Mark D. Crew

1.2k total citations
36 papers, 1.0k citations indexed

About

Mark D. Crew is a scholar working on Immunology, Molecular Biology and Surgery. According to data from OpenAlex, Mark D. Crew has authored 36 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Immunology, 18 papers in Molecular Biology and 6 papers in Surgery. Recurrent topics in Mark D. Crew's work include Immune Cell Function and Interaction (14 papers), T-cell and B-cell Immunology (10 papers) and Xenotransplantation and immune response (6 papers). Mark D. Crew is often cited by papers focused on Immune Cell Function and Interaction (14 papers), T-cell and B-cell Immunology (10 papers) and Xenotransplantation and immune response (6 papers). Mark D. Crew collaborates with scholars based in United States, Switzerland and South Korea. Mark D. Crew's co-authors include S R Spindler, Didier Portilla, Roy L. Walford, Stephen R. Spindler, P.A.L. Wight, Benjamin G. Lilienfeld, Jörg D. Seebach, Bounleut Phanavanh, Alan D. Proia and Frank J. Gonzalez and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and The Journal of Immunology.

In The Last Decade

Mark D. Crew

36 papers receiving 1.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mark D. Crew United States 18 388 257 227 198 190 36 1.0k
Philippe Ancian France 15 619 1.6× 234 0.9× 257 1.1× 86 0.4× 33 0.2× 26 1.1k
Gabrielle S. Sellick United Kingdom 22 762 2.0× 610 2.4× 227 1.0× 273 1.4× 149 0.8× 40 1.6k
Albert Ricken Germany 17 497 1.3× 143 0.6× 225 1.0× 102 0.5× 96 0.5× 50 1.0k
M J Keller United States 7 598 1.5× 198 0.8× 136 0.6× 50 0.3× 65 0.3× 7 826
I. Ishida Japan 20 384 1.0× 153 0.6× 551 2.4× 111 0.6× 60 0.3× 37 1.2k
Miwa Sohda Japan 21 699 1.8× 93 0.4× 92 0.4× 123 0.6× 98 0.5× 38 1.1k
Christine Powers United States 20 768 2.0× 209 0.8× 77 0.3× 137 0.7× 109 0.6× 23 1.3k
Janelle E. Collinge Australia 12 768 2.0× 179 0.7× 287 1.3× 80 0.4× 22 0.1× 19 1.4k
P. L. Kaye Australia 25 907 2.3× 402 1.6× 245 1.1× 190 1.0× 253 1.3× 49 1.8k
A-C Syvänen Sweden 17 442 1.1× 339 1.3× 323 1.4× 63 0.3× 55 0.3× 29 1.1k

Countries citing papers authored by Mark D. Crew

Since Specialization
Citations

This map shows the geographic impact of Mark D. Crew's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mark D. Crew with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mark D. Crew more than expected).

Fields of papers citing papers by Mark D. Crew

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mark D. Crew. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mark D. Crew. The network helps show where Mark D. Crew may publish in the future.

