Kelly E. Glajch

1.6k total citations
20 papers, 1.2k citations indexed

About

Kelly E. Glajch is a scholar working on Cellular and Molecular Neuroscience, Neurology and Molecular Biology. According to data from OpenAlex, Kelly E. Glajch has authored 20 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Cellular and Molecular Neuroscience, 11 papers in Neurology and 6 papers in Molecular Biology. Recurrent topics in Kelly E. Glajch's work include Parkinson's Disease Mechanisms and Treatments (9 papers), Genetic Neurodegenerative Diseases (5 papers) and Neuroscience and Neuropharmacology Research (4 papers). Kelly E. Glajch is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (9 papers), Genetic Neurodegenerative Diseases (5 papers) and Neuroscience and Neuropharmacology Research (4 papers). Kelly E. Glajch collaborates with scholars based in United States, Italy and Germany. Kelly E. Glajch's co-authors include Ghazaleh Sadri‐Vakili, Pavel Osten, D. James Surmeier, Sheila M. Fleming, C. Savio Chan, Caroline Benn, Qiaoling Cui, George J. Yohrling, Nicholas J. Justice and Tim W. Clark and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and Nature Neuroscience.

In The Last Decade

Kelly E. Glajch

19 papers receiving 1.1k citations

Peers

Kelly E. Glajch
Mihaela Stavarache United States
Rainer von Coelln United States
Matthew J. Benskey United States
Craig Meyers United States
Deborah E. Cabin United States
Mihaela Stavarache United States
Kelly E. Glajch
Citations per year, relative to Kelly E. Glajch Kelly E. Glajch (= 1×) peers Mihaela Stavarache

Countries citing papers authored by Kelly E. Glajch

Since Specialization
Citations

This map shows the geographic impact of Kelly E. Glajch's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Kelly E. Glajch with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Kelly E. Glajch more than expected).

Fields of papers citing papers by Kelly E. Glajch

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Kelly E. Glajch. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Kelly E. Glajch. The network helps show where Kelly E. Glajch may publish in the future.

