Natasha Khatri

445 total citations
8 papers, 329 citations indexed

About

Natasha Khatri is a scholar working on Molecular Biology, Genetics and Cellular and Molecular Neuroscience. According to data from OpenAlex, Natasha Khatri has authored 8 papers receiving a total of 329 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 3 papers in Genetics and 2 papers in Cellular and Molecular Neuroscience. Recurrent topics in Natasha Khatri's work include Genetics and Neurodevelopmental Disorders (3 papers), Ubiquitin and proteasome pathways (2 papers) and Genetic Syndromes and Imprinting (1 paper). Natasha Khatri is often cited by papers focused on Genetics and Neurodevelopmental Disorders (3 papers), Ubiquitin and proteasome pathways (2 papers) and Genetic Syndromes and Imprinting (1 paper). Natasha Khatri collaborates with scholars based in United States, Pakistan and India. Natasha Khatri's co-authors include Heng‐Ye Man, James Gilbert, Yuda Huo, Guan Wang, Qingming Hou, James E. Huettner, Emiliano Biasini, David A. Harris, Tania Massignan and Isaac H. Solomon and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Neuroscience and Scientific Reports.

In The Last Decade

Natasha Khatri

8 papers receiving 329 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Natasha Khatri United States	6	219	103	64	55	45	8	329
Sharee Kuny Canada	14	281 1.3×	105 1.0×	93 1.5×	18 0.3×	38 0.8×	21	471
Ranmal A. Samarasinghe United States	8	226 1.0×	74 0.7×	101 1.6×	24 0.4×	53 1.2×	9	504
Francesco Scalabrì Italy	9	227 1.0×	74 0.7×	71 1.1×	43 0.8×	20 0.4×	11	324
Nina M. Rzechorzek United Kingdom	9	154 0.7×	65 0.6×	53 0.8×	47 0.9×	12 0.3×	19	362
Fernando J. Bustos Chile	10	216 1.0×	72 0.7×	110 1.7×	28 0.5×	51 1.1×	24	376
Alfonso Oyarzábal Spain	11	242 1.1×	79 0.8×	38 0.6×	19 0.3×	25 0.6×	18	452
David Lewis‐Smith United Kingdom	11	195 0.9×	80 0.8×	54 0.8×	23 0.4×	17 0.4×	18	288
Katharina Pernhorst Germany	9	116 0.5×	61 0.6×	116 1.8×	42 0.8×	14 0.3×	9	290
Daji Guo China	10	123 0.6×	70 0.7×	44 0.7×	25 0.5×	51 1.1×	15	254
István Bock Hungary	11	290 1.3×	56 0.5×	74 1.2×	30 0.5×	14 0.3×	23	451

Countries citing papers authored by Natasha Khatri

Since Specialization

Citations

This map shows the geographic impact of Natasha Khatri's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Natasha Khatri with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Natasha Khatri more than expected).

Fields of papers citing papers by Natasha Khatri

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Natasha Khatri. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Natasha Khatri. The network helps show where Natasha Khatri may publish in the future.

Co-authorship network of co-authors of Natasha Khatri

This figure shows the co-authorship network connecting the top 25 collaborators of Natasha Khatri. A scholar is included among the top collaborators of Natasha Khatri based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Natasha Khatri. Natasha Khatri is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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8 of 8 papers shown

1.

Abraham, Neeta A., Nadine F. Joseph, Sarah Geisler, et al.. (2024). Dose-dependent reduction of somatic expansions but not Htt aggregates by di-valent siRNA-mediated silencing of MSH3 in HdhQ111 mice. Scientific Reports. 14(1). 2061–2061. 5 indexed citations

2.

Khatri, Natasha, et al.. (2021). Delay in Permanent Vascular Access Formation and Referral to a Nephrologist in Incident Hemodialysis Patients: A Single Center Experience. Cureus. 13(12). e20728–e20728. 5 indexed citations

3.

Oyler, Karl D., et al.. (2021). Primary Explosive Processing in the Resonant Acoustic Mixer. Propellants Explosives Pyrotechnics. 46(5). 697–704. 14 indexed citations

4.

Khatri, Natasha & Heng‐Ye Man. (2019). The Autism and Angelman Syndrome Protein Ube3A/E6AP: The Gene, E3 Ligase Ubiquitination Targets and Neurobiological Functions. Frontiers in Molecular Neuroscience. 12. 109–109. 57 indexed citations

5.

Khatri, Natasha, James Gilbert, Yuda Huo, et al.. (2017). The Autism Protein Ube3A/E6AP Remodels Neuronal Dendritic Arborization via Caspase-Dependent Microtubule Destabilization. Journal of Neuroscience. 38(2). 363–378. 56 indexed citations

6.

Huo, Yuda, Natasha Khatri, Qingming Hou, et al.. (2015). The deubiquitinating enzyme USP46 regulates AMPA receptor ubiquitination and trafficking. Journal of Neurochemistry. 134(6). 1067–1080. 65 indexed citations

7.

Khatri, Natasha & Heng‐Ye Man. (2013). Synaptic Activity and Bioenergy Homeostasis: Implications in Brain Trauma and Neurodegenerative Diseases. Frontiers in Neurology. 4. 199–199. 73 indexed citations

8.

Solomon, Isaac H., Natasha Khatri, Emiliano Biasini, et al.. (2011). An N-terminal Polybasic Domain and Cell Surface Localization Are Required for Mutant Prion Protein Toxicity. Journal of Biological Chemistry. 286(16). 14724–14736. 54 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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