Michihito Wada
- Nephrology top 0.5%
- Genetics top 10%
- Nutrition and Dietetics top 5%
- Pathology and Forensic Medicine top 5%
- Molecular Biology
- Co-authors
- Nobuo NaganoEdward F. NemethJohn FoxTakehisa KawataYoshihiro FURUYAHiromi IshiiDavid P. MartinEdward Shatzen
- Topics
- Parathyroid Disorders and Treatments (20 papers)Genetic Syndromes and Imprinting (10 papers)Magnesium in Health and Disease (9 papers)
- Partner nations
- JapanUnited StatesSingapore
In The Last Decade
Michihito Wada
24 papers receiving 956 citations
Peers
Comparison fields: 5 of 64
- Nephrology 860
- Genetics 304
- Nutrition and Dietetics 295
- Pathology and Forensic Medicine 237
- Molecular Biology 192
Countries citing papers authored by Michihito Wada
This map shows the geographic impact of Michihito Wada's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michihito Wada with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michihito Wada more than expected).
Fields of papers citing papers by Michihito Wada
This network shows the impact of papers produced by Michihito Wada. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michihito Wada. The network helps show where Michihito Wada may publish in the future.
Co-authorship network of co-authors of Michihito Wada
This figure shows the co-authorship network connecting the top 25 collaborators of Michihito Wada. A scholar is included among the top collaborators of Michihito Wada based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michihito Wada. Michihito Wada is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 6 | |
| 2 | 7 | |
| 3 | 6 | |
| 4 | 7 | |
| 5 | 47 | |
| 6 | 14 | |
| 7 | 3 | |
| 8 | 197 | |
| 9 | 85 | |
| 10 | 3 | |
| 11 | 32 | |
| 12 | 26 | |
| 13 | 13 | |
| 14 | [Effects of intermittent treatment with sevelamer hydrochloride on parathyroid hyperplasia and vascular calcification in rats with chronic kidney disease]. | 5 |
| 15 | 130 | |
| 16 | 30 | |
| 17 | 17 | |
| 18 | 105 | |
| 19 | 80 | |
| 20 | 120 |
About Michihito Wada
Michihito Wada is a scholar working on Nephrology, Nutrition and Dietetics and Genetics, having authored 24 papers that have together received 987 indexed citations. Recurring topics across this work include Parathyroid Disorders and Treatments (20 papers), Genetic Syndromes and Imprinting (10 papers) and Magnesium in Health and Disease (9 papers). The work is most often cited by research in Nephrology (860 citations), Nutrition and Dietetics (295 citations) and Pathology and Forensic Medicine (237 citations). Michihito Wada has collaborated with scholars based in Japan, United States and Singapore. Frequent co-authors include Nobuo Nagano, Edward F. Nemeth, John Fox, Takehisa Kawata, Yoshihiro FURUYA, Hiromi Ishii, David P. Martin, Edward Shatzen, David L. Lacey and Catherine Stehman‐Breen. Their work appears in journals such as PLoS ONE, Kidney International and Journal of the American Society of Nephrology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.