Itaru Urakawa

5.1k total citations · 1 hit paper
22 papers, 3.6k citations indexed

About

Itaru Urakawa is a scholar working on Molecular Biology, Nephrology and Genetics. According to data from OpenAlex, Itaru Urakawa has authored 22 papers receiving a total of 3.6k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 13 papers in Nephrology and 9 papers in Genetics. Recurrent topics in Itaru Urakawa's work include Parathyroid Disorders and Treatments (12 papers), Genetic Syndromes and Imprinting (8 papers) and Fibroblast Growth Factor Research (7 papers). Itaru Urakawa is often cited by papers focused on Parathyroid Disorders and Treatments (12 papers), Genetic Syndromes and Imprinting (8 papers) and Fibroblast Growth Factor Research (7 papers). Itaru Urakawa collaborates with scholars based in Japan, Singapore and United States. Itaru Urakawa's co-authors include Takashi Shimada, Seiji Fukumoto, Takeyoshi Yamashita, Toshiro Fujita, Hisashi Hasegawa, Katsuya Okawa, Y. Yamazaki, Yuji Yamazaki, Takashi Yoneya and Yasuhiro Takeuchi and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and The Journal of Cell Biology.

In The Last Decade

Itaru Urakawa

21 papers receiving 3.6k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Klotho converts canonical FGF receptor into a specific receptor for FGF23

2006 1.4k citations Itaru Urakawa, Yuji Yamazaki et al. Nature profile →

Peers

Countries citing papers authored by Itaru Urakawa

Since Specialization

Citations

This map shows the geographic impact of Itaru Urakawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Itaru Urakawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Itaru Urakawa more than expected).

Fields of papers citing papers by Itaru Urakawa

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Itaru Urakawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Itaru Urakawa. The network helps show where Itaru Urakawa may publish in the future.

