Pablo Cannata‐Ortiz

2.0k total citations · 1 hit paper
29 papers, 1.6k citations indexed

About

Pablo Cannata‐Ortiz is a scholar working on Molecular Biology, Immunology and Nephrology. According to data from OpenAlex, Pablo Cannata‐Ortiz has authored 29 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 9 papers in Immunology and 7 papers in Nephrology. Recurrent topics in Pablo Cannata‐Ortiz's work include Renal and related cancers (6 papers), Chronic Kidney Disease and Diabetes (5 papers) and Trace Elements in Health (4 papers). Pablo Cannata‐Ortiz is often cited by papers focused on Renal and related cancers (6 papers), Chronic Kidney Disease and Diabetes (5 papers) and Trace Elements in Health (4 papers). Pablo Cannata‐Ortiz collaborates with scholars based in Spain, Germany and Chile. Pablo Cannata‐Ortiz's co-authors include Alberto Ortíz, Marta Ruiz‐Ortega, Ana B. Sanz, Jesús Egido, María Dolores Sánchez-Niño, Susana Carrasco, Olga Ruiz‐Andrés, Diego Martín‐Sánchez, Jonay Poveda and Andreas Linkermann and has published in prestigious journals such as Proceedings of the National Academy of Sciences, PLoS ONE and Kidney International.

In The Last Decade

Pablo Cannata‐Ortiz

29 papers receiving 1.6k citations

Hit Papers

Ferroptosis, but Not Necr... 2016 2026 2019 2022 2016 100 200 300 400
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Ferroptosis, but Not Necroptosis, Is Important in Nephrotoxic Folic Acid–Induced AKI
    2016 445 citations Diego Martín‐Sánchez, Olga Ruiz‐Andrés et al. Journal of the American Society of Nephrology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Pablo Cannata‐Ortiz Spain 20 770 379 362 294 270 29 1.6k
Susana Carrasco Spain 17 583 0.8× 348 0.9× 358 1.0× 213 0.7× 282 1.0× 27 1.4k
Olga Ruiz‐Andrés Spain 13 616 0.8× 387 1.0× 316 0.9× 181 0.6× 234 0.9× 15 1.2k
Diego Martín‐Sánchez Spain 17 841 1.1× 368 1.0× 578 1.6× 176 0.6× 397 1.5× 21 1.6k
Amie Traylor United States 21 868 1.1× 281 0.7× 393 1.1× 129 0.4× 220 0.8× 34 1.7k
Chang Hyun Byon United States 11 549 0.7× 470 1.2× 186 0.5× 199 0.7× 185 0.7× 12 1.4k
Raquel Rodrigues‐Díez Spain 25 730 0.9× 311 0.8× 190 0.5× 313 1.1× 128 0.5× 45 1.6k
Zhanmei Zhou China 23 743 1.0× 609 1.6× 193 0.5× 175 0.6× 313 1.2× 46 1.6k
Ming‐Jiang Xu China 22 626 0.8× 458 1.2× 148 0.4× 280 1.0× 194 0.7× 37 1.7k
Sandrine Placier France 23 549 0.7× 492 1.3× 288 0.8× 166 0.6× 144 0.5× 53 1.8k
Bo Young Nam South Korea 20 528 0.7× 548 1.4× 268 0.7× 109 0.4× 259 1.0× 34 1.3k

Countries citing papers authored by Pablo Cannata‐Ortiz

Since Specialization
Citations

This map shows the geographic impact of Pablo Cannata‐Ortiz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Pablo Cannata‐Ortiz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Pablo Cannata‐Ortiz more than expected).

Fields of papers citing papers by Pablo Cannata‐Ortiz

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Pablo Cannata‐Ortiz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Pablo Cannata‐Ortiz. The network helps show where Pablo Cannata‐Ortiz may publish in the future.

