Nassima Fodil

1.7k total citations · 1 hit paper
22 papers, 1.2k citations indexed

About

Nassima Fodil is a scholar working on Immunology, Genetics and Molecular Biology. According to data from OpenAlex, Nassima Fodil has authored 22 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Immunology, 7 papers in Genetics and 4 papers in Molecular Biology. Recurrent topics in Nassima Fodil's work include Immune Cell Function and Interaction (11 papers), T-cell and B-cell Immunology (8 papers) and Immunotherapy and Immune Responses (4 papers). Nassima Fodil is often cited by papers focused on Immune Cell Function and Interaction (11 papers), T-cell and B-cell Immunology (8 papers) and Immunotherapy and Immune Responses (4 papers). Nassima Fodil collaborates with scholars based in Canada, France and United States. Nassima Fodil's co-authors include Seiamak Bahram, Renato C. Monteiro, Virginie Verkarre, Nadine Cerf–Bensussan, Shaoling Zhang, Sophie Hüe, Christophe Cellier, Sophie Caillat‐Zucman, Jacques Schmitz and Philippe Gros and has published in prestigious journals such as Nature Communications, The Journal of Experimental Medicine and Immunity.

In The Last Decade

Nassima Fodil

22 papers receiving 1.2k citations

Hit Papers

A Direct Role for NKG2D/M... 2004 2026 2011 2018 2004 100 200 300 400 500

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Nassima Fodil Canada 14 728 331 305 206 150 22 1.2k
Cezary Ciszewski United States 12 884 1.2× 667 2.0× 505 1.7× 72 0.3× 281 1.9× 15 1.4k
Allan Thompson Netherlands 17 716 1.0× 540 1.6× 376 1.2× 108 0.5× 206 1.4× 26 1.2k
I‐Ting Chow United States 16 583 0.8× 89 0.3× 113 0.4× 243 1.2× 278 1.9× 27 1.1k
Nobumasa Watanabe Japan 13 886 1.2× 71 0.2× 157 0.5× 378 1.8× 59 0.4× 19 1.3k
Britt‐Sabina Petersen Germany 15 269 0.4× 38 0.1× 124 0.4× 270 1.3× 117 0.8× 24 885
M Y Choy United Kingdom 9 299 0.4× 41 0.1× 231 0.8× 129 0.6× 125 0.8× 9 795
Barry Healy United Kingdom 15 579 0.8× 56 0.2× 79 0.3× 379 1.8× 346 2.3× 20 1.5k
Stefanie Groß Germany 14 512 0.7× 33 0.1× 80 0.3× 416 2.0× 65 0.4× 25 986
Ralph Carvalho Netherlands 16 568 0.8× 59 0.2× 101 0.3× 377 1.8× 863 5.8× 17 1.5k
Noor Dawany United States 16 243 0.3× 20 0.1× 177 0.6× 286 1.4× 141 0.9× 28 815

Countries citing papers authored by Nassima Fodil

Since Specialization
Citations

This map shows the geographic impact of Nassima Fodil's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nassima Fodil with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nassima Fodil more than expected).

Fields of papers citing papers by Nassima Fodil

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Nassima Fodil. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nassima Fodil. The network helps show where Nassima Fodil may publish in the future.

