Nathan O. Stitziel

13.1k total citations · 3 hit papers
56 papers, 3.1k citations indexed

About

Nathan O. Stitziel is a scholar working on Molecular Biology, Genetics and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Nathan O. Stitziel has authored 56 papers receiving a total of 3.1k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 15 papers in Genetics and 14 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Nathan O. Stitziel's work include Lipoproteins and Cardiovascular Health (12 papers), Genetic Associations and Epidemiology (11 papers) and Diabetes, Cardiovascular Risks, and Lipoproteins (9 papers). Nathan O. Stitziel is often cited by papers focused on Lipoproteins and Cardiovascular Health (12 papers), Genetic Associations and Epidemiology (11 papers) and Diabetes, Cardiovascular Risks, and Lipoproteins (9 papers). Nathan O. Stitziel collaborates with scholars based in United States, United Kingdom and Netherlands. Nathan O. Stitziel's co-authors include Shamil Sunyaev, Adam Kieżun, Warren Gish, Hamideh Zakeri, Mark Yandell, Pui–Yan Kwok, Ian Korf, Gábor Marth, Zhijie Gu and Sekar Kathiresan and has published in prestigious journals such as Nature, New England Journal of Medicine and Proceedings of the National Academy of Sciences.

In The Last Decade

Nathan O. Stitziel

52 papers receiving 3.0k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Genetic risk, coronary heart disease events, and the clinical benefit of statin therapy: an analysis of primary and secondary prevention trials

2015 455 citations Jessica L. Mega, Nathan O. Stitziel et al. The Lancet profile →
Polygenic Risk Score Identifies Subgroup With Higher Burden of Atherosclerosis and Greater Relative Benefit From Statin Therapy in the Primary Prevention Setting

2017 349 citations Pradeep Natarajan, Robin Young et al. Circulation profile →
Coding Variation in ANGPTL4, LPL, and SVEP1 and the Risk of Coronary Disease

2016 337 citations Nathan O. Stitziel New England Journal of Medicine profile →

Peers

Countries citing papers authored by Nathan O. Stitziel

Since Specialization

Citations

This map shows the geographic impact of Nathan O. Stitziel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nathan O. Stitziel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nathan O. Stitziel more than expected).

Fields of papers citing papers by Nathan O. Stitziel

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nathan O. Stitziel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nathan O. Stitziel. The network helps show where Nathan O. Stitziel may publish in the future.

