Masakazu Ibi

2.6k total citations
46 papers, 2.2k citations indexed

About

Masakazu Ibi is a scholar working on Physiology, Immunology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Masakazu Ibi has authored 46 papers receiving a total of 2.2k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Physiology, 17 papers in Immunology and 16 papers in Cellular and Molecular Neuroscience. Recurrent topics in Masakazu Ibi's work include Nitric Oxide and Endothelin Effects (15 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (14 papers) and Neuroscience and Neuropharmacology Research (7 papers). Masakazu Ibi is often cited by papers focused on Nitric Oxide and Endothelin Effects (15 papers), Neutrophil, Myeloperoxidase and Oxidative Mechanisms (14 papers) and Neuroscience and Neuropharmacology Research (7 papers). Masakazu Ibi collaborates with scholars based in Japan, United States and China. Masakazu Ibi's co-authors include Hideyuki Sawada, Akinori Akaike, Shun Shimohama, Takeshi Kihara, Chihiro Yabe‐Nishimura, Makoto Urushitani, Masato Katsuyama, K Iwata, Miki Nakanishi and Kuniharu Matsuno and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Neuroscience and Hepatology.

In The Last Decade

Masakazu Ibi

45 papers receiving 2.1k citations

Peers

Masakazu Ibi
Chandramohan Wakade United States
Yadong Goodman United States
Meliha Karsak Germany
Shigeru Nogawa Japan
Mohammad M. Khan United States
Soraya L. Vallés Spain
Luigi Formisano Italy
Chun‐Hsien Chu Taiwan
Silvia Giatti Italy
Evelyn Perez United States
Chandramohan Wakade United States
Masakazu Ibi
Citations per year, relative to Masakazu Ibi Masakazu Ibi (= 1×) peers Chandramohan Wakade

Countries citing papers authored by Masakazu Ibi

Since Specialization
Citations

This map shows the geographic impact of Masakazu Ibi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Masakazu Ibi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Masakazu Ibi more than expected).

Fields of papers citing papers by Masakazu Ibi

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Masakazu Ibi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Masakazu Ibi. The network helps show where Masakazu Ibi may publish in the future.