Co-authorship network of co-authors of Kelly E. Glajch

This figure shows the co-authorship network connecting the top 25 collaborators of Kelly E. Glajch. A scholar is included among the top collaborators of Kelly E. Glajch based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Kelly E. Glajch. Kelly E. Glajch is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Hering, Heike, Thierry Bussière, Chia‐Chen Liu, et al.. (2025). A manifesto for Alzheimer’s disease drug discovery in the era of disease-modifying therapies. Molecular Neurodegeneration. 20(1). 88–88.
2.
Abraham, Neeta A., Nadine F. Joseph, Sarah Geisler, et al.. (2024). Dose-dependent reduction of somatic expansions but not Htt aggregates by di-valent siRNA-mediated silencing of MSH3 in HdhQ111 mice. Scientific Reports. 14(1). 2061–2061. 5 indexed citations
3.
Laar, Amber D. Van, Matthew T. Keeney, Victor S. Van Laar, et al.. (2023). Transient exposure to rotenone causes degeneration and progressive parkinsonian motor deficits, neuroinflammation, and synucleinopathy. npj Parkinson s Disease. 9(1). 121–121. 37 indexed citations
4.
Patterson, Joseph R., Warren D. Hirst, Jacob W. Howe, et al.. (2022). Beta2-adrenoreceptor agonist clenbuterol produces transient decreases in alpha-synuclein mRNA but no long-term reduction in protein. npj Parkinson s Disease. 8(1). 61–61. 8 indexed citations
5.
Glajch, Kelly E., Tim E. Moors, Yi Chen, et al.. (2021). Wild-type GBA1 increases the α-synuclein tetramer–monomer ratio, reduces lipid-rich aggregates, and attenuates motor and cognitive deficits in mice. Proceedings of the National Academy of Sciences. 118(31). 32 indexed citations
6.
Petrozziello, Tiziana, Sali M.K. Farhan, Kaly A. Mueller, et al.. (2020). Lipocalin‐2 is increased in amyotrophic lateral sclerosis. Muscle & Nerve. 62(2). 272–283. 8 indexed citations
7.
Apicco, Daniel J., Evgeny Shlevkov, Catherine L. Nezich, et al.. (2020). The Parkinson’s disease-associated gene ITPKB protects against α-synuclein aggregation by regulating ER-to-mitochondria calcium release. Proceedings of the National Academy of Sciences. 118(1). 44 indexed citations
8.
Mueller, Kaly A., Kelly E. Glajch, Megan N. Huizenga, et al.. (2018). Hippo Signaling Pathway Dysregulation in Human Huntington’s Disease Brain and Neuronal Stem Cells. Scientific Reports. 8(1). 11355–11355. 54 indexed citations
9.
Glajch, Kelly E., Laura Ferraiuolo, Kaly A. Mueller, et al.. (2016). MicroNeurotrophins Improve Survival in Motor Neuron-Astrocyte Co-Cultures but Do Not Improve Disease Phenotypes in a Mutant SOD1 Mouse Model of Amyotrophic Lateral Sclerosis. PLoS ONE. 11(10). e0164103–e0164103. 19 indexed citations
10.
Glajch, Kelly E., Qiaoling Cui, Harry S. Xenias, et al.. (2016). Npas1+Pallidal Neurons Target Striatal Projection Neurons. Journal of Neuroscience. 36(20). 5472–5488. 70 indexed citations
11.
Cui, Qiaoling, Kelly E. Glajch, Jason Pitt, et al.. (2015). Parvalbumin+ Neurons and Npas1+ Neurons Are Distinct Neuron Classes in the Mouse External Globus Pallidus. Journal of Neuroscience. 35(34). 11830–11847. 106 indexed citations
12.
Glajch, Kelly E. & Ghazaleh Sadri‐Vakili. (2015). Upcoming Meetings Related to Huntington's Disease. Journal of Huntington s Disease. 4(1). 107–107. 34 indexed citations
13.
Glajch, Kelly E. & Ghazaleh Sadri‐Vakili. (2015). Epigenetic Mechanisms Involved in Huntington's Disease Pathogenesis. Journal of Huntington s Disease. 4(1). 1–15. 27 indexed citations
14.
Chan, C. Savio, Jeffrey D. Peterson, Kelly E. Glajch, et al.. (2012). Strain-Specific Regulation of Striatal Phenotype in Drd2-eGFP BAC Transgenic Mice. Journal of Neuroscience. 32(27). 9124–9132. 54 indexed citations
15.
Glajch, Kelly E., Sheila M. Fleming, D. James Surmeier, & Pavel Osten. (2011). Sensorimotor assessment of the unilateral 6-hydroxydopamine mouse model of Parkinson's disease. Behavioural Brain Research. 230(2). 309–316. 122 indexed citations
16.
Chan, C. Savio, Kelly E. Glajch, Tracy S. Gertler, et al.. (2010). HCN channelopathy in external globus pallidus neurons in models of Parkinson's disease. Nature Neuroscience. 14(1). 85–92. 143 indexed citations
17.
Kopp, Sarah J., Ghazal Banisadr, Kelly E. Glajch, et al.. (2009). Infection of neurons and encephalitis after intracranial inoculation of herpes simplex virus requires the entry receptor nectin-1. Proceedings of the National Academy of Sciences. 106(42). 17916–17920. 78 indexed citations
18.
Sadri‐Vakili, Ghazaleh, Caroline Benn, Ryan Overland, et al.. (2007). Histones associated with downregulated genes are hypo-acetylated in Huntington's disease models. Human Molecular Genetics. 16(11). 1293–1306. 170 indexed citations
19.
Guillozet-Bongaarts, Angela, Kelly E. Glajch, Michael E. Cahill, et al.. (2007). Phosphorylation and cleavage of tau in non-AD tauopathies. Acta Neuropathologica. 113(5). 513–520. 55 indexed citations
20.
Chen‐Plotkin, Alice, Ghazaleh Sadri‐Vakili, George J. Yohrling, et al.. (2006). Decreased association of the transcription factor Sp1 with genes downregulated in Huntington's disease. Neurobiology of Disease. 22(2). 233–241. 90 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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