Neng Zhou

412 total citations
10 papers, 152 citations indexed

About

Neng Zhou is a scholar working on Molecular Biology, Immunology and Hematology. According to data from OpenAlex, Neng Zhou has authored 10 papers receiving a total of 152 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 4 papers in Immunology and 3 papers in Hematology. Recurrent topics in Neng Zhou's work include Hematopoietic Stem Cell Transplantation (3 papers), T-cell and B-cell Immunology (2 papers) and Immune Cell Function and Interaction (2 papers). Neng Zhou is often cited by papers focused on Hematopoietic Stem Cell Transplantation (3 papers), T-cell and B-cell Immunology (2 papers) and Immune Cell Function and Interaction (2 papers). Neng Zhou collaborates with scholars based in China and Thailand. Neng Zhou's co-authors include Cai‐Wen Duan, Bing Wang, Xian Chen, Maomao Zeng, Yi‐Zeng Liang, Jing Wu, Dongsheng Cao, Hong‐Dong Li, Mei Wang and Zhonghua Tang and has published in prestigious journals such as Nature Cell Biology, Biochemical and Biophysical Research Communications and Transplantation.

In The Last Decade

Neng Zhou

9 papers receiving 150 citations

Peers

Neng Zhou

MB

ONCOL

PHYSI

IMMUN

HEMAT

Claudia Einer Germany

Liu-Qin Yang China

Elena Samochatova Russia

Xianyanling Yi China

Yaming Xi China

Ryota Shirai Japan

F Bonanni Italy

Hyemin Park South Korea

Cheng Zu China

Claudia Einer Germany

Neng Zhou

68 ×0.7

MB

34 ×1.4

ONCOL

11 ×0.5

PHYSI

17 ×0.8

IMMUN

9 ×0.5

HEMAT

Citations per year, relative to Neng Zhou Neng Zhou (= 1×) peers Claudia Einer

Countries citing papers authored by Neng Zhou

Since Specialization

Citations

This map shows the geographic impact of Neng Zhou's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Neng Zhou with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Neng Zhou more than expected).

Fields of papers citing papers by Neng Zhou

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Neng Zhou. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Neng Zhou. The network helps show where Neng Zhou may publish in the future.

Co-authorship network of co-authors of Neng Zhou

This figure shows the co-authorship network connecting the top 25 collaborators of Neng Zhou. A scholar is included among the top collaborators of Neng Zhou based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Neng Zhou. Neng Zhou is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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10 of 10 papers shown

1.

Shi, Rongyi, Neng Zhou, Xuan Li, et al.. (2024). Trafficking circuit of CD8+ T cells between the intestine and bone marrow governs antitumour immunity. Nature Cell Biology. 26(8). 1346–1358. 4 indexed citations

2.

Zhu, Jianmin, Liting Yang, Jing Xia, et al.. (2024). Interleukin-27 Promotes the Generation of Myeloid-derived Suppressor Cells to Alleviate Graft-versus-host Disease. Transplantation. 108(12). e404–e416.

3.

Zhang, Sulin, Lifan Chen, Yan Wang, et al.. (2023). Goliath induces inflammation in obese mice by linking fatty acid β‐oxidation to glycolysis. EMBO Reports. 24(4). e56932–e56932. 7 indexed citations

4.

Xia, Jing, Minghao Li, Hang Zhou, et al.. (2021). CDK7 Inhibitor THZ1 Induces the Cell Apoptosis of B-Cell Acute Lymphocytic Leukemia by Perturbing Cellular Metabolism. Frontiers in Oncology. 11. 663360–663360. 14 indexed citations

5.

Huang, Wenli, Jing Xia, Lei Chu, et al.. (2021). CDK9 Inhibitor Induces the Apoptosis of B-Cell Acute Lymphocytic Leukemia by Inhibiting c-Myc-Mediated Glycolytic Metabolism. Frontiers in Cell and Developmental Biology. 9. 641271–641271. 18 indexed citations

6.

Liu, Shengli, Wenli Huang, Weiwei Zheng, et al.. (2020). Bromodomains and Extra-Terminal (BET) Inhibitor JQ1 Suppresses Proliferation of Acute Lymphocytic Leukemia by Inhibiting c-Myc-Mediated Glycolysis. Medical Science Monitor. 26. e923411–e923411. 10 indexed citations

7.

Liu, Shengli, Huiying Sun, Xia Qin, et al.. (2020). All‐trans‐retinoic acid shifts Th1 towards Th2 cell differentiation by targeting NFAT1 signalling to ameliorate immune‐mediated aplastic anaemia. British Journal of Haematology. 191(5). 906–919. 10 indexed citations

8.

Liu, Shengli, Neng Zhou, Zhonghua Tang, et al.. (2019). LGR6 promotes osteogenesis by activating the Wnt/β-catenin signaling pathway. Biochemical and Biophysical Research Communications. 519(1). 1–7. 31 indexed citations

9.

Liu, Shengli, et al.. (2019). Rapamycin ameliorates immune-mediated aplastic anemia by inhibiting the proliferation and metabolism of T cells. Biochemical and Biophysical Research Communications. 518(2). 212–218. 6 indexed citations

10.

Zeng, Maomao, Yi‐Zeng Liang, Hong‐Dong Li, et al.. (2010). Plasma metabolic fingerprinting of childhood obesity by GC/MS in conjunction with multivariate statistical analysis. Journal of Pharmaceutical and Biomedical Analysis. 52(2). 265–272. 52 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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