Robert E. Sigler

3.0k total citations · 1 hit paper
48 papers, 2.4k citations indexed

About

Robert E. Sigler is a scholar working on Molecular Biology, Internal Medicine and Hematology. According to data from OpenAlex, Robert E. Sigler has authored 48 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 11 papers in Internal Medicine and 11 papers in Hematology. Recurrent topics in Robert E. Sigler's work include Venous Thromboembolism Diagnosis and Management (11 papers), Blood Coagulation and Thrombosis Mechanisms (10 papers) and Cell Adhesion Molecules Research (5 papers). Robert E. Sigler is often cited by papers focused on Venous Thromboembolism Diagnosis and Management (11 papers), Blood Coagulation and Thrombosis Mechanisms (10 papers) and Cell Adhesion Molecules Research (5 papers). Robert E. Sigler collaborates with scholars based in United States, Canada and China. Robert E. Sigler's co-authors include Richard A. Miller, Michael Smith‐Wheelock, James M. Harper, Yayi Chang, Cassandra R. Zylstra, Bart O. Williams, Thomas L. Clemens, Mary Bouxsein, Sheri L. Holmen and Marie–Claude Faugere and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Blood.

In The Last Decade

Robert E. Sigler

47 papers receiving 2.3k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Methionine‐deficient diet extends mouse lifespan, slows immune and lens aging, alters glucose, T4, IGF‐I and insulin levels, and increases hepatocyte MIF levels and stress resistance

2005 568 citations Richard A. Miller, Yayi Chang et al. Aging Cell profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Robert E. Sigler United States	21	1.1k	418	396	317	267	48	2.4k
David A. Alcorta United States	25	1.4k 1.3×	594 1.4×	578 1.5×	112 0.4×	172 0.6×	38	2.6k
Thomas H. Norwood United States	34	1.7k 1.6×	519 1.2×	759 1.9×	185 0.6×	536 2.0×	79	3.4k
Martine Ffrench France	32	1.4k 1.2×	908 2.2×	401 1.0×	104 0.3×	153 0.6×	112	3.8k
Charles K. Lumpkin United States	34	1.8k 1.6×	635 1.5×	463 1.2×	79 0.2×	565 2.1×	69	3.4k
Alberto Benguría Spain	20	1.8k 1.6×	542 1.3×	921 2.3×	330 1.0×	149 0.6×	58	3.1k
Kovalev Gi United States	17	1.1k 1.0×	441 1.1×	699 1.8×	213 0.7×	206 0.8×	43	2.6k
Thomas G. Bird United Kingdom	24	1.2k 1.1×	391 0.9×	762 1.9×	181 0.6×	191 0.7×	77	3.7k
Chad Torrice United States	19	1.8k 1.6×	635 1.5×	1.4k 3.5×	396 1.2×	373 1.4×	35	3.6k
Eric Campeau United States	17	2.2k 2.0×	558 1.3×	1.2k 2.9×	201 0.6×	214 0.8×	32	3.6k
Jian Guo China	33	1.7k 1.5×	349 0.8×	292 0.7×	173 0.5×	297 1.1×	93	3.8k

Countries citing papers authored by Robert E. Sigler

Since Specialization

Citations

This map shows the geographic impact of Robert E. Sigler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert E. Sigler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert E. Sigler more than expected).

Fields of papers citing papers by Robert E. Sigler

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert E. Sigler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert E. Sigler. The network helps show where Robert E. Sigler may publish in the future.

Co-authorship network of co-authors of Robert E. Sigler

This figure shows the co-authorship network connecting the top 25 collaborators of Robert E. Sigler. A scholar is included among the top collaborators of Robert E. Sigler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert E. Sigler. Robert E. Sigler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Khan, Safeera, Jessica Chavez, Xuewei Zhu, et al.. (2021). Carbon Nanodots Inhibit Oxidized Low Density Lipoprotein-Induced Injury and Monocyte Adhesion to Endothelial Cells Through Scavenging Reactive Oxygen Species. Journal of Biomedical Nanotechnology. 17(8). 1654–1667. 5 indexed citations

Myers, Daniel D., Patrick A. Lester, Reheman Adili, et al.. (2021). E-selectin inhibitor is superior to low-molecular-weight heparin for the treatment of experimental venous thrombosis. Journal of Vascular Surgery Venous and Lymphatic Disorders. 10(1). 211–220. 6 indexed citations

Myers, Daniel D., Patrick A. Lester, Reheman Adili, et al.. (2020). A new way to treat proximal deep venous thrombosis using E-selectin inhibition. Journal of Vascular Surgery Venous and Lymphatic Disorders. 8(2). 268–278. 14 indexed citations

