Nathan Lanning

1.1k citations
19 papers · 872 indexed · h-index 16

Nathan Lanning

18 papers receiving 862 citations

Peers

Nathan Lanning
Comparison fields: 5 of 97
  • Cancer Research 238
  • Endocrinology, Diabetes and Metabolism 188
  • Molecular Biology 530
  • Hepatology 58
  • Oncology 178
Replace Robert V. Martinez with:
Robert V. Martinez United States
Anja Krones‐Herzig Germany
Takao Nammo Japan
Nora Zizlsperger United States
Pierre Scotney Australia
Inmaculada Ibañez de Cáceres Spain
Xiaoli Zhang China
Elizabeth Rico-Bautista Sweden
Alphonse Le Cam France
Maria T. Bengoechea-Alonso Sweden
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Citations per year

Countries citing papers authored by Nathan Lanning

Since Specialization
Citations

This map shows the geographic impact of Nathan Lanning's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nathan Lanning with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nathan Lanning more than expected).

Fields of papers citing papers by Nathan Lanning

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nathan Lanning. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nathan Lanning. The network helps show where Nathan Lanning may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Nathan Lanning, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Nathan Lanning Line = papers co-authored together Nathan Lanning links everyone, so they are left out of the graph.

All Works

19 of 19 papers shown
#Work
1 20235
2 20212
3 20201
4
Phosphorylation of OXPHOS Machinery Subunits: Functional Implications in Cell Biology and Disease.
201932
5 201817
6 2017129
7 201720
8 201422
9 201470
10 201326
11 201318
12 201198
13 201133
14 201131
15 201115
16 200844
17 2006171
18 200575
19 200463

About Nathan Lanning

Nathan Lanning is a scholar working on Cancer Research, Immunology and Allergy, Endocrinology, Diabetes and Metabolism, Oncology and Cell Biology, having authored 19 papers that have together received 872 indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (5 papers), Cancer, Hypoxia, and Metabolism (5 papers), ATP Synthase and ATPases Research (5 papers), RNA modifications and cancer (4 papers), Growth Hormone and Insulin-like Growth Factors (4 papers), Cytokine Signaling Pathways and Interactions (4 papers), Protein Tyrosine Phosphatases (3 papers) and Lipid metabolism and disorders (2 papers). The work is most often cited by research in Cancer Research (238 citations), Endocrinology, Diabetes and Metabolism (188 citations), Molecular Biology (530 citations), Hepatology (58 citations) and Oncology (178 citations). Nathan Lanning has collaborated with scholars based in United States, Israel and China. Frequent co-authors include Christin Carter‐Su, Jeffrey P. MacKeigan, Natalie M. Niemi, Hui Jin, Yanli Su, George F. Vande Woude, Joshua Castle, Simar Singh, Fabian V. Filipp and Elizabeth A. Tovar. Their work appears in journals such as Molecular Endocrinology, Journal of Cell Science, PLoS ONE, Neoplasia and Cell Reports.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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