 0.7×	246	23.5k
David Carling United Kingdom	85	28.9k 1.4×	11.6k 0.8×	11.9k 1.6×	8.2k 1.7×	5.3k 1.2×	183	40.7k
Jason K. Kim United States	82	13.5k 0.7×	9.4k 0.6×	3.6k 0.5×	6.6k 1.3×	3.7k 0.8×	197	25.4k
Benoı̂t Viollet France	98	27.2k 1.4×	9.5k 0.6×	9.7k 1.3×	10.8k 2.2×	5.3k 1.2×	344	42.4k
Jørgen F. P. Wojtaszewski Denmark	79	11.3k 0.6×	10.2k 0.7×	4.3k 0.6×	2.0k 0.4×	1.8k 0.4×	262	18.4k
Antonio Vidal‐Puig United Kingdom	86	12.2k 0.6×	12.8k 0.9×	2.6k 0.3×	7.4k 1.5×	2.5k 0.6×	313	26.0k

Countries citing papers authored by Laurie J. Goodyear

Since Specialization

Citations

This map shows the geographic impact of Laurie J. Goodyear's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Laurie J. Goodyear with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Laurie J. Goodyear more than expected).

Fields of papers citing papers by Laurie J. Goodyear

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Laurie J. Goodyear. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Laurie J. Goodyear. The network helps show where Laurie J. Goodyear may publish in the future.

Co-authorship network of co-authors of Laurie J. Goodyear

This figure shows the co-authorship network connecting the top 25 collaborators of Laurie J. Goodyear. A scholar is included among the top collaborators of Laurie J. Goodyear based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Laurie J. Goodyear. Laurie J. Goodyear is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Corvera, Silvia, Akhila Rajan, Kristy L. Townsend, et al.. (2025). Advances in Adipose Tissue Biology. Endocrine Reviews. 47(1). 75–92.

Hevener, Andrea L., Laurie J. Goodyear, Sue C. Bodine, et al.. (2025). Exercise training remodels inter-organ endocrine networks. Molecular Metabolism. 99. 102219–102219.

Alves-Wagner, Ana Bárbara, et al.. (2025). Grandmaternal exercise improves the metabolic health of second-generation offspring generated from F1 females. Journal of sport and health science. 15. 101096–101096.

Collier, David N., James DeVente, Tomoko Kaneko-Tarui, et al.. (2024). Paternal obesity decreases infant MSC mitochondrial functional capacity. American Journal of Physiology-Endocrinology and Metabolism. 327(4). E441–E448. 4 indexed citations

Alves-Wagner, Ana Bárbara, Joji Kusuyama, Pasquale Nigro, et al.. (2022). Grandmaternal exercise improves metabolic health of second-generation offspring. Molecular Metabolism. 60. 101490–101490. 7 indexed citations

Hernández‐Saavedra, Diego, Christina A. Markunas, Hirokazu Takahashi, et al.. (2022). Maternal Exercise and Paternal Exercise Induce Distinct Metabolite Signatures in Offspring Tissues. Diabetes. 71(10). 2094–2105. 10 indexed citations

Nigro, Pasquale, Roeland J.W. Middelbeek, Christiano R. R. Alves, et al.. (2021). Exercise Training Promotes Sex-Specific Adaptations in Mouse Inguinal White Adipose Tissue. Diabetes. 70(6). 1250–1264. 22 indexed citations

Wang, Guoxiao, Yingying Yu, Weikang Cai, et al.. (2020). Muscle-Specific Insulin Receptor Overexpression Protects Mice From Diet-Induced Glucose Intolerance but Leads to Postreceptor Insulin Resistance. Diabetes. 69(11). 2294–2309. 9 indexed citations

Pinckard, Kelsey M., Lisa A. Baer, Peter J. Arts, et al.. (2020). Exercise-induced 3′-sialyllactose in breast milk is a critical mediator to improve metabolic health and cardiac function in mouse offspring. Nature Metabolism. 2(8). 678–687. 51 indexed citations

10.

Teuho, Jarmo, Teemu Saari, Kirsi A. Virtanen, et al.. (2019). Exercise training alters lipoprotein particles independent of brown adipose tissue metabolic activity. Obesity Science & Practice. 5(3). 258–272. 5 indexed citations

11.

Ernande, Laura, Kristin I. Stanford, Robrecht Thoonen, et al.. (2016). Relationship of brown adipose tissue perfusion and function: a study through β2-adrenoreceptor stimulation. Journal of Applied Physiology. 120(8). 825–832. 16 indexed citations

12.

Mul, Joram D., Kristin I. Stanford, Michael F. Hirshman, & Laurie J. Goodyear. (2015). Exercise and Regulation of Carbohydrate Metabolism. Progress in molecular biology and translational science. 135. 17–37. 97 indexed citations

13.

Sinha, Manisha, Young C. Jang, Juhyun Oh, et al.. (2014). Restoring Systemic GDF11 Levels Reverses Age-Related Dysfunction in Mouse Skeletal Muscle. Science. 344(6184). 649–652. 621 indexed citations breakdown →

14.

Treebak, Jonas T., Christian Pehmøller, Jonas M. Kristensen, et al.. (2013). Acute exercise and physiological insulin induce distinct phosphorylation signatures on TBC1D1 and TBC1D4 proteins in human skeletal muscle. The Journal of Physiology. 592(2). 351–375. 92 indexed citations

15.

Witczak, Carol A., Niels Jessen, Tarō Toyoda, et al.. (2010). CaMKII regulates contraction- but not insulin-induced glucose uptake in mouse skeletal muscle. American Journal of Physiology-Endocrinology and Metabolism. 298(6). E1150–E1160. 67 indexed citations

16.

Luo, Ji, et al.. (2006). Loss of class IA PI3K signaling in muscle leads to impaired muscle growth, insulin response, and hyperlipidemia. Cell Metabolism. 3(5). 355–366. 97 indexed citations

17.

Norris, Andrew W., Lihong Chen, Simon J. Fisher, et al.. (2003). Muscle-specific PPARγ-deficient mice develop increased adiposity and insulin resistance but respond to thiazolidinediones. Journal of Clinical Investigation. 112(4). 608–618. 354 indexed citations

18.

Mauvais‐Jarvis, Franck, Kohjiro Ueki, Michael F. Hirshman, et al.. (2002). Reduced expression of the murine p85α subunit of phosphoinositide 3-kinase improves insulin signaling and ameliorates diabetes. Journal of Clinical Investigation. 109(1). 141–149. 10 indexed citations

19.

Zhou, Gaochao, Robert P. Myers, Ying Li, et al.. (2001). Role of AMP-activated protein kinase in mechanism of metformin action. Journal of Clinical Investigation. 108(8). 1167–1174. 406 indexed citations

20.

Markuns, Jeffrey F., Jørgen F. P. Wojtaszewski, & Laurie J. Goodyear. (1999). Insulin and Exercise Decrease Glycogen Synthase Kinase-3 Activity by Different Mechanisms in Rat Skeletal Muscle. Journal of Biological Chemistry. 274(35). 24896–24900. 108 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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