Yingbi Zhou

1.3k total citations
49 papers, 1.1k citations indexed

About

Yingbi Zhou is a scholar working on Physiology, Pharmacology and Biochemistry. According to data from OpenAlex, Yingbi Zhou has authored 49 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 27 papers in Physiology, 25 papers in Pharmacology and 25 papers in Biochemistry. Recurrent topics in Yingbi Zhou's work include Nitric Oxide and Endothelin Effects (27 papers), Eicosanoids and Hypertension Pharmacology (25 papers) and Inflammatory mediators and NSAID effects (25 papers). Yingbi Zhou is often cited by papers focused on Nitric Oxide and Endothelin Effects (27 papers), Eicosanoids and Hypertension Pharmacology (25 papers) and Inflammatory mediators and NSAID effects (25 papers). Yingbi Zhou collaborates with scholars based in China, United States and United Kingdom. Yingbi Zhou's co-authors include Wenhong Luo, Muthu Periasamy, Bin Liu, Wessel P. Dirksen, Jay L. Zweíer, Bin Liu, Gopal J. Babu, Dongyang Huang, Saradhadevi Varadharaj and Nicholas A. Flavahan and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of the American College of Cardiology.

In The Last Decade

Yingbi Zhou

48 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Yingbi Zhou China 21 399 364 349 303 224 49 1.1k
Daniel Abran Canada 25 407 1.0× 222 0.6× 223 0.6× 125 0.4× 388 1.7× 38 1.4k
T Satake Japan 18 409 1.0× 202 0.6× 84 0.2× 302 1.0× 362 1.6× 66 1.0k
Philip G. Baer United States 17 166 0.4× 170 0.5× 182 0.5× 183 0.6× 367 1.6× 47 1.2k
Kumi Nakamura Japan 22 330 0.8× 163 0.4× 93 0.3× 191 0.6× 286 1.3× 64 1.0k
Ana Sánchez Spain 16 271 0.7× 76 0.2× 62 0.2× 204 0.7× 214 1.0× 30 800
Jennifer L. Busch United States 5 330 0.8× 70 0.2× 70 0.2× 173 0.6× 421 1.9× 9 864
Ming-Hui Zou United States 11 357 0.9× 80 0.2× 196 0.6× 185 0.6× 423 1.9× 14 1.1k
Uffe Bang Olsen Denmark 18 260 0.7× 106 0.3× 212 0.6× 173 0.6× 526 2.3× 50 1.4k
Tinatin Chabrashvili United States 9 393 1.0× 132 0.4× 45 0.1× 299 1.0× 364 1.6× 12 1.0k
Achim Feurer Germany 11 503 1.3× 93 0.3× 43 0.1× 291 1.0× 445 2.0× 14 1.1k

Countries citing papers authored by Yingbi Zhou

Since Specialization
Citations

This map shows the geographic impact of Yingbi Zhou's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Yingbi Zhou with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Yingbi Zhou more than expected).

Fields of papers citing papers by Yingbi Zhou

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Yingbi Zhou. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Yingbi Zhou. The network helps show where Yingbi Zhou may publish in the future.