Co-authorship network of co-authors of Pablo Cannata‐Ortiz

This figure shows the co-authorship network connecting the top 25 collaborators of Pablo Cannata‐Ortiz. A scholar is included among the top collaborators of Pablo Cannata‐Ortiz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Pablo Cannata‐Ortiz. Pablo Cannata‐Ortiz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Rayego‐Mateos, Sandra, Pablo Cannata‐Ortiz, Susana Carrasco, et al.. (2023). STING1 deficiency ameliorates immune‐mediated crescentic glomerulonephritis in mice. The Journal of Pathology. 261(3). 309–322. 1 indexed citations
2.
Cuarental, Leticia, Aránzazu Pintor‐Chocano, Sol Carriazo, et al.. (2022). The transcription factor Fosl1 preserves Klotho expression and protects from acute kidney injury. Kidney International. 103(4). 686–701. 23 indexed citations
3.
Rodrigues-Díez, Raúl R., Antonio Tejera‐Muñoz, Vanesa Esteban, et al.. (2021). CCN2 (Cellular Communication Network Factor 2) Deletion Alters Vascular Integrity and Function Predisposing to Aneurysm Formation. Hypertension. 79(3). e42–e55. 15 indexed citations
4.
Pérez-Gómez, María Vanessa, Elena Gomá-Garcés, Maria Soledad Pizarro-Sánchez, et al.. (2021). MO458URINARY GROWTH DIFFERENTIATION FACTOR-15 (GDF15) LEVELS AS BIOMARKER OF ADVERSE OUTCOMES AND BIOPSY FINDINGS IN CHRONIC KIDNEY DISEASE. Nephrology Dialysis Transplantation. 36(Supplement_1). 2 indexed citations
5.
Pérez-Gómez, María Vanessa, Maria Soledad Pizarro-Sánchez, Carolina Gracia-Iguacel, et al.. (2021). Urinary Growth Differentiation Factor-15 (GDF15) levels as a biomarker of adverse outcomes and biopsy findings in chronic kidney disease. Journal of Nephrology. 34(6). 1819–1832. 35 indexed citations
6.
Fontecha‐Barriuso, Miguel, Diego Martín‐Sánchez, Julio M. Martínez‐Moreno, et al.. (2019). PGC‐1α deficiency causes spontaneous kidney inflammation and increases the severity of nephrotoxic AKI. The Journal of Pathology. 249(1). 65–78. 88 indexed citations
7.
Orejudo, Macarena, Raúl R. Rodrigues-Díez, Raquel Rodrigues‐Díez, et al.. (2019). Interleukin 17A Participates in Renal Inflammation Associated to Experimental and Human Hypertension. Frontiers in Pharmacology. 10. 1015–1015. 50 indexed citations
8.
Fernández-Prado, Raúl, et al.. (2019). Positive/retained SDHB immunostaining in renal cell carcinomas associated to germline SDHB-deficiency: case report. Diagnostic Pathology. 14(1). 42–42. 7 indexed citations
9.
Martín‐Sánchez, Diego, Miguel Fontecha‐Barriuso, Susana Carrasco, et al.. (2018). TWEAK and RIPK1 mediate a second wave of cell death during AKI. Proceedings of the National Academy of Sciences. 115(16). 4182–4187. 118 indexed citations
10.
Benavente, R. Cabello, et al.. (2018). [Aggressive variants of castration resistant prostate cancer (CRPC): neuroendocrine prostate cancer.]. PubMed. 71(8). 721–734. 1 indexed citations
11.
Poveda, Jonay, Ana B. Sanz, Susana Carrasco, et al.. (2017). Bcl3: a regulator of NF-κB inducible by TWEAK in acute kidney injury with anti-inflammatory and antiapoptotic properties in tubular cells. Experimental & Molecular Medicine. 49(7). e352–e352. 43 indexed citations
12.
Suárez-Álvarez, Beatriz, José Luis Morgado‐Pascual, Sandra Rayego‐Mateos, et al.. (2016). Inhibition of Bromodomain and Extraterminal Domain Family Proteins Ameliorates Experimental Renal Damage. Journal of the American Society of Nephrology. 28(2). 504–519. 59 indexed citations
13.
Martín‐Sánchez, Diego, Olga Ruiz‐Andrés, Jonay Poveda, et al.. (2016). Ferroptosis, but Not Necroptosis, Is Important in Nephrotoxic Folic Acid–Induced AKI. Journal of the American Society of Nephrology. 28(1). 218–229. 445 indexed citations breakdown →
14.
González‐Guerrero, Cristian, Pablo Cannata‐Ortiz, Consuelo Guerri, et al.. (2016). TLR4-mediated inflammation is a key pathogenic event leading to kidney damage and fibrosis in cyclosporine nephrotoxicity. Archives of Toxicology. 91(4). 1925–1939. 74 indexed citations
15.
Cannata‐Ortiz, Pablo, Antonio Barat, Miguel Ángel Martínez‐González, et al.. (2016). Small vessel microembolization and acute glomerulonephritis following infection of aesthetic filler implants. Diagnostic Pathology. 11(1). 2–2. 10 indexed citations
16.
Ruiz‐Andrés, Olga, María Dolores Sánchez-Niño, Pablo Cannata‐Ortiz, et al.. (2016). SP170HISTONE LYSINE-CROTONYLATION IN ACUTE KIDNEY INJURY. Nephrology Dialysis Transplantation. 31(suppl_1). i143–i143. 1 indexed citations
17.
Martín‐Fernández, Beatriz, Alfonso Rubio‐Navarro, Isabel Cortegano, et al.. (2016). Aldosterone Induces Renal Fibrosis and Inflammatory M1-Macrophage Subtype via Mineralocorticoid Receptor in Rats. PLoS ONE. 11(1). e0145946–e0145946. 86 indexed citations
18.
Ruiz‐Andrés, Olga, María Dolores Sánchez-Niño, Pablo Cannata‐Ortiz, et al.. (2016). Histone lysine crotonylation during acute kidney injury in mice. Disease Models & Mechanisms. 9(6). 633–645. 125 indexed citations
19.
Sánchez-Niño, María Dolores, Beatriz Fernández‐Fernández, María Vanessa Pérez-Gómez, et al.. (2015). Albumin-induced apoptosis of tubular cells is modulated by BASP1. Cell Death and Disease. 6(2). e1644–e1644. 31 indexed citations
20.
González‐Guerrero, Cristian, Sergio Berzal, Susana Carrasco, et al.. (2013). Calcineurin inhibitors recruit protein kinases JAK2 and JNK, TLR signaling and the UPR to activate NF-κB-mediated inflammatory responses in kidney tubular cells. Toxicology and Applied Pharmacology. 272(3). 825–841. 37 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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