Natalia Grinkina
- Molecular Biology
- Neurology top 10%
- Neurology top 10%
- Epidemiology
- Immunology
- Co-authors
- Peter J. BergoldRaj WadgaonkarMargalit HaberJohnson HoElena NikulinaJing LiuJoe G. N. GarciaSara A. Orsi
- Topics
- Traumatic Brain Injury and Neurovascular Disturbances (5 papers)Sphingolipid Metabolism and Signaling (5 papers)Neuroinflammation and Neurodegeneration Mechanisms (4 papers)
- Cited by
- NeurologyDevelopmental Neuroscience
- Partner nations
- United StatesPortugalGermany
In The Last Decade
Natalia Grinkina
13 papers receiving 494 citations
Peers
Comparison fields: 5 of 67
- Molecular Biology 279
- Neurology 169
- Neurology 110
- Epidemiology 103
- Immunology 73
Countries citing papers authored by Natalia Grinkina
This map shows the geographic impact of Natalia Grinkina's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Natalia Grinkina with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Natalia Grinkina more than expected).
Fields of papers citing papers by Natalia Grinkina
This network shows the impact of papers produced by Natalia Grinkina. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Natalia Grinkina. The network helps show where Natalia Grinkina may publish in the future.
Co-authorship network of co-authors of Natalia Grinkina
This figure shows the co-authorship network connecting the top 25 collaborators of Natalia Grinkina. A scholar is included among the top collaborators of Natalia Grinkina based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Natalia Grinkina. Natalia Grinkina is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 47 | |
| 2 | 33 | |
| 3 | 55 | |
| 4 | 29 | |
| 5 | 12 | |
| 6 | The anti-cancer peptide, PNC-27, induces tumor cell necrosis of a poorly differentiated non-solid tissue human leukemia cell line that depends on expression of HDM-2 in the plasma membrane of these cells. | 14 |
| 7 | 22 | |
| 8 | 84 | |
| 9 | 19 | |
| 10 | 35 | |
| 11 | 30 | |
| 12 | 42 | |
| 13 | 78 |
About Natalia Grinkina
Natalia Grinkina is a scholar working on Neurology, Developmental Neuroscience and Neurology, having authored 13 papers that have together received 500 indexed citations. Recurring topics across this work include Traumatic Brain Injury and Neurovascular Disturbances (5 papers), Sphingolipid Metabolism and Signaling (5 papers) and Neuroinflammation and Neurodegeneration Mechanisms (4 papers). The work is most often cited by research in Neurology (110 citations), Neurology (169 citations) and Developmental Neuroscience (44 citations). Natalia Grinkina has collaborated with scholars based in United States, Portugal and Germany. Frequent co-authors include Peter J. Bergold, Raj Wadgaonkar, Margalit Haber, Johnson Ho, Elena Nikulina, Jing Liu, Joe G. N. Garcia, Sara A. Orsi, Carol A. Romano and Saad Sammani. Their work appears in journals such as PLoS ONE, Journal of Cerebral Blood Flow & Metabolism and Experimental Neurology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.