Linda S. Wicker

18.9k total citations
180 papers, 10.9k citations indexed

About

Linda S. Wicker is a scholar working on Genetics, Immunology and Surgery. According to data from OpenAlex, Linda S. Wicker has authored 180 papers receiving a total of 10.9k indexed citations (citations by other indexed papers that have themselves been cited), including 122 papers in Genetics, 105 papers in Immunology and 74 papers in Surgery. Recurrent topics in Linda S. Wicker's work include Diabetes and associated disorders (113 papers), T-cell and B-cell Immunology (73 papers) and Pancreatic function and diabetes (70 papers). Linda S. Wicker is often cited by papers focused on Diabetes and associated disorders (113 papers), T-cell and B-cell Immunology (73 papers) and Pancreatic function and diabetes (70 papers). Linda S. Wicker collaborates with scholars based in United States, United Kingdom and Germany. Linda S. Wicker's co-authors include John A. Todd, Laurence B. Peterson, B J Miller, Paul Lyons, M C Appel, Patricia L. Podolin, Jan‐Bas Prins, Yoko Mullen, Neil Walker and Daniel B. Rainbow and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Linda S. Wicker

178 papers receiving 10.6k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Linda S. Wicker United States 59 6.1k 5.7k 3.5k 2.1k 2.0k 180 10.9k
Laurence B. Peterson United States 43 3.1k 0.5× 2.7k 0.5× 2.0k 0.6× 1.3k 0.6× 859 0.4× 102 5.7k
Roland Tisch United States 44 4.1k 0.7× 5.0k 0.9× 2.3k 0.7× 1.2k 0.6× 1.6k 0.8× 130 8.2k
Naoto Itoh Japan 26 1.3k 0.2× 3.5k 0.6× 1.1k 0.3× 4.2k 2.0× 644 0.3× 50 7.9k
Manuela Battaglia Italy 39 1.8k 0.3× 5.2k 0.9× 1.1k 0.3× 1.2k 0.6× 594 0.3× 110 7.8k
Eugen Koren United States 37 1.3k 0.2× 1.8k 0.3× 2.7k 0.8× 1.7k 0.8× 672 0.3× 104 5.8k
Lucy S. K. Walker United Kingdom 48 1.4k 0.2× 6.6k 1.2× 635 0.2× 1.2k 0.6× 459 0.2× 91 9.1k
Raquel Seruca Portugal 63 1.3k 0.2× 1.6k 0.3× 3.9k 1.1× 6.3k 3.0× 643 0.3× 236 13.1k
Katherine A. Siminovitch Canada 52 1.6k 0.3× 4.2k 0.7× 626 0.2× 3.6k 1.7× 128 0.1× 136 8.8k
Peter C. Lucas United States 43 1.1k 0.2× 1.7k 0.3× 1.2k 0.3× 4.2k 2.0× 446 0.2× 163 8.4k
Kristin A. Hogquist United States 69 1.2k 0.2× 15.8k 2.8× 586 0.2× 4.1k 1.9× 561 0.3× 162 19.8k

Countries citing papers authored by Linda S. Wicker

Since Specialization
Citations

This map shows the geographic impact of Linda S. Wicker's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Linda S. Wicker with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Linda S. Wicker more than expected).

Fields of papers citing papers by Linda S. Wicker

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Linda S. Wicker. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Linda S. Wicker. The network helps show where Linda S. Wicker may publish in the future.

