Nobuhiro Inoue
- Molecular Biology
- Cellular and Molecular Neuroscience top 10%
- Neurology top 10%
- Neurology top 10%
- Physiology
- Co-authors
- Satoshi GotoEishichi MiyamotoYosuke MiharaYasuhiko MatsukadoYukitaka UshioKazumichi YamadaTakafumi IwasaE. Miyamoto
- Topics
- Neuroinflammation and Neurodegeneration Mechanisms (7 papers)Neuroscience and Neuropharmacology Research (6 papers)S100 Proteins and Annexins (5 papers)
- Partner nations
- JapanUnited StatesCanada
In The Last Decade
Nobuhiro Inoue
55 papers receiving 627 citations
Peers
Comparison fields: 5 of 106
- Molecular Biology 302
- Cellular and Molecular Neuroscience 239
- Neurology 108
- Neurology 95
- Physiology 92
Countries citing papers authored by Nobuhiro Inoue
This map shows the geographic impact of Nobuhiro Inoue's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nobuhiro Inoue with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nobuhiro Inoue more than expected).
Fields of papers citing papers by Nobuhiro Inoue
This network shows the impact of papers produced by Nobuhiro Inoue. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nobuhiro Inoue. The network helps show where Nobuhiro Inoue may publish in the future.
Co-authorship network of co-authors of Nobuhiro Inoue
This figure shows the co-authorship network connecting the top 25 collaborators of Nobuhiro Inoue. A scholar is included among the top collaborators of Nobuhiro Inoue based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nobuhiro Inoue. Nobuhiro Inoue is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 5 | |
| 2 | 1 | |
| 3 | 8 | |
| 4 | 0 | |
| 5 | 4 | |
| 6 | 13 | |
| 7 | 1 | |
| 8 | 6 | |
| 9 | 2 | |
| 10 | 15 | |
| 11 | 20 | |
| 12 | 7 | |
| 13 | Liver injury and alterations of hepatic microsomal monooxygenase system due to dimethylformamide (DMF) in rats. | 5 |
| 14 | 7 | |
| 15 | 19 | |
| 16 | 3 | |
| 17 | 24 | |
| 18 | 2 | |
| 19 | 7 | |
| 20 | [Comparative study of anti-aldosterone agents and hypotensive diuretics in essential hypertension by a double blind test]. | 2 |
About Nobuhiro Inoue
Nobuhiro Inoue is a scholar working on Neurology, Neurology and Structural Biology, having authored 60 papers that have together received 648 indexed citations. Recurring topics across this work include Neuroinflammation and Neurodegeneration Mechanisms (7 papers), Neuroscience and Neuropharmacology Research (6 papers) and S100 Proteins and Annexins (5 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (239 citations), Neurology (95 citations) and Developmental Neuroscience (33 citations). Nobuhiro Inoue has collaborated with scholars based in Japan, United States and Canada. Frequent co-authors include Satoshi Goto, Eishichi Miyamoto, Yosuke Mihara, Yasuhiko Matsukado, Yukitaka Ushio, Kazumichi Yamada, Takafumi Iwasa, E. Miyamoto, W. Roy Slaunwhite and A.A. Sandberg. Their work appears in journals such as Journal of Clinical Investigation, Cancer and Brain Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.