Nanda R. Rodrigues
- Molecular Biology top 5%
- Genetics top 2%
- Oncology top 5%
- Surgery top 5%
- Genetics top 1%
- Co-authors
- R. Duncan CampbellRichard G.H. CottonWalter F. BodmerI B KerrDavid P. LaneAndrew RowanJulian GannonMark E. Smith
- Topics
- Neurogenetic and Muscular Disorders Research (10 papers)RNA modifications and cancer (9 papers)Diabetes and associated disorders (8 papers)
- Cited by
- GeneticsMolecular Biology
- Partner nations
- United KingdomUnited StatesBrazil
In The Last Decade
Nanda R. Rodrigues
33 papers receiving 3.8k citations
Hit Papers
Peers
Comparison fields: 5 of 103
- Molecular Biology 2.3k
- Genetics 1.1k
- Oncology 765
- Surgery 754
- Genetics 606
Countries citing papers authored by Nanda R. Rodrigues
This map shows the geographic impact of Nanda R. Rodrigues's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Nanda R. Rodrigues with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Nanda R. Rodrigues more than expected).
Fields of papers citing papers by Nanda R. Rodrigues
This network shows the impact of papers produced by Nanda R. Rodrigues. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Nanda R. Rodrigues. The network helps show where Nanda R. Rodrigues may publish in the future.
Co-authorship network of co-authors of Nanda R. Rodrigues
This figure shows the co-authorship network connecting the top 25 collaborators of Nanda R. Rodrigues. A scholar is included among the top collaborators of Nanda R. Rodrigues based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Nanda R. Rodrigues. Nanda R. Rodrigues is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | Genetic analysis of autoimmune type 1 diabetes mellitus in mice. 1991. | 0 |
| 2 | 25 | |
| 3 | 133 | |
| 4 | 126 | |
| 5 | 4 | |
| 6 | 13 | |
| 7 | 341 | |
| 8 | 7 | |
| 9 | MOLECULAR ANALYSIS OF CHILDHOOD-ONSET SPINAL MUSCULAR-ATROPHY | 3 |
| 10 | 24 | |
| 11 | 14 | |
| 12 | 36 | |
| 13 | 251 | |
| 14 | 11 | |
| 15 | 3 | |
| 16 | 36 | |
| 17 | 16 | |
| 18 | 103 | |
| 19 | 411 | |
| 20 | 19 |
About Nanda R. Rodrigues
Nanda R. Rodrigues is a scholar working on Genetics, Genetics and Molecular Biology, having authored 34 papers that have together received 3.8k indexed citations. Recurring topics across this work include Neurogenetic and Muscular Disorders Research (10 papers), RNA modifications and cancer (9 papers) and Diabetes and associated disorders (8 papers). The work is most often cited by research in Genetics (606 citations), Genetics (1.1k citations) and Molecular Biology (2.3k citations). Nanda R. Rodrigues has collaborated with scholars based in United Kingdom, United States and Brazil. Frequent co-authors include R. Duncan Campbell, Richard G.H. Cotton, Walter F. Bodmer, I B Kerr, David P. Lane, Andrew Rowan, Julian Gannon, Mark E. Smith, John A. Todd and Jan‐Bas Prins. Their work appears in journals such as Nature, Science and Proceedings of the National Academy of Sciences.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.