Co-authorship network of co-authors of Mark D. Crew

This figure shows the co-authorship network connecting the top 25 collaborators of Mark D. Crew. A scholar is included among the top collaborators of Mark D. Crew based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mark D. Crew. Mark D. Crew is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Li, Shenyang, Kiran K. Nagothu, Varsha G. Desai, et al.. (2009). Transgenic expression of proximal tubule peroxisome proliferator–activated receptor-α in mice confers protection during acute kidney injury. Kidney International. 76(10). 1049–1062. 117 indexed citations
2.
Crew, Mark D.. (2007). Play it in E or G: utilization of HLA‐E and ‐G in xenotransplantation. Xenotransplantation. 14(3). 198–207. 28 indexed citations
3.
Phanavanh, Bounleut, et al.. (2006). Characterization of porcine TAP genes: alternative splicing of TAP1. Immunogenetics. 58(5-6). 374–382. 5 indexed citations
4.
Boerma, Marjan, William P. Fiser, Grant Hoyt, et al.. (2006). Influence of mast cells on outcome after heterotopic cardiac transplantation in rats. Transplant International. 20(3). 256–265. 31 indexed citations
5.
Lilienfeld, Benjamin G., et al.. (2006). Porcine UL16-Binding Protein 1 Expressed on the Surface of Endothelial Cells Triggers Human NK Cytotoxicity through NKG2D. The Journal of Immunology. 177(4). 2146–2152. 56 indexed citations
6.
Crew, Mark D., et al.. (2005). An HLA-E single chain trimer inhibits human NK cell reactivity towards porcine cells. Molecular Immunology. 42(10). 1205–1214. 42 indexed citations
7.
Crew, Mark D., et al.. (2004). Sequence and mRNA expression of nonclassical SLA class I genes SLA-7 and SLA-8. Immunogenetics. 56(2). 111–114. 18 indexed citations
8.
Phanavanh, Bounleut, et al.. (2004). Molecular cloning and characterization of a porcine UL16 binding protein (ULBP)-like cDNA. Molecular Immunology. 42(6). 665–671. 15 indexed citations
9.
Crew, Mark D., et al.. (2003). Sequence, expression, and polymorphism of the Peromyscus leucopus Mhc class Ib gene, M4. Immunogenetics. 55(2). 95–99. 1 indexed citations
10.
Crew, Mark D. & Bounleut Phanavanh. (2003). Exploiting virus stealth technology for xenotransplantation: reduced human T cell responses to porcine cells expressing herpes simplex virus ICP47. Xenotransplantation. 10(1). 50–59. 6 indexed citations
11.
Cannon, Martin J., Timothy J. O’Brien, Lowell J. Underwood, et al.. (2002). Novel target antigens for dendritic cell-based immunotherapy against ovarian cancer. Expert Review of Anticancer Therapy. 2(1). 97–105. 19 indexed citations
12.
13.
14.
Crew, Mark D.. (1997). COMPILATION OF DISTINCT HLA‐A, ‐B AND ‐C TRANSMEMBRANE AND CYTOPLASMIC DOMAIN‐ENCODING SEQUENCES. European Journal of Immunogenetics. 24(6). 443–449. 2 indexed citations
15.
Crew, Mark D.. (1993). Genes of the major histocompatibility complex and the evolutionary genetics of lifespan. Genetica. 91(1-3). 225–238. 1 indexed citations
16.
Spindler, Stephen R., Mark D. Crew, Patricia L. Mote, Judith Grizzle, & Roy L. Walford. (1990). Dietary Energy Restriction in Mice Reduces Hepatic Expression of Glucose-Regulated Protein 78 (BiP) and 94 mRNA. Journal of Nutrition. 120(11). 1412–1417. 49 indexed citations
17.
Crew, Mark D., et al.. (1989). Polymorphism in the major histocompatibility complex class II genes ofPeromyscus leucopus. Immunogenetics. 30(3). 214–217. 11 indexed citations
18.
Spindler, S R, Mark D. Crew, & Jennifer K. Nyborg. (1989). Thyroid Hormone Transcriptional Regulatory Region of the Growth Hormone Gene. Endocrine Research. 15(4). 475–493. 6 indexed citations
19.
Wight, P.A.L., Mark D. Crew, & Stephen R. Spindler. (1988). Sequences Essential for Activity of the Thyroid Hormone Responsive Transcription Stimulatory Element of the Rat Growth Hormone Gene. Molecular Endocrinology. 2(6). 536–542. 31 indexed citations
20.
Crew, Mark D., Stephen R. Spindler, Roy L. Walford, & Akio Koizumi. (1987). Age-Related Decrease of Growth Hormone and Prolactin Gene Expression in the Mouse Pituitary*. Endocrinology. 121(4). 1251–1255. 37 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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