Movsas, Tammy Z., Kwoon Y. Wong, Michael D. Ober, et al.. (2018). Confirmation of Luteinizing Hormone (LH) in Living Human Vitreous and the Effect of LH Receptor Reduction on Murine Electroretinogram. Neuroscience. 385. 1–10. 13 indexed citations

Song, Jun, Guoshun Wang, Mark J. Hoenerhoff, et al.. (2018). Bacterial and Pneumocystis Infections in the Lungs of Gene-Knockout Rabbits with Severe Combined Immunodeficiency. Frontiers in Immunology. 9. 429–429. 18 indexed citations

Hawley, Angela E., Suman L. Sood, Robert E. Sigler, et al.. (2017). E-selectin inhibition with GMI-1271 decreases venous thrombosis without profoundly affecting tail vein bleeding in a mouse model. Thrombosis and Haemostasis. 117(6). 1171–1181. 24 indexed citations

Monsma, David J., Noel R. Monks, David Cherba, et al.. (2012). Genomic characterization of explant tumorgraft models derived from fresh patient tumor tissue. Journal of Translational Medicine. 10(1). 125–125. 55 indexed citations

Díaz, José A., et al.. (2012). Circulating and vein wall P-selectin promote venous thrombogenesis during aging in a rodent model. Thrombosis Research. 131(1). 42–48. 36 indexed citations

Graveel, Carrie R., et al.. (2010). Germline Met Mutations in Mice Reveal Mutation- and Background-Associated Differences in Tumor Profiles. PLoS ONE. 5(10). e13586–e13586. 7 indexed citations

10.

Graveel, Carrie R., Yanli Su, Julie Koeman, et al.. (2009). Met induces diverse mammary carcinomas in mice and is associated with human basal breast cancer. Proceedings of the National Academy of Sciences. 106(31). 12909–12914. 91 indexed citations

11.

Xie, Qian, Ryan C. Thompson, Lisa M. DeCamp, et al.. (2008). A highly invasive human glioblastoma pre-clinical model for testing therapeutics. Journal of Translational Medicine. 6(1). 77–77. 46 indexed citations

12.

Baxi, Sanjiv M., David L. Crandall, Shirley K. Wrobleski, et al.. (2008). Dose-dependent thrombus resolution due to oral plasminogen activator inhibitor (PAI)-1 inhibition with tiplaxtinin in a rat stenosis model of venous thrombosis. Thrombosis and Haemostasis. 99(4). 749–758. 26 indexed citations

13.

Bruxvoort, Katia, Troy A. Giambernardi, Chao-Nan Qian, et al.. (2007). Inactivation of Apc in the Mouse Prostate Causes Prostate Carcinoma. Cancer Research. 67(6). 2490–2496. 78 indexed citations

14.

Eisenmann, Kathryn M., Richard West, Dagmar Hildebrand, et al.. (2007). T Cell Responses in Mammalian Diaphanous-related Formin mDia1 Knock-out Mice. Journal of Biological Chemistry. 282(34). 25152–25158. 103 indexed citations

15.

Staal, Ben, Yingchun Su, P J Swiatek, et al.. (2006). Evidence that MIG-6 is a tumor-suppressor gene. Oncogene. 26(2). 269–276. 107 indexed citations

16.

Miller, Richard A., et al.. (2005). Methionine‐deficient diet extends mouse lifespan, slows immune and lens aging, alters glucose, T4, IGF‐I and insulin levels, and increases hepatocyte MIF levels and stress resistance. Aging Cell. 4(3). 119–125. 568 indexed citations breakdown →

17.

Johnson, Ron J., W D Black, Robert E. Sigler, Vijaykumar M. Baragi, & Alec W. Gough. (2000). Evaluation of toxicokinetic variables and histologic appearance of arthropathic changes in juvenile rabbits after oral administration of an investigational fluoroquinolone, PD 117596. American Journal of Veterinary Research. 61(11). 1396–1402.

18.

Baragi, Vijaykumar M., Richard R. Renkiewicz, Luping Qiu, et al.. (1997). Transplantation of adenovirally transduced allogeneic chondrocytes into articular cartilage defects in vivo. Osteoarthritis and Cartilage. 5(4). 275–282. 101 indexed citations

19.

Sigler, Robert E., et al.. (1995). Absence of Ki-ras mutations in exocrine pancreatic tumors from male rats chronically exposed to gabapentin. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 327(1-2). 151–160. 7 indexed citations

20.

Sigler, Robert E., Carnell Newkirk, & Elizabeth M. McDowell. (1988). Histogenesis and morphogenesis of epidermoid metaplasia in hamster tracheal organ explant culture. Virchows Archiv B Cell Pathology. 55(1). 47–55. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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