Co-authorship network of co-authors of Nathan O. Stitziel

This figure shows the co-authorship network connecting the top 25 collaborators of Nathan O. Stitziel. A scholar is included among the top collaborators of Nathan O. Stitziel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nathan O. Stitziel. Nathan O. Stitziel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	ANGPTL3 deficiency impairs lipoprotein production and produces adaptive changes in hepatic lipid metabolism 2024 ·Journal of Lipid Research ·(unknown), Yan Xie, (unknown), In‐Hyuk Jung, (unknown), Paul C. Lee, (unknown), (unknown), (unknown), (unknown), Arturo Alisio, Ira J. Goldberg, Brian N. Finck, Edward A. Fisher, Nicholas O. Davidson, Nathan O. Stitziel	9
2	Molecular Regulation and Therapeutic Targeting of VLDL Production in Cardiometabolic Disease 2024 ·Cellular and Molecular Gastroenterology and Hepatology ·(unknown), Nathan O. Stitziel, Nicholas O. Davidson	1
3	SVEP1 is an endogenous ligand for the orphan receptor PEAR1 2023 ·Nature Communications ·(unknown), (unknown), Katrina Ashworth, Chul Joo Kang, (unknown), (unknown), In‐Hyuk Jung, Paul C. Lee, (unknown), Junedh Amrute, Robert P. Mecham, Carmen M. Halabi, Arturo Alisio, Jorge Di Paola, Nathan O. Stitziel	17
4	Functional assays reveal the pathogenic mechanism of a de novo tropomyosin variant identified in patient with dilated cardiomyopathy 2023 ·Journal of Molecular and Cellular Cardiology ·(unknown), (unknown), (unknown), Shanshan Zhang, Chieh‐Yu Lin, Nathan O. Stitziel, Michael J. Greenberg	3
5	Abstract 16384: Interaction of Socioeconomic Status With Polygenic Risk for Coronary Artery Disease in a Novel Biobank 2023 ·Circulation ·(unknown), Nicolas Ledru, Nathan O. Stitziel	0
6	Distribution of 54 polygenic risk scores for common diseases in long lived individuals and their offspring 2022 ·GeroScience ·(unknown), Michael Wainberg, Zeyuan Song, Stacy L. Andersen, Robert M. Boudreau, Mary F. Feitosa, Qihua Tan, May E. Montasser, Jeffrey R. O’Connell, Nathan O. Stitziel, Nathan D. Price, Thomas T. Perls, Nicholas J. Schork, Paola Sebastiani	7
7	SVEP1 is a human coronary artery disease locus that promotes atherosclerosis 2021 ·Science Translational Medicine ·In‐Hyuk Jung, (unknown), Arturo Alisio, (unknown), Erica P. Young, Chul Joo Kang, Puja Kachroo, Kory J. Lavine, Babak Razani, Robert P. Mecham, Nathan O. Stitziel	34
8	Mapping and characterization of structural variation in 17,795 human genomes 2020 ·Nature ·Haley Abel, David E. Larson, Allison Regier, Colby Chiang, Indraniel Das, Krishna Kanchi, Ryan M. Layer, Benjamin M. Neale, William Salerno, Catherine Reeves, Steven Buyske, Tara C. Matise, Donna M. Muzny, Michael C. Zody, Eric S. Lander, Susan K. Dutcher, Nathan O. Stitziel, Ira M. Hall	148
9	Non-parametric Polygenic Risk Prediction via Partitioned GWAS Summary Statistics 2020 ·The American Journal of Human Genetics ·Sung Chun, Maxim Imakaev, Daniel Hui, Nikolaos A. Patsopoulos, Benjamin M. Neale, Sekar Kathiresan, Nathan O. Stitziel, Shamil Sunyaev	24
10	Intracellular retention of mutant lysyl oxidase leads to aortic dilation in response to increased hemodynamic stress 2019 ·JCI Insight ·Vivian S. Lee, Carmen M. Halabi, Thomas J. Broekelmann, Philip C. Trackman, Nathan O. Stitziel, Robert P. Mecham	14
11	New Sequencing technologies help revealing unexpected mutations in Autosomal Dominant Hypercholesterolemia 2018 ·Scientific Reports ·Sandy Elbitar, Delia Susan‐Resiga, Youmna Ghaleb, Petra El Khoury, Gina M. Peloso, Nathan O. Stitziel, Jean‐Pierre Rabès, Valérie Carreau, Josée Hamelin, (unknown), Éric Bruckert, Cathérine Boileau, Nabil G. Seidah, Mathilde Varret, Marianne Abifadel	25
12	Genetics of the extracellular matrix in aortic aneurysmal diseases 2018 ·Matrix Biology ·Chien‐Jung Lin, Chieh‐Yu Lin, Nathan O. Stitziel	21
13	Polygenic Risk Score Identifies Subgroup With Higher Burden of Atherosclerosis and Greater Relative Benefit From Statin Therapy in the Primary Prevention Setting breakdown → 2017 ·Circulation ·Pradeep Natarajan, Robin Young, Nathan O. Stitziel, Sandosh Padmanabhan, Usman Baber, Roxana Mehran, Samantha Sartori, Valentı́n Fuster, Dermot F. Reilly, Adam S. Butterworth, Daniel J. Rader, Ian Ford, Naveed Sattar, Sekar Kathiresan	349
14	Coding Variation in ANGPTL4, LPL, and SVEP1 and the Risk of Coronary Disease breakdown → 2016 ·New England Journal of Medicine ·Nathan O. Stitziel	337
15	Leveraging human genetics to guide drug target discovery 2016 ·Trends in Cardiovascular Medicine ·Nathan O. Stitziel, Sekar Kathiresan	23
16	Diagnosis and management of adult hereditary cardio-neuromuscular disorders: A model for the multidisciplinary care of complex genetic disorders 2016 ·Trends in Cardiovascular Medicine ·R. Brian Sommerville, (unknown), (unknown), Anne Casey, Nathan O. Stitziel, Anne M. Connolly, Douglas L. Mann	16
17	Genetic risk, coronary heart disease events, and the clinical benefit of statin therapy: an analysis of primary and secondary prevention trials breakdown → 2015 ·The Lancet ·Jessica L. Mega, Nathan O. Stitziel, J. G. Smith, Daniel I. Chasman, Mark J. Caulfield, James J. Devlin, Francesco Nordio, Craig Hyde, Christopher P. Cannon, Frank M. Sacks, Neil R Poulter, Peter Sever, Paul M. Ridker, Eugene Braunwald, Olle Melander, Sekar Kathiresan, Marc S. Sabatine	455
18	APOE p.Leu167del mutation in familial hypercholesterolemia 2013 ·Atherosclerosis ·Zuhier Awan, Hong Y. Choi, Nathan O. Stitziel, Isabelle L. Ruel, Mary A. Bamimore, (unknown), Marie-Hélène Gagnon, (unknown), Gina M. Peloso, Robert A. Hegele, Nabil G. Seidah, Sekar Kathiresan, Jacques Genest	81
19	Abstract 13065: Rare Coding Mutations and Risk for Early-Onset Myocardial Infarction: An Exome Sequencing Study of >2,000 Cases and Controls 2011 ·Circulation ·Nathan O. Stitziel	2
20	Computational and statistical approaches to analyzing variants identified by exome sequencing 2011 ·Genome Biology ·Nathan O. Stitziel, Adam Kieżun, Shamil Sunyaev	97

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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