Mikiro Nawa

867 citations

23 papers · 714 indexed · h-index 15

Aging top 10%
- Genetics, Aging, and Longevity in Model Organisms 2
Developmental Neuroscience top 10%
Rheumatology top 5%
- GDF15 and Related Biomarkers 10
Physiology top 10%
- Alzheimer's disease research and treatments 12
Cellular and Molecular Neuroscience top 10%
- Nuclear Receptors and Signaling 7
- Neuroscience and Neuropharmacology Research 2
Molecular Biology
- TGF-β signaling in diseases 3
- PI3K/AKT/mTOR signaling in cancer 2
Neurology
- Amyotrophic Lateral Sclerosis Research 3

Co-authors: Sadakazu Aiso Masaaki Matsuoka Yuichi Hashimoto Ikuo Nishimoto Kohsuke Kanekura Takako Niikura Tomohiro Chiba Kenzo Terashita
Cited by: Aging Developmental Neuroscience Rheumatology
Journals: Journal of Biological Chemistry (2 papers)Journal of Clinical Investigation (1 paper)Journal of Neuroscience (1 paper)
Partner nations: Japan United States

In The Last Decade

Mikiro Nawa

22 papers receiving 704 citations

Peers

Countries citing papers authored by Mikiro Nawa

Since Specialization

Citations

This map shows the geographic impact of Mikiro Nawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mikiro Nawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mikiro Nawa more than expected).

Fields of papers citing papers by Mikiro Nawa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mikiro Nawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mikiro Nawa. The network helps show where Mikiro Nawa may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Mikiro Nawa, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Mikiro Nawa Line = papers co-authored together Mikiro Nawa links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