Co-authorship network of co-authors of Linda S. Wicker

This figure shows the co-authorship network connecting the top 25 collaborators of Linda S. Wicker. A scholar is included among the top collaborators of Linda S. Wicker based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Linda S. Wicker. Linda S. Wicker is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Marcovecchio, M. Loredana, Emile Hendriks, Tadej Battelino, et al.. (2024). The INNODIA Type 1 Diabetes Natural History Study: a European cohort of newly diagnosed children, adolescents and adults. Diabetologia. 67(6). 995–1008. 14 indexed citations
2.
Zhang, Jiayuan, Justin P. Whalley, Julian C. Knight, et al.. (2023). SARS-CoV-2 infection induces a long-lived pro-inflammatory transcriptional profile. Genome Medicine. 15(1). 69–69. 19 indexed citations
3.
Zhang, Jiayuan, Fiona Hamey, Dominik Trzupek, et al.. (2022). Low-dose IL-2 reduces IL-21+ T cell frequency and induces anti-inflammatory gene expression in type 1 diabetes. Nature Communications. 13(1). 7324–7324. 18 indexed citations
4.
Inshaw, Jamie, Antony J. Cutler, Daniel J. M. Crouch, Linda S. Wicker, & John A. Todd. (2019). Genetic Variants Predisposing Most Strongly to Type 1 Diabetes Diagnosed Under Age 7 Years Lie Near Candidate Genes That Function in the Immune System and in Pancreatic β-Cells. Diabetes Care. 43(1). 169–177. 75 indexed citations
5.
Seelig, Eleonora, Linsey Porter, Lucy Truman, et al.. (2018). The DILfrequency study is an adaptive trial to identify optimal IL-2 dosing in patients with type 1 diabetes. JCI Insight. 3(19). 29 indexed citations
6.
Maine, Christian J., Emma E. Hamilton‐Williams, Jocelyn Cheung, et al.. (2012). PTPN22 Alters the Development of Regulatory T Cells in the Thymus. The Journal of Immunology. 188(11). 5267–5275. 96 indexed citations
7.
Kissler, Stephan, Kay A. Fischer, Peilin Zheng, Dan Rainbow, & Linda S. Wicker. (2009). The soluble CTLA-4 splice variant affects the function of CD4+CD25+ regulatory T cells (49.10). The Journal of Immunology. 182(Supplement_1). 49.10–49.10. 2 indexed citations
8.
Araki, M., Denise Chung, Sue Min Liu, et al.. (2009). Genetic Evidence That the Differential Expression of the Ligand-Independent Isoform of CTLA-4 Is the Molecular Basis of the Idd5.1 Type 1 Diabetes Region in Nonobese Diabetic Mice. The Journal of Immunology. 183(8). 5146–5157. 54 indexed citations
9.
Ridgway, William M., Laurence B. Peterson, John A. Todd, et al.. (2008). Chapter 6 Gene–Gene Interactions in the NOD Mouse Model of Type 1 Diabetes. Advances in immunology. 100. 151–175. 56 indexed citations
10.
Maier, Lisa M., Sarah Howlett, Kara Rainbow, et al.. (2008). NKG2D-RAE-1 Receptor-Ligand Variation Does Not Account for the NK Cell Defect in Nonobese Diabetic Mice. The Journal of Immunology. 181(10). 7073–7080. 13 indexed citations
11.
Hunter, Kara, Dan Rainbow, Vincent Plagnol, et al.. (2007). Interactions between Idd5.1/Ctla4 and Other Type 1 Diabetes Genes. The Journal of Immunology. 179(12). 8341–8349. 45 indexed citations
12.
Silveira, Pablo A., Harold D. Chapman, Jessica Stolp, et al.. (2006). Genes within the Idd5 and Idd9/11 Diabetes Susceptibility Loci Affect the Pathogenic Activity of B Cells in Nonobese Diabetic Mice. The Journal of Immunology. 177(10). 7033–7041. 25 indexed citations
13.
Martínez, Xavier, Huub T. C. Kreuwel, William L. Redmond, et al.. (2005). CD8+ T Cell Tolerance in Nonobese Diabetic Mice Is Restored by Insulin-Dependent Diabetes Resistance Alleles. The Journal of Immunology. 175(3). 1677–1685. 33 indexed citations
14.
Greve, Bernhard, Lalitha Vijayakrishnan, Raymond A. Sobel, et al.. (2004). The Diabetes Susceptibility Locus Idd5.1 on Mouse Chromosome 1 Regulates ICOS Expression and Modulates Murine Experimental Autoimmune Encephalomyelitis. The Journal of Immunology. 173(1). 157–163. 48 indexed citations
15.
Wicker, Linda S., Giselle Chamberlain, Kara Hunter, et al.. (2004). Fine Mapping, Gene Content, Comparative Sequencing, and Expression Analyses Support Ctla4 and Nramp1 as Candidates for Idd5.1 and Idd5.2 in the Nonobese Diabetic Mouse. The Journal of Immunology. 173(1). 164–173. 91 indexed citations
16.
Pearson, Todd, Thomas G. Markees, David Serreze, et al.. (2003). Genetic Disassociation of Autoimmunity and Resistance to Costimulation Blockade-Induced Transplantation Tolerance in Nonobese Diabetic Mice. The Journal of Immunology. 171(1). 185–195. 59 indexed citations
17.
Wicker, Linda S., S L Chen, Gerald T. Nepom, et al.. (1996). Naturally processed T cell epitopes from human glutamic acid decarboxylase identified using mice transgenic for the type 1 diabetes-associated human MHC class II allele, DRB1*0401.. Journal of Clinical Investigation. 98(11). 2597–2603. 119 indexed citations
18.
Ghosh, Soumitra, Sheila Palmer, Nanda R. Rodrigues, et al.. (1993). Polygenic control of autoimmune diabetes in nonobese diabetic mice. Nature Genetics. 4(4). 404–409. 251 indexed citations
19.
Koo, G C, et al.. (1988). Activation of murine natural killer cells and macrophages by 8-bromoguanosine.. The Journal of Immunology. 140(9). 3249–3252. 29 indexed citations
20.
Clark, Edward A., Richard C. Harmon, & Linda S. Wicker. (1977). Resistance of H-2 heterozygous mice to parental tumors. II. Characterization of Hh-1 controlled hybrid resistance to syngeneic fibrosarcomas and the EL-4 lymphoma.. Munich Personal RePEc Archive (Ludwig Maximilian University of Munich). 119(2). 648–56. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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