Noriko Sato
- Molecular Biology top 5%
- Immunology top 2%
- Oncology top 2%
- Cancer Research top 5%
- Physiology top 10%
- Co-authors
- Atsushi MiyajimaToshio KitamuraTadashi MatsudaYuichi SekineNaohiro TeradaKazuhiro SakamakiKatsuhiro AraiFrederick W. Quelle
- Topics
- Cytokine Signaling Pathways and Interactions (14 papers)Epigenetics and DNA Methylation (11 papers)Birth, Development, and Health (8 papers)
- Partner nations
- JapanUnited StatesFrance
In The Last Decade
Noriko Sato
98 papers receiving 3.8k citations
Hit Papers
Peers
Comparison fields: 5 of 135
- Molecular Biology 1.8k
- Immunology 1.4k
- Oncology 1.2k
- Cancer Research 376
- Physiology 353
Countries citing papers authored by Noriko Sato
This map shows the geographic impact of Noriko Sato's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Noriko Sato with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Noriko Sato more than expected).
Fields of papers citing papers by Noriko Sato
This network shows the impact of papers produced by Noriko Sato. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Noriko Sato. The network helps show where Noriko Sato may publish in the future.
Co-authorship network of co-authors of Noriko Sato
This figure shows the co-authorship network connecting the top 25 collaborators of Noriko Sato. A scholar is included among the top collaborators of Noriko Sato based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Noriko Sato. Noriko Sato is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 2 | |
| 3 | 5 | |
| 4 | 3 | |
| 5 | 12 | |
| 6 | 16 | |
| 7 | 10 | |
| 8 | A 2-year-old Female Infant with Transplacental HIV Transmission Despite Mother-to-child Transmission Prophylaxis | 0 |
| 9 | 16 | |
| 10 | 24 | |
| 11 | 34 | |
| 12 | 21 | |
| 13 | 73 | |
| 14 | 64 | |
| 15 | 68 | |
| 16 | 73 | |
| 17 | 10 | |
| 18 | 75 | |
| 19 | 83 | |
| 20 | Expression cloning of the human IL-3 receptor cDNA reveals a shared β subunit for the human IL-3 and GM-CSF receptorsbreakdown → | 538 |
About Noriko Sato
Noriko Sato is a scholar working on Obstetrics and Gynecology, Immunology and Immunology and Allergy, having authored 100 papers that have together received 3.9k indexed citations. Recurring topics across this work include Cytokine Signaling Pathways and Interactions (14 papers), Epigenetics and DNA Methylation (11 papers) and Birth, Development, and Health (8 papers). The work is most often cited by research in Immunology (1.4k citations), Oncology (1.2k citations) and Immunology and Allergy (226 citations). Noriko Sato has collaborated with scholars based in Japan, United States and France. Frequent co-authors include Atsushi Miyajima, Toshio Kitamura, Tadashi Matsuda, Yuichi Sekine, Naohiro Terada, Kazuhiro Sakamaki, Katsuhiro Arai, Frederick W. Quelle, James D. Griffin and James N. Ihle. Their work appears in journals such as Cell, Journal of Biological Chemistry and The EMBO Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.