Co-authorship network of co-authors of Yingbi Zhou

This figure shows the co-authorship network connecting the top 25 collaborators of Yingbi Zhou. A scholar is included among the top collaborators of Yingbi Zhou based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Yingbi Zhou. Yingbi Zhou is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Rahman, Jubayer, Jack Bibby, Parul Singh, et al.. (2025). A CD4+ T cell-intrinsic complement C5aR2-prostacyclin-IL-1R2 axis orchestrates Th1 contraction and is perturbed in Th1-driven disease states. Immunobiology. 230(4). 152979–152979.
4.
Pires, Maria Elisa Lopes, Paul C. Armstrong, Blerina Ahmetaj‐Shala, et al.. (2023). Widening the Prostacyclin Paradigm: Tissue Fibroblasts Are a Critical Site of Production and Antithrombotic Protection. Arteriosclerosis Thrombosis and Vascular Biology. 44(1). 271–286. 5 indexed citations
5.
Vaja, Ricky, Maria Elisa Lopes Pires, Marilena Crescente, et al.. (2023). Renal Function Underpins the Cyclooxygenase-2: Asymmetric Dimethylarginine Axis in Mouse and Man. Kidney International Reports. 8(6). 1231–1238. 3 indexed citations
6.
Liu, Bin, et al.. (2020). Effects of thromboxane prostanoid receptor deficiency on diabetic nephropathy induced by high fat diet and streptozotocin in mice. European Journal of Pharmacology. 882. 173254–173254. 6 indexed citations
7.
Liu, Bin, et al.. (2019). EP3 Blockade Adds to the Effect of TP Deficiency in Alleviating Endothelial Dysfunction in Atherosclerotic Mouse Aortas. Frontiers in Physiology. 10. 1247–1247. 6 indexed citations
8.
Liu, Bin, et al.. (2018). Prostaglandin D2 evokes potent uterine contraction via the F prostanoid receptor in postpartum rats. European Journal of Pharmacology. 836. 11–17. 5 indexed citations
11.
12.
Liu, Yanmin, Qi Li, Gefei Liu, et al.. (2014). Oxidized Low-Density Lipoprotein Suppresses Expression of Prostaglandin E Receptor Subtype EP3 in Human THP-1 Macrophages. PLoS ONE. 9(10). e110828–e110828. 6 indexed citations
13.
Zhou, Yingbi, et al.. (2013). Cyclo‐oxygenase‐1 or ‐2‐mediated metabolism of arachidonic acid in endothelium‐dependent contraction of mouse arteries. Experimental Physiology. 98(7). 1225–1234. 20 indexed citations
14.
Liu, Bin, et al.. (2012). Effect of celecoxib on cyclooxygenase-1-mediated prostacyclin synthesis and endothelium-dependent contraction in mouse arteries. European Journal of Pharmacology. 698(1-3). 354–361. 10 indexed citations
15.
Liu, Bin, et al.. (2011). Involvement of cyclo‐oxygenase‐1‐mediated prostacyclin synthesis in the vasoconstrictor activity evoked by ACh in mouse arteries. Experimental Physiology. 97(2). 277–289. 50 indexed citations
16.
Świergiel, Artur H., Yingbi Zhou, & Alison M. Dunn. (2007). Effects of chronic footshock, restraint and corticotropin-releasing factor on freezing, ultrasonic vocalization and forced swim behavior in rats. Behavioural Brain Research. 183(2). 178–187. 50 indexed citations
17.
Zhou, Yingbi, Wessel P. Dirksen, Yung‐Ming Chen, et al.. (2005). A major role for AT receptor in mouse mesenteric resistance vessels and its distribution in heart and neuroendocrine tissues. Journal of Molecular and Cellular Cardiology. 38(4). 693–696. 18 indexed citations
18.
Babu, Gopal J., Gail J. Pyne, Yingbi Zhou, et al.. (2004). Isoform switching from SM-B to SM-A myosin results in decreased contractility and altered expression of thin filament regulatory proteins. American Journal of Physiology-Cell Physiology. 287(3). C723–C729. 33 indexed citations
19.
Zhou, Yingbi, Wessel P. Dirksen, Jay L. Zweíer, & Muthu Periasamy. (2004). Endothelin-1-induced responses in isolated mouse vessels: the expression and function of receptor types. American Journal of Physiology-Heart and Circulatory Physiology. 287(2). H573–H578. 30 indexed citations
20.
Rott, David, Jianhui Zhu, Mary Susan Burnett, et al.. (2003). Effects of MF-tricyclic, a selective cyclooxygenase-2 inhibitor, on atherosclerosis progression and susceptibility to cytomegalovirus replication in apolipoprotein-E knockout mice. Journal of the American College of Cardiology. 41(10). 1812–1819. 68 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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