Murray C.H. Clarke

5.3k total citations · 2 hit papers
40 papers, 4.1k citations indexed

About

Murray C.H. Clarke is a scholar working on Immunology, Molecular Biology and Immunology and Allergy. According to data from OpenAlex, Murray C.H. Clarke has authored 40 papers receiving a total of 4.1k indexed citations (citations by other indexed papers that have themselves been cited), including 33 papers in Immunology, 17 papers in Molecular Biology and 10 papers in Immunology and Allergy. Recurrent topics in Murray C.H. Clarke's work include Atherosclerosis and Cardiovascular Diseases (13 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (10 papers) and Cell Adhesion Molecules Research (10 papers). Murray C.H. Clarke is often cited by papers focused on Atherosclerosis and Cardiovascular Diseases (13 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (10 papers) and Cell Adhesion Molecules Research (10 papers). Murray C.H. Clarke collaborates with scholars based in United Kingdom, United States and Portugal. Murray C.H. Clarke's co-authors include Martin R. Bennett, Nichola Figg, Janet J. Maguire, Trevor D. Littlewood, Helle F. Jørgensen, Ziad Mallat, Gemma Basatemur, Melanie Humphry, Anthony P. Davenport and Martin Goddard and has published in prestigious journals such as Journal of Biological Chemistry, Circulation and Nature Medicine.

In The Last Decade

Murray C.H. Clarke

39 papers receiving 4.1k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Vascular smooth muscle cells in atherosclerosis

2019 768 citations Helle F. Jørgensen, Murray C.H. Clarke et al. profile →
Apoptosis of vascular smooth muscle cells induces features of plaque vulnerability in atherosclerosis

2006 538 citations Murray C.H. Clarke, Nichola Figg et al. Nature Medicine profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Murray C.H. Clarke United Kingdom	25	2.0k	1.7k	573	547	538	40	4.1k
Coleen A. McNamara United States	42	2.2k 1.1×	2.0k 1.2×	633 1.1×	478 0.9×	787 1.5×	114	5.3k
Arif Yurdagul United States	28	1.7k 0.8×	2.5k 1.5×	459 0.8×	634 1.2×	675 1.3×	50	4.8k
Joseph J. Boyle United Kingdom	36	1.3k 0.6×	1.4k 0.8×	364 0.6×	310 0.6×	587 1.1×	66	3.4k
Philipp J. Hohensinner Austria	30	1.4k 0.7×	925 0.6×	527 0.9×	354 0.6×	391 0.7×	76	3.1k
Shiro Kitamoto Japan	31	1.3k 0.7×	1.2k 0.8×	608 1.1×	507 0.9×	451 0.8×	52	3.8k
Yvonne Döring Germany	33	1.3k 0.6×	2.4k 1.5×	324 0.6×	256 0.5×	574 1.1×	85	4.0k
Mitsuhiko Okigaki Japan	34	1.7k 0.9×	834 0.5×	323 0.6×	354 0.6×	463 0.9×	65	4.2k
Bhama Ramkhelawon United States	30	2.3k 1.2×	2.0k 1.2×	933 1.6×	496 0.9×	928 1.7×	63	5.2k
Amanda C. Doran United States	24	1.4k 0.7×	2.5k 1.5×	393 0.7×	513 0.9×	748 1.4×	38	4.2k
Qingzhong Xiao United Kingdom	44	3.3k 1.7×	1.1k 0.6×	1.5k 2.5×	437 0.8×	546 1.0×	128	5.9k

Countries citing papers authored by Murray C.H. Clarke

Since Specialization

Citations

This map shows the geographic impact of Murray C.H. Clarke's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Murray C.H. Clarke with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Murray C.H. Clarke more than expected).

Fields of papers citing papers by Murray C.H. Clarke

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Murray C.H. Clarke. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Murray C.H. Clarke. The network helps show where Murray C.H. Clarke may publish in the future.

Co-authorship network of co-authors of Murray C.H. Clarke

This figure shows the co-authorship network connecting the top 25 collaborators of Murray C.H. Clarke. A scholar is included among the top collaborators of Murray C.H. Clarke based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Murray C.H. Clarke. Murray C.H. Clarke is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Mahbubani, Krishnaa T., Kirsty Foote, Nichola Figg, et al.. (2025). Delineation of a thrombin receptor-stimulated vascular smooth muscle cell transition generating cells in the plaque-stabilizing fibrous cap. Cardiovascular Research. 121(9). 1359–1372. 1 indexed citations

Rahman, Jubayer, Jack Bibby, Parul Singh, et al.. (2025). A CD4+ T cell-intrinsic complement C5aR2-prostacyclin-IL-1R2 axis orchestrates Th1 contraction and is perturbed in Th1-driven disease states. Immunobiology. 230(4). 152979–152979.

