P.A.L. Wight

3.5k total citations · 1 hit paper
102 papers, 2.8k citations indexed

About

P.A.L. Wight is a scholar working on Molecular Biology, Animal Science and Zoology and Epidemiology. According to data from OpenAlex, P.A.L. Wight has authored 102 papers receiving a total of 2.8k indexed citations (citations by other indexed papers that have themselves been cited), including 45 papers in Molecular Biology, 29 papers in Animal Science and Zoology and 14 papers in Epidemiology. Recurrent topics in P.A.L. Wight's work include Animal Nutrition and Physiology (26 papers), RNA Research and Splicing (18 papers) and RNA Interference and Gene Delivery (14 papers). P.A.L. Wight is often cited by papers focused on Animal Nutrition and Physiology (26 papers), RNA Research and Splicing (18 papers) and RNA Interference and Gene Delivery (14 papers). P.A.L. Wight collaborates with scholars based in United States, Italy and United Kingdom. P.A.L. Wight's co-authors include Karen J. Moore, G. Bulfield, W.G. Siller, Grace Mackenzie, Wendy B. Macklin, W. A. Dewar, R.B. Burns, B. Rothwell, Mark D. Crew and Cynthia S. Duchala and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and The Lancet.

In The Last Decade

P.A.L. Wight

100 papers receiving 2.7k citations

Hit Papers

X chromosome-linked muscular dystrophy (mdx) in the mouse. 1984 2026 1998 2012 1984 400 800 1.2k
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. X chromosome-linked muscular dystrophy (mdx) in the mouse.
    1984 1.4k citations P.A.L. Wight et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
P.A.L. Wight United States 21 1.9k 421 404 361 305 102 2.8k
Jason D. White Australia 28 1.4k 0.8× 307 0.7× 230 0.6× 264 0.7× 184 0.6× 69 2.1k
Yoshihide Ikeuchi Japan 26 1.2k 0.7× 489 1.2× 280 0.7× 107 0.3× 207 0.7× 78 2.0k
Luc Grobet Belgium 13 1.7k 0.9× 436 1.0× 264 0.7× 996 2.8× 73 0.2× 23 2.4k
Ryuichi Tatsumi Japan 28 2.3k 1.2× 769 1.8× 236 0.6× 215 0.6× 312 1.0× 88 3.3k
Koichi Ojima Japan 25 1.1k 0.6× 261 0.6× 355 0.9× 137 0.4× 219 0.7× 64 1.7k
D. R. Campion United States 22 744 0.4× 383 0.9× 773 1.9× 471 1.3× 69 0.2× 86 2.1k
Dominique Poncelet Belgium 9 1.9k 1.1× 338 0.8× 218 0.5× 939 2.6× 53 0.2× 15 2.4k
Sandra G. Velleman United States 31 1.6k 0.9× 452 1.1× 1.3k 3.3× 601 1.7× 119 0.4× 149 3.0k
D. Paulin France 33 2.2k 1.2× 217 0.5× 77 0.2× 397 1.1× 474 1.6× 75 3.6k
Benoît Brouwers Belgium 6 1.3k 0.7× 336 0.8× 225 0.6× 876 2.4× 49 0.2× 7 1.9k

Countries citing papers authored by P.A.L. Wight

Since Specialization
Citations

This map shows the geographic impact of P.A.L. Wight's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by P.A.L. Wight with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites P.A.L. Wight more than expected).

Fields of papers citing papers by P.A.L. Wight

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by P.A.L. Wight. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by P.A.L. Wight. The network helps show where P.A.L. Wight may publish in the future.

Co-authorship network of co-authors of P.A.L. Wight

This figure shows the co-authorship network connecting the top 25 collaborators of P.A.L. Wight. A scholar is included among the top collaborators of P.A.L. Wight based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with P.A.L. Wight. P.A.L. Wight is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Douglas, James C., et al.. (2021). Ethanol modulation of hippocampal neuroinflammation, myelination, and neurodevelopment in a postnatal mouse model of fetal alcohol spectrum disorders. Neurotoxicology and Teratology. 87. 107015–107015. 13 indexed citations
3.
Dobretsov, Maxim, Abdallah Hayar, Maxim Kozhemyakin, et al.. (2018). A Transgenic Mouse Model to Selectively Identify α3 Na,K-ATPase Expressing Cells in the Nervous System. Neuroscience. 398. 274–294. 9 indexed citations
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Johnson, Jennifer W., et al.. (2008). Proteomic analysis of nuclear factors binding to an intronic enhancer in the myelin proteolipid protein gene. Journal of Neurochemistry. 105(5). 1979–1995. 17 indexed citations
6.
Wight, P.A.L., Cynthia S. Duchala, H. Elizabeth Shick, Tatyana I. Gudz, & Wendy B. Macklin. (2006). Expression of a Myelin Proteolipid Protein (Plp)-lacZ Transgene is Reduced in both the CNS and PNS of Plp jp Mice. Neurochemical Research. 32(2). 343–351. 7 indexed citations
7.
Kokorina, Natalia A., et al.. (2004). Potentiation of myelin proteolipid protein (Plp) gene expression is mediated through AP‐1‐like binding sites. Journal of Neurochemistry. 90(6). 1500–1510. 9 indexed citations
8.
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Kokorina, Natalia A., et al.. (2000). Functional Characterization of a cis‐Acting DNA Antisilencer Region that Modulates Myelin Proteolipid Protein Gene Expression. Journal of Neurochemistry. 75(4). 1368–1376. 15 indexed citations
10.
Wight, P.A.L., et al.. (1999). Antisilencing. Journal of Neurochemistry. 72(6). 2227–2237. 21 indexed citations
11.
Wight, P.A.L., et al.. (1998). Myelin Proteolipid Protein Intron 1 Sequences Do Not Appear to Enhance Myelin Proteolipid Protein Gene Transcription. Developmental Neuroscience. 20(6). 552–558. 2 indexed citations
12.
Wight, P.A.L., et al.. (1997). Regulation of murine myelin proteolipid protein gene expression. Journal of Neuroscience Research. 50(6). 917–927. 13 indexed citations
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14.
Wight, P.A.L., et al.. (1986). Zinc toxicity in the fowl: Ultrastructural pathology and relationship to Selenium, lead and copper. Avian Pathology. 15(1). 23–38. 18 indexed citations
15.
Williams, Roger, Duilio Cascio, P.A.L. Wight, & Stephen R. Spindler. (1985). A Computer Method for Scanning and Analysis of Dot Blots. DNA. 4(3). 255–262. 1 indexed citations
16.
Wight, P.A.L. & D. W. F. Shannon. (1985). The morphology of the thyroid glands of quails and fowls maintained on diets containing rapeseed. Avian Pathology. 14(3). 383–399. 4 indexed citations
17.
Gentle, M. J., et al.. (1982). The effects of high dietary zinc on food intake in the domestic fowl. Appetite. 3(1). 53–60. 8 indexed citations
18.
Wight, P.A.L., et al.. (1979). The induction by muscle stimulation of a deep pectoral myopathy in the fowl. Avian Pathology. 8(1). 115–121. 16 indexed citations
19.
Siller, W.G., et al.. (1979). Effects of subfascial pressure in experimental deep pectoral myopathy of the fowl: An angiographic study. Avian Pathology. 8(4). 425–436. 18 indexed citations
20.
Wight, P.A.L., et al.. (1976). Three unusual intracranial tumours of the domesticated fowl. Avian Pathology. 5(3). 201–214. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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