Müge Kıray
- Physiology top 5%
- Molecular Biology
- Pediatrics, Perinatology and Child Health top 10%
- Developmental Neuroscience top 5%
- Cellular and Molecular Neuroscience top 10%
- Co-authors
- Nazan Uysalİlkay AksuBaşak BaykaraAlper BağrıyanıkÇetin PekçetinBekir Uğur ErgürCaner ÇetinkayaAli Rıza Şişman
- Topics
- Stress Responses and Cortisol (10 papers)Adipose Tissue and Metabolism (8 papers)Cardiac Ischemia and Reperfusion (8 papers)
- Journals
- SHILAP Revista de lepidopterologíaBehavioural Brain ResearchUrology
- Partner nations
- TürkiyeUnited StatesMalaysia
In The Last Decade
Müge Kıray
72 papers receiving 1.5k citations
Peers
Comparison fields: 5 of 116
- Physiology 368
- Molecular Biology 271
- Pediatrics, Perinatology and Child Health 165
- Developmental Neuroscience 164
- Cellular and Molecular Neuroscience 161
Countries citing papers authored by Müge Kıray
This map shows the geographic impact of Müge Kıray's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Müge Kıray with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Müge Kıray more than expected).
Fields of papers citing papers by Müge Kıray
This network shows the impact of papers produced by Müge Kıray. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Müge Kıray. The network helps show where Müge Kıray may publish in the future.
Co-authorship network of co-authors of Müge Kıray
This figure shows the co-authorship network connecting the top 25 collaborators of Müge Kıray. A scholar is included among the top collaborators of Müge Kıray based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Müge Kıray. Müge Kıray is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 2 | |
| 3 | 4 | |
| 4 | 4 | |
| 5 | 15 | |
| 6 | 19 | |
| 7 | 4 | |
| 8 | Effects of inhaled L-arginine administration in a murine model of acute asthma. | 3 |
| 9 | 1 | |
| 10 | 32 | |
| 11 | 60 | |
| 12 | 23 | |
| 13 | 5 | |
| 14 | 15 | |
| 15 | 1 | |
| 16 | 43 | |
| 17 | 20 | |
| 18 | 40 | |
| 19 | 30 | |
| 20 | 154 |
About Müge Kıray
Müge Kıray is a scholar working on Behavioral Neuroscience, Developmental Neuroscience and Biophysics, having authored 72 papers that have together received 1.5k indexed citations. Recurring topics across this work include Stress Responses and Cortisol (10 papers), Adipose Tissue and Metabolism (8 papers) and Cardiac Ischemia and Reperfusion (8 papers). The work is most often cited by research in Behavioral Neuroscience (146 citations), Developmental Neuroscience (164 citations) and Biological Psychiatry (57 citations). Müge Kıray has collaborated with scholars based in Türkiye, United States and Malaysia. Frequent co-authors include Nazan Uysal, İlkay Aksu, Başak Baykara, Alper Bağrıyanık, Çetin Pekçetin, Bekir Uğur Ergür, Caner Çetinkaya, Ali Rıza Şişman, Osman Yılmaz and Kazım Tuğyan. Their work appears in journals such as SHILAP Revista de lepidopterología, Behavioural Brain Research and Urology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.