Pyrillou, Katerina, Melanie Humphry, Meritxell Nus, et al.. (2024). Loss of T follicular regulatory cell-derived IL-1R2 augments germinal centre reactions via increased IL-1. JCI Insight. 9(5). 2 indexed citations

Burzynski, Laura C., et al.. (2023). Thrombin-activated interleukin-1α drives atherogenesis, but also promotes vascular smooth muscle cell proliferation and collagen production. Cardiovascular Research. 119(12). 2179–2189. 6 indexed citations

Cassidy, Liam D., Andrew Young, Chris Young, et al.. (2020). Temporal inhibition of autophagy reveals segmental reversal of ageing with increased cancer risk. Nature Communications. 11(1). 307–307. 77 indexed citations

Foote, Kirsty, Nichola Figg, Alison Finigan, et al.. (2020). Cytokine regulation of apoptosis-induced apoptosis and apoptosis-induced cell proliferation in vascular smooth muscle cells. APOPTOSIS. 25(9-10). 648–662. 28 indexed citations

Wiggins, Kimberley A., Aled Parry, Liam D. Cassidy, et al.. (2019). IL‐1α cleavage by inflammatory caspases of the noncanonical inflammasome controls the senescence‐associated secretory phenotype. Aging Cell. 18(3). e12946–e12946. 82 indexed citations

Wang, Julie, Anna Uryga, Johannes Reinhold, et al.. (2015). Vascular Smooth Muscle Cell Senescence Promotes Atherosclerosis and Features of Plaque Vulnerability. Circulation. 132(20). 1909–1919. 275 indexed citations

Burzynski, Laura C., Melanie Humphry, Martin R. Bennett, & Murray C.H. Clarke. (2015). 191 Interleukin-1 Alpha is Directly Activated During Haemostasis by Thrombin Cleavage. A107.1–A107. 1 indexed citations

10.

Burzynski, Laura C., Melanie Humphry, Martin R. Bennett, & Murray C.H. Clarke. (2015). Interleukin-1α Activity in Necrotic Endothelial Cells Is Controlled by Caspase-1 Cleavage of Interleukin-1 Receptor-2. Journal of Biological Chemistry. 290(41). 25188–25196. 21 indexed citations

11.

Yu, Emma, Patrick A. Calvert, John R. Mercer, et al.. (2013). Mitochondrial DNA Damage Can Promote Atherosclerosis Independently of Reactive Oxygen Species Through Effects on Smooth Muscle Cells and Monocytes and Correlates With Higher-Risk Plaques in Humans. Circulation. 128(7). 702–712. 232 indexed citations

12.

Zheng, Yue, Melanie Humphry, Janet J. Maguire, Martin R. Bennett, & Murray C.H. Clarke. (2013). Intracellular Interleukin-1 Receptor 2 Binding Prevents Cleavage and Activity of Interleukin-1α, Controlling Necrosis-Induced Sterile Inflammation. Immunity. 38(2). 285–295. 168 indexed citations

13.

Maguire, Janet J., K. L. Jones, Rhoda E. Kuc, et al.. (2013). The CCR5 chemokine receptor mediates vasoconstriction and stimulates intimal hyperplasia in human vessels in vitro. Cardiovascular Research. 101(3). 513–521. 25 indexed citations

14.

Bennett, Martin R., Haixiang Yu, & Murray C.H. Clarke. (2012). Signalling from dead cells drives inflammation and vessel remodelling. Vascular Pharmacology. 56(5-6). 187–192. 24 indexed citations

15.

Yu, Haixiang, Murray C.H. Clarke, Nichola Figg, Trevor D. Littlewood, & Martin R. Bennett. (2011). Smooth Muscle Cell Apoptosis Promotes Vessel Remodeling and Repair via Activation of Cell Migration, Proliferation, and Collagen Synthesis. Arteriosclerosis Thrombosis and Vascular Biology. 31(11). 2402–2409. 58 indexed citations

16.

Calvert, Patrick A., Isabelle Gorenne, Murray C.H. Clarke, et al.. (2011). Leukocyte Telomere Length Is Associated With High-Risk Plaques on Virtual Histology Intravascular Ultrasound and Increased Proinflammatory Activity. Arteriosclerosis Thrombosis and Vascular Biology. 31(9). 2157–2164. 60 indexed citations

17.

Clarke, Murray C.H., Nichola Figg, Janet J. Maguire, et al.. (2006). Apoptosis of vascular smooth muscle cells induces features of plaque vulnerability in atherosclerosis. Nature Medicine. 12(9). 1075–1080. 538 indexed citations breakdown →

18.

Clarke, Murray C.H. & Martin R. Bennett. (2006). Defining the Role of Vascular Smooth Muscle Cell Apoptosis in Atherosclerosis. Cell Cycle. 5(20). 2329–2331. 29 indexed citations

19.

Wickenden, Julie A., Murray C.H. Clarke, Adriano G. Rossi, et al.. (2003). Cigarette Smoke Prevents Apoptosis through Inhibition of Caspase Activation and Induces Necrosis. American Journal of Respiratory Cell and Molecular Biology. 29(5). 562–570. 107 indexed citations

20.

Brown, Simon, et al.. (2000). Constitutive Death of Platelets Leading to Scavenger Receptor-mediated Phagocytosis. Journal of Biological Chemistry. 275(8). 5987–5996. 141 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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