O Sakai

768 total citations
27 papers, 566 citations indexed

About

O Sakai is a scholar working on Molecular Biology, Nephrology and Surgery. According to data from OpenAlex, O Sakai has authored 27 papers receiving a total of 566 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 10 papers in Nephrology and 5 papers in Surgery. Recurrent topics in O Sakai's work include Renal Diseases and Glomerulopathies (9 papers), Amyloidosis: Diagnosis, Treatment, Outcomes (5 papers) and Drug Transport and Resistance Mechanisms (3 papers). O Sakai is often cited by papers focused on Renal Diseases and Glomerulopathies (9 papers), Amyloidosis: Diagnosis, Treatment, Outcomes (5 papers) and Drug Transport and Resistance Mechanisms (3 papers). O Sakai collaborates with scholars based in Japan and Czechia. O Sakai's co-authors include K Takazoe, Hitoshi Kubo, Kimiyoshi Ichida, Naohiko Katoh, Tatsuo Hosoya, Hiroshi Hayakawa, Naoyuki Kamatani, Toshio Hasegawa, Masaaki Nakayama and Kenichi Yamada and has published in prestigious journals such as Journal of Clinical Investigation, Kidney International and American Journal of Kidney Diseases.

In The Last Decade

O Sakai

26 papers receiving 541 citations

Peers

O Sakai
Comparison fields: 5 of 72
  • Nephrology 343
  • Surgery 150
  • Molecular Biology 134
  • Clinical Biochemistry 113
  • Pathology and Forensic Medicine 79
Replace Paola Cirina with:
Paola Cirina Italy
Irene de Lourdes Noronha Brazil
Ichiro Hirahara Japan
Przemyslaw Hirszel United States
Karl‐Martin Koch Germany
Kinga Musiał Poland
R. Bader Germany
Maria Bartosova Germany
Izaya Nakaya Japan
Mogensen Ce Denmark
Paola Cirina Italy View profile →
Citations per field, relative to O Sakai
O Sakai · 1×
Citations per year, relative to O Sakai
O Sakai · 1×

Countries citing papers authored by O Sakai

Since Specialization
Citations

This map shows the geographic impact of O Sakai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by O Sakai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites O Sakai more than expected).

Fields of papers citing papers by O Sakai

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by O Sakai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by O Sakai. The network helps show where O Sakai may publish in the future.

Co-authorship network of co-authors of O Sakai

This figure shows the co-authorship network connecting the top 25 collaborators of O Sakai. A scholar is included among the top collaborators of O Sakai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with O Sakai. O Sakai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 54
2 253
3 106
4 9
5
Possible mechanism of progressive renal failure in Crow-Fukase syndrome.
16
6
[A case of primary antiphospholipid antibody syndrome with severe nephrotic syndrome showing remarkable endothelial cell damage in the capillary lumen].
3
7 6
8 11
9
[Fundamental and clinical aspects of IgA nephropathy].
1
10
[RAEB in T with monosomy 7 after treatment of severe aplastic anemia with long term G-CSF].
3
11
Characteristics of anemia in patients with nephrotic syndrome.
4
12
Changes of blood volume distribution by postural changes in rats.
2
13 14
14
Pravastatin administration to hyperlipidemia in patients with nephrotic syndrome.
5
15
[Interferon-alpha therapy in the myeloproliferative variants of hypereosinophilic syndrome].
9
16 10
17
[D-penicillamine induced myasthenia gravis in a patient with rheumatoid arthritis].
1
18 37
19 1
20
[Allopurinol induced aplastic anemia in a patient with chronic renal failure].
1

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026