Co-authorship network of co-authors of Itaru Urakawa

This figure shows the co-authorship network connecting the top 25 collaborators of Itaru Urakawa. A scholar is included among the top collaborators of Itaru Urakawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Itaru Urakawa. Itaru Urakawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Inflammation-associated molecules in the glomerular-endothelium in mild IgA-nephropathy patients identified by single-cell and spatial transcriptome 2025 ·Communications Biology ·(unknown), Nagako Kawashima, (unknown), (unknown), (unknown), Takashi Sano, Itaru Urakawa, Masahiro Matsubara, (unknown)	1
2	Effects of NRF2 polymorphisms on safety and efficacy of bardoxolone methyl: subanalysis of TSUBAKI study 2023 ·Clinical and Experimental Nephrology ·(unknown), Takafumi Suzuki, Satsuki Muto, (unknown), Kengo Yamawaki, (unknown), Itaru Urakawa, Yuichi Aoki, Akihito Otsuki, Fumiki Katsuoka, Kengo Kinoshita, Masaomi Nangaku, Tadao Akizawa, Masayuki Yamamoto	0
3	Single-cell RNA sequencing identifies Fgf23-expressing osteocytes in response to 1,25-dihydroxyvitamin D3 treatment 2023 ·Frontiers in Physiology ·(unknown), (unknown), (unknown), Kazuhiro Masuda, Itaru Urakawa, Masahiro Abe, Y. Yamazaki, Seiji Fukumoto	6
4	Spatial and single-cell transcriptome analysis reveals changes in gene expression in response to drug perturbation in rat kidney 2022 ·DNA Research ·(unknown), (unknown), (unknown), (unknown), Itaru Urakawa, Masahide Seki, Junko Zenkoh, Ayako Suzuki, Yutaka Suzuki	14
5	Robust CD8+ T-cell proliferation and diversification after mogamulizumab in patients with adult T-cell leukemia-lymphoma 2020 ·Blood Advances ·(unknown), Toshihiko Ishii, Itaru Urakawa, (unknown), Ayako Masaki, Asahi Ito, Shigeru Kusumoto, Susumu Suzuki, Takeshi Takahashi, Akimichi Morita, Hiroshi Inagaki, Shinsuke Iida, Takashi Ishida	16
6	Mogamulizumab Treatment Elicits Autoantibodies Attacking the Skin in Patients with Adult T-Cell Leukemia-Lymphoma 2019 ·Clinical Cancer Research ·(unknown), (unknown), Toshihiko Ishii, Itaru Urakawa, (unknown), Ayako Masaki, Asahi Ito, Shigeru Kusumoto, Susumu Suzuki, (unknown), Takeshi Takahashi, Akimichi Morita, Hiroshi Inagaki, Shinsuke Iida, Takashi Ishida	20
7	Direct evidence for a causative role of FGF23 in the abnormal renal phosphate handling and vitamin D metabolism in rats with early-stage chronic kidney disease 2010 ·Kidney International ·Hisashi Hasegawa, Nobuo Nagano, Itaru Urakawa, Y. Yamazaki, (unknown), Toshiro Fujita, Takeyoshi Yamashita, Seiji Fukumoto, Takashi Shimada	251
8	Relevant use of Klotho in FGF19 subfamily signaling system in vivo 2010 ·Proceedings of the National Academy of Sciences ·Kenichi Tomiyama, Ryota Maeda, Itaru Urakawa, Yuji Yamazaki, Tomohiro Tanaka, Shinji Ito, Yoko Nabeshima, Tsutomu Tomita, Shinji Odori, Kiminori Hosoda, Kazuwa Nakao, Akihiro Imura, Yo‐ichi Nabeshima	124
9	Therapeutic Effects of Anti-FGF23 Antibodies in Hypophosphatemic Rickets/Osteomalacia 2009 ·Journal of Bone and Mineral Research ·(unknown), Y. Yamazaki, (unknown), Takehisa Kawata, Hisashi Hasegawa, Itaru Urakawa, Toshiro Fujita, Michihito Wada, Takeyoshi Yamashita, Seiji Fukumoto, Takashi Shimada	197
10	Circulating Fibroblast Growth Factor 23 in Patients with End-Stage Renal Disease Treated by Peritoneal Dialysis Is Intact and Biologically Active 2009 ·The Journal of Clinical Endocrinology & Metabolism ·Takashi Shimada, Itaru Urakawa, Tamara Isakova, Y. Yamazaki, Michael F. Epstein, Katherine Wesseling‐Perry, Myles Wolf, Isidro B. Salusky, Harald Jüppner	184
11	Anti-FGF23 Neutralizing Antibodies Show the Physiological Role and Structural Features of FGF23 2008 ·Journal of Bone and Mineral Research ·Y. Yamazaki, Taro Tamada, Noriyuki Kasai, Itaru Urakawa, (unknown), Hisashi Hasegawa, Toshiro Fujita, Ryota Kuroki, Takeyoshi Yamashita, Seiji Fukumoto, Takashi Shimada	155
12	Klotho converts canonical FGF receptor into a specific receptor for FGF23 breakdown → 2006 ·Nature ·Itaru Urakawa, Yuji Yamazaki, Takashi Shimada, (unknown), Hisashi Hasegawa, Katsuya Okawa, Toshiro Fujita, Seiji Fukumoto, Takeyoshi Yamashita	1435
13	Klotho is an essential molecule responsible for tissue specific FGF23 signaling 2006 ·Bone ·Itaru Urakawa, Y. Yamazaki, Takashi Shimada, (unknown), (unknown), Toshiro Fujita, Seiji Fukumoto, Takefumi Yamashita	1
14	FGF23 induces expression of two isoforms of NAB2, which are corepressors of Egr-1 2006 ·Biochemical and Biophysical Research Communications ·Toru Fukuda, Kazuhiro Kanomata, Junya Nojima, Itaru Urakawa, Tetsuo Suzawa, (unknown), Akiko Kukita, Ryutaro Kamijo, Takeyoshi Yamashita, Takenobu Katagiri	11
15	Vitamin D receptor-independent FGF23 actions in regulating phosphate and vitamin D metabolism 2005 ·American Journal of Physiology-Renal Physiology ·Takashi Shimada, Y. Yamazaki, M Takahashi, Hisashi Hasegawa, Itaru Urakawa, Takeshi Oshima, Kaori Ono, Makoto Kakitani, Kazuma Tomizuka, Toshiro Fujita, Seiji Fukumoto, Takeyoshi Yamashita	273
16	FGF-23 transgenic mice demonstrate hypophosphatemic rickets with reduced expression of sodium phosphate cotransporter type IIa 2004 ·Biochemical and Biophysical Research Communications ·Takashi Shimada, Itaru Urakawa, Y. Yamazaki, Hisashi Hasegawa, (unknown), Takashi Yoneya, Yasuhiro Takeuchi, Toshiro Fujita, Seiji Fukumoto, Takeyoshi Yamashita	340
17	Mutant FGF-23 Responsible for Autosomal Dominant Hypophosphatemic Rickets Is Resistant to Proteolytic Cleavage and Causes Hypophosphatemia in Vivo 2002 ·Endocrinology ·Takashi Shimada, Takanori Muto, Itaru Urakawa, Takashi Yoneya, Y. Yamazaki, Katsuya Okawa, Yasuhiro Takeuchi, Toshiro Fujita, Seiji Fukumoto, Takeyoshi Yamashita	336
18	Direct interaction of insulin-like growth factor-1 receptor with leukemia-associated RhoGEF 2001 ·The Journal of Cell Biology ·Shinichiro Taya, Naoyuki Inagaki, (unknown), Hiroshi Makino, Akihiro Iwamatsu, Itaru Urakawa, Kenji Nagao, Shiro Kataoka, Kozo Kaibuchi	92
19	Molecular Characterization of the Mouse mtprd Gene, a Homologue of Human TPRD: Unique Gene Expression Suggesting Its Critical Role in the Pathophysiology of Down Syndrome 1998 ·The Journal of Biochemistry ·Fujiko Tsukahara, Itaru Urakawa, Masahira Hattori, Masami Yokota Hirai, Ken‐ichi Ohba, Takao Yoshioka, Yoshiyuki Sakaki, Takamura Muraki	19
20	Bacillus subtilis TmrB protein confers resistance to tunicamycin on Escherichia coli. 1996 ·The Journal of General and Applied Microbiology ·Yoichi Noda, Itaru Urakawa, Koji Yoda, Makari Yamasaki	2

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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