#	Work
1	Excessive expression of progranulin leads to neurotoxicity rather than neuroprotection Neurobiology of Disease ·Shinya Kusakari,Hiroaki Suzuki,Mikiro Nawa,Katsuko Sudo,Rio Yamazaki,Tamami Miyagi,Tomoko Ohara,Masaaki Matsuoka,Kohsuke Kanekura	2025	0
2	Restoration of the reduced CLSP activity alleviates memory impairment in Alzheimer disease Translational Psychiatry ·Yuichi Hashimoto,Shinya Kusakari,Mikiro Nawa,Koichi Okamoto,Yuka Toyama,Masaaki Matsuoka	2021	1
3	Calmodulin‐like skin protein suppresses the increase in senescence‐associated β‐galactosidase induced by hydrogen peroxide or ultraviolet irradiation in keratinocytes Cell Biology International ·Yusuke Takahara,Nobuyuki Miyachi,Mikiro Nawa,Masaaki Matsuoka	2019	2
4	An Alzheimer Disease-linked Rare Mutation Potentiates Netrin Receptor Uncoordinated-5C-induced Signaling That Merges with Amyloid β Precursor Protein Signaling Journal of Biological Chemistry ·Yuichi Hashimoto,Yuka Toyama,Shinya Kusakari,Mikiro Nawa,Masaaki Matsuoka	2016	19
5	KCTD20, a relative of BTBD10, is a positive regulator of Akt BMC Biochemistry ·Mikiro Nawa,Masaaki Matsuoka	2013	12
6	Secreted calmodulin-like skin protein inhibits neuronal death in cell-based Alzheimer’s disease models via the heterotrimeric Humanin receptor Cell Death and Disease ·Yuuri Hashimoto,Mikiro Nawa,Megumi Kurita,Mutsutomo Tokizawa,Akihiro Iwamatsu,Masaaki Matsuoka	2013	22
7	SH3-binding Protein 5 Mediates the Neuroprotective Effect of the Secreted Bioactive Peptide Humanin by Inhibiting c-Jun NH2-terminal Kinase Journal of Biological Chemistry ·Yuji Takeshita,Yuichi Hashimoto,Mikiro Nawa,Hiroyuki Uchino,Masaaki Matsuoka	2013	19
8	Reduced expression of BTBD10, an Akt activator, leads to motor neuron death Cell Death and Differentiation ·Mikiro Nawa,Eriko Kage‐Nakadai,Sadakazu Aiso,Koichi Okamoto,Shohei Mitani,Masaaki Matsuoka	2012	25
9	The Method of the Body Bending Assay Using Caenorhabditis elegans BIO-PROTOCOL ·Mikiro Nawa,Masaaki Matsuoka	2012	14
10	Transforming Growth Factor β2 Level is Elevated in Neurons of Alzheimer's Disease Brains International Journal of Neuroscience ·Atsushi Noguchi,Mikiro Nawa,Sadakazu Aiso,Koichi Okamoto,Masaaki Matsuoka	2010	14
11	TAG-1 is an inhibitor of TGFβ2-induced neuronal death via amyloid β precursor protein Biochemical and Biophysical Research Communications ·Nobuyuki Tachi,Yuichi Hashimoto,Mikiro Nawa,Masaaki Matsuoka	2010	12
12	Abnormal expression of TIP30 and arrested nucleocytoplasmic transport within oligodendrocyte precursor cells in multiple sclerosis Journal of Clinical Investigation ·Jin Nakahara,Kohsuke Kanekura,Mikiro Nawa,Sadakazu Aiso,Norihiro Suzuki	2008	84
13	A novel Akt/PKB-interacting protein promotes cell adhesion and inhibits familial amyotrophic lateral sclerosis-linked mutant SOD1-induced neuronal death via inhibition of PP2A-mediated dephosphorylation of Akt/PKB Cellular Signalling ·Mikiro Nawa,Kohsuke Kanekura,Yuichi Hashimoto,Sadakazu Aiso,Masaaki Matsuoka	2007	30
14	Transforming growth factor β2 autocrinally mediates neuronal cell death induced by amyloid‐β Journal of Neuroscience Research ·Yuichi Hashimoto,Mikiro Nawa,Tomohiro Chiba,Sadakazu Aiso,Ikuo Nishimoto,Masaaki Matsuoka	2006	23
15	A humanin derivative, S14G‐HN, prevents amyloid‐β‐induced memory impairment in mice Journal of Neuroscience Research ·Hirohisa Tajima,Masaoki Kawasumi,Tomohiro Chiba,Marina Yamada,Kaoru Yamashita,Mikiro Nawa,Yoshiko Kita,Keisuke Kouyama,Sadakazu Aiso,Masaaki Matsuoka,Takako Niikura,Ikuo Nishimoto	2005	84
16	Implanted cannula-mediated repetitive administration of Aβ25–35 into the mouse cerebral ventricle effectively impairs spatial working memory Behavioural Brain Research ·Marina Yamada,Tomohiro Chiba,Jumpei Sasabe,Mikiro Nawa,Hirohisa Tajima,Takako Niikura,Kenzo Terashita,Sadakazu Aiso,Yoshiko Kita,Masaaki Matsuoka,Ikuo Nishimoto	2005	51
17	A tripartite motif protein TRIM11 binds and destabilizes Humanin, a neuroprotective peptide against Alzheimer's disease‐relevant insults European Journal of Neuroscience ·Takako Niikura,Yuichi Hashimoto,Hirohisa Tajima,Miho Ishizaka,Yohichi Yamagishi,Masaoki Kawasumi,Mikiro Nawa,Kenzo Terashita,Sadakazu Aiso,Ikuo Nishimoto	2003	100
18	Analysis of Neurons Created from Wild-Type and Alzheimer's Mutation Knock-In Embryonic Stem Cells by a Highly Efficient Differentiation Protocol Journal of Neuroscience ·Yoichiro Abe,Keisuke Kouyama,Taisuke Tomita,Yusuke Tomita,Norimitsu Ban,Mikiro Nawa,Masaaki Matsuoka,Takako Niikura,Sadakazu Aiso,Yoshiko Kita,Takeshi Iwatsubo,Ikuo Nishimoto	2003	15
19	Two serine residues distinctly regulate the rescue function of Humanin, an inhibiting factor of Alzheimer's disease‐related neurotoxicity: functional potentiation by isomerization and dimerization Journal of Neurochemistry ·Kenzo Terashita,Yuichi Hashimoto,Takako Niikura,Hirohisa Tajima,Yohichi Yamagishi,Miho Ishizaka,Masaoki Kawasumi,Tomohiro Chiba,Kohsuke Kanekura,Marina Yamada,Mikiro Nawa,Yoshiko Kita,Sadakazu Aiso,Ikuo Nishimoto	2003	66
20	The Cytoplasmic Domain of Alzheimer’s Amyloid-β Protein Precursor Causes Sustained Apoptosis Signal-Regulating Kinase 1/c-Jun NH2-Terminal Kinase-Mediated Neurotoxic Signal via Dimerization Journal of Pharmacology and Experimental Therapeutics ·Yuichi Hashimoto,Takako Niikura,Tomohiro Chiba,Emi Tsukamoto,Hisae Kadowaki,Hideki Nishitoh,Yohichi Yamagishi,Miho Ishizaka,Marina Yamada,Mikiro Nawa,Kenzo Terashita,Sadakazu Aiso,Hidenori Ichijo,Ikuo Nishimoto	2003	65

About Mikiro Nawa

Mikiro Nawa is a scholar working on Aging, Rheumatology and Physiology, having authored 23 papers that have together received 714 indexed citations. Recurring topics across this work include Alzheimer's disease research and treatments (12 papers), GDF15 and Related Biomarkers (10 papers), Nuclear Receptors and Signaling (7 papers), TGF-β signaling in diseases (3 papers), Amyotrophic Lateral Sclerosis Research (3 papers), Neuroscience and Neuropharmacology Research (2 papers), Genetics, Aging, and Longevity in Model Organisms (2 papers) and PI3K/AKT/mTOR signaling in cancer (2 papers). The work is most often cited by research in Aging (26 citations), Developmental Neuroscience (60 citations) and Rheumatology (213 citations). Mikiro Nawa has collaborated with scholars based in Japan and United States. Frequent co-authors include Sadakazu Aiso, Masaaki Matsuoka, Yuichi Hashimoto, Ikuo Nishimoto, Kohsuke Kanekura, Takako Niikura, Tomohiro Chiba, Kenzo Terashita, Hirohisa Tajima and Marina Yamada. Their work appears in journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and Journal of Neuroscience.

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