Co-authorship network of co-authors of Nassima Fodil

This figure shows the co-authorship network connecting the top 25 collaborators of Nassima Fodil. A scholar is included among the top collaborators of Nassima Fodil based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nassima Fodil. Nassima Fodil is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Langlais, David, Maria J. Polyak, Luc Galarneau, et al.. (2024). CCDC88B interacts with RASAL3 and ARHGEF2 and regulates dendritic cell function in neuroinflammation and colitis. Communications Biology. 7(1). 77–77. 3 indexed citations
2.
Martinez, Sébastien, Ahmad Malik, Ramona Weber, et al.. (2024). In vivo CRISPR screens reveal SCAF1 and USP15 as drivers of pancreatic cancer. Nature Communications. 15(1). 5266–5266. 8 indexed citations
3.
Yang, Yunxiang, et al.. (2023). Biphosphoglycerate Mutase: A Novel Therapeutic Target for Malaria?. Transfusion Medicine Reviews. 37(3). 150748–150748. 3 indexed citations
4.
Gros, Philippe, et al.. (2021). USP15: a review of its implication in immune and inflammatory processes and tumor progression. Genes and Immunity. 22(1). 12–23. 31 indexed citations
5.
Scholtes, Charlotte, Claudia Champagne, Luc Galarneau, et al.. (2021). Estrogen-related receptor alpha (ERRα) is a key regulator of intestinal homeostasis and protects against colitis. Scientific Reports. 11(1). 15073–15073. 19 indexed citations
6.
Fodil, Nassima, et al.. (2020). CCDC88B is required for mobility and inflammatory functions of dendritic cells. Journal of Leukocyte Biology. 108(6). 1787–1802. 7 indexed citations
7.
Fodil, Nassima, Lauren Van Der Kraak, Charles Meunier, et al.. (2019). Inactivation of Interferon Regulatory Factor 1 Causes Susceptibility to Colitis-Associated Colorectal Cancer. Scientific Reports. 9(1). 18897–18897. 17 indexed citations
8.
Teyra, Joan, Alex U. Singer, Frank W. Schmitges, et al.. (2019). Structural and Functional Characterization of Ubiquitin Variant Inhibitors of USP15. Structure. 27(4). 590–605.e5. 43 indexed citations
9.
Fodil, Nassima, Irena Radovanovic, Manuel Flores Molina, et al.. (2017). CCDC88B is required for pathogenesis of inflammatory bowel disease. Nature Communications. 8(1). 932–932. 23 indexed citations
10.
Petrov, Jessica C., et al.. (2017). A role for the histone H2A deubiquitinase MYSM1 in maintenance of CD8+ T cells. Immunology. 151(1). 110–121. 13 indexed citations
11.
Fodil, Nassima, David Langlais, & Philippe Gros. (2016). Primary Immunodeficiencies and Inflammatory Disease: A Growing Genetic Intersection. Trends in Immunology. 37(2). 126–140. 40 indexed citations
12.
Langlais, David, Nassima Fodil, & Philippe Gros. (2016). Genetics of Infectious and Inflammatory Diseases: Overlapping Discoveries from Association and Exome-Sequencing Studies. Annual Review of Immunology. 35(1). 1–30. 33 indexed citations
13.
Abrahamsen, Greger, Nassima Fodil, Erik Dissen, et al.. (2015). Reduced MCMV Δm157 viral clearance in the absence of TSAd. Scientific Reports. 5(1). 9219–9219. 4 indexed citations
14.
Fodil, Nassima, David Langlais, Irena Radovanovic, et al.. (2014). Specific Dysregulation of IFNγ Production by Natural Killer Cells Confers Susceptibility to Viral Infection. PLoS Pathogens. 10(12). e1004511–e1004511. 13 indexed citations
15.
Kennedy, James, Nassima Fodil, Silayuv E. Bongfen, et al.. (2014). CCDC88B is a novel regulator of maturation and effector functions of T cells during pathological inflammation. The Journal of Experimental Medicine. 211(13). 2519–2535. 38 indexed citations
16.
Nataf, Serge, Adrienne Anginot, Carine Vuaillat, et al.. (2005). Brain and Bone Damage in KARAP/DAP12 Loss-of-Function Mice Correlate with Alterations in Microglia and Osteoclast Lineages. American Journal Of Pathology. 166(1). 275–286. 65 indexed citations
17.
Hüe, Sophie, Renato C. Monteiro, Shaoling Zhang, et al.. (2004). A Direct Role for NKG2D/MICA Interaction in Villous Atrophy during Celiac Disease. Immunity. 21(3). 367–377. 549 indexed citations breakdown →
18.
Fodil, Nassima, Philippe Pellet, Laurent Laloux, et al.. (1999). MICA haplotypic diversity. Immunogenetics. 49(6). 557–560. 53 indexed citations
19.
Renaud, Marc, Nassima Fodil, Laurent Laloux, et al.. (1997). Allelic repertoire of the human MICB gene. Immunogenetics. 46(5). 434–436. 41 indexed citations
20.
Fodil, Nassima, Laurent Laloux, Philippe Pellet, et al.. (1996). Allelic repertoire of the humanMHC class IMICA gene. Immunogenetics. 44(5). 351–357. 146 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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