Co-authorship network of co-authors of Masakazu Ibi

This figure shows the co-authorship network connecting the top 25 collaborators of Masakazu Ibi. A scholar is included among the top collaborators of Masakazu Ibi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Masakazu Ibi. Masakazu Ibi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Asaoka, Nozomi, Masakazu Ibi, K Iwata, et al.. (2021). NOX1/NADPH Oxidase Promotes Synaptic Facilitation Induced by Repeated D2Receptor Stimulation: Involvement in Behavioral Repetition. Journal of Neuroscience. 41(12). 2780–2794. 9 indexed citations
2.
Yamada, Hiroyuki, Keita Yamamoto, Takeshi Sugimoto, et al.. (2020). Social Stress Increases Vulnerability to High-Fat Diet-Induced Insulin Resistance by Enhancing Neutrophil Elastase Activity in Adipose Tissue. Cells. 9(4). 996–996. 11 indexed citations
3.
Ibi, Masakazu, K Iwata, Misaki Matsumoto, et al.. (2020). NOX1/NADPH oxidase affects the development of autism-like behaviors in a maternal immune activation model. Biochemical and Biophysical Research Communications. 534. 59–66. 18 indexed citations
4.
Iwata, K, Xueqing Zhang, Kai Zhu, et al.. (2019). NOX1/NADPH oxidase regulates the expression of multidrug resistance‐associated protein 1 and maintains intracellular glutathione levels. FEBS Journal. 286(4). 678–687. 12 indexed citations
5.
Liu, Junjie, K Iwata, Kai Zhu, et al.. (2019). NOX1/NADPH oxidase in bone marrow-derived cells modulates intestinal barrier function. Free Radical Biology and Medicine. 147. 90–101. 20 indexed citations
6.
Katsuyama, Masato, Masakazu Ibi, K Iwata, Misaki Matsumoto, & Chihiro Yabe‐Nishimura. (2018). Clioquinol increases the expression of interleukin-8 by down-regulating GATA-2 and GATA-3. NeuroToxicology. 67. 296–304. 6 indexed citations
7.
Iwata, K, Kuniharu Matsuno, Kai Zhu, et al.. (2018). Up-regulation of NOX1/NADPH oxidase following drug-induced myocardial injury promotes cardiac dysfunction and fibrosis. Free Radical Biology and Medicine. 120. 277–288. 33 indexed citations
8.
Ibi, Masakazu, Junjie Liu, Noriaki Arakawa, et al.. (2017). Depressive-Like Behaviors Are Regulated by NOX1/NADPH Oxidase by Redox Modification of NMDA Receptor 1. Journal of Neuroscience. 37(15). 4200–4212. 52 indexed citations
9.
Matsumoto, Misaki, Jia Zhang, Xueqing Zhang, et al.. (2017). The NOX1 isoform of NADPH oxidase is involved in dysfunction of liver sinusoids in nonalcoholic fatty liver disease. Free Radical Biology and Medicine. 115. 412–420. 64 indexed citations
10.
Zhu, Kai, Tomoko Kakehi, Misaki Matsumoto, et al.. (2015). NADPH oxidase NOX1 is involved in activation of protein kinase C and premature senescence in early stage diabetic kidney. Free Radical Biology and Medicine. 83. 21–30. 67 indexed citations
11.
Katsuyama, Masato, et al.. (2014). Clioquinol Increases the Expression of VGF, a Neuropeptide Precursor, Through Induction of c-Fos Expression. Journal of Pharmacological Sciences. 124(4). 427–432. 6 indexed citations
12.
Matsuno, Kuniharu, K Iwata, Misaki Matsumoto, et al.. (2012). NOX1/NADPH oxidase is involved in endotoxin-induced cardiomyocyte apoptosis. Free Radical Biology and Medicine. 53(9). 1718–1728. 70 indexed citations
13.
Katsuyama, Masato, Tetsuo Kaneko, K Iwata, et al.. (2008). The AP-1 site is essential for the promoter activity of NOX1/NADPH oxidase, a vascular superoxide-producing enzyme: Possible involvement of the ERK1/2-JunB pathway. Biochemical and Biophysical Research Communications. 374(2). 351–355. 39 indexed citations
14.
Sawada, Hideyuki, Ryuichi Kohno, Takeshi Kihara, et al.. (2004). Proteasome Mediates Dopaminergic Neuronal Degeneration, and Its Inhibition Causes α-Synuclein Inclusions. Journal of Biological Chemistry. 279(11). 10710–10719. 96 indexed citations
15.
Sawada, Hideyuki, Masakazu Ibi, Takeshi Kihara, et al.. (2002). Estradiol protects dopaminergic neurons in a MPP+Parkinson’s disease model. Neuropharmacology. 42(8). 1056–1064. 100 indexed citations
16.
Katsuki, Hiroshi, Hisashi Shirakawa, Seiichiro Shimazu, et al.. (2001). Superoxide dismutase activity in organotypic midbrain–striatum co‐cultures is associated with resistance of dopaminergic neurons to excitotoxicity. Journal of Neurochemistry. 76(5). 1336–1345. 22 indexed citations
17.
Ibi, Masakazu, Hideyuki Sawada, Miki Nakanishi, et al.. (2001). Protective effects of 1α,25-(OH)2D3 against the neurotoxicity of glutamate and reactive oxygen species in mesencephalic culture. Neuropharmacology. 40(6). 761–771. 195 indexed citations
18.
Ibi, Masakazu, Hideyuki Sawada, Hiroshi Katsuki, et al.. (1999). Depletion of Intracellular Glutathione Increases Susceptibility to Nitric Oxide in Mesencephalic Dopaminergic Neurons. Journal of Neurochemistry. 73(4). 1696–1703. 47 indexed citations
19.
Sawada, Hideyuki, Masakazu Ibi, Takeshi Kihara, et al.. (1998). Dopamine D2‐type agonists protect mesencephalic neurons from glutamate neurotoxicity: Mechanisms of neuroprotective treatment against oxidative stress. Annals of Neurology. 44(1). 110–119. 161 indexed citations
20.
Urushitani, Makoto, Shun Shimohama, Takeshi Kihara, et al.. (1998). Mechanism of selective motor neuronal death after exposure of spinal cord to glutamate: Involvement of glutamate‐induced nitric oxide in motor neuron toxicity and nonmotor neuron protection. Annals of Neurology. 44(5). 796–807. 78 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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