Michele M. Johnson

888 citations
5 papers · 678 indexed · 1 hit paper · h-index 4

Michele M. Johnson

5 papers receiving 664 citations

Hit Papers

Fas Preassociation Required for Apoptosis Signaling and D...5042000202620082017100200300400500

Peers

Michele M. Johnson
Comparison fields: 5 of 70
  • Immunology 395
  • Cancer Research 87
  • Molecular Biology 393
  • Immunology and Allergy 29
  • Pathology and Forensic Medicine 77
Replace Gemma Texidó with:
Gemma Texidó Italy
Kathrin Kläsener Germany
Claude Tschopp Switzerland
Xiaochuan Shan United States
Soula Ganiatsas United States
Shefali Shah United States
Frédérique Brockly France
Tove Irene Klokk Norway
Revati J. Tatake United States
Gregory B. Carey United States
Michele M. Johnson relative to Gemma Texidó Italy Gemma Texidó's profile →
Citations per field
00.5×1.5×
Gemma Texidó · 1×
Citations per year

Countries citing papers authored by Michele M. Johnson

Since Specialization
Citations

This map shows the geographic impact of Michele M. Johnson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michele M. Johnson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michele M. Johnson more than expected).

Fields of papers citing papers by Michele M. Johnson

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michele M. Johnson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michele M. Johnson. The network helps show where Michele M. Johnson may publish in the future.

Co-authorship network

The 25 scholars most cited alongside Michele M. Johnson, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Michele M. Johnson Line = papers co-authored together Michele M. Johnson links everyone, so they are left out of the graph.

All Works

5 of 5 papers shown
#Work
1 20023
2 200118
3 200193
4
Fas Preassociation Required for Apoptosis Signaling and Dominant Inhibition by Pathogenic Mutationsbreakdown →
2000504
5 199960

About Michele M. Johnson

Michele M. Johnson is a scholar working on Physiology, Immunology and Oncology, having authored 5 papers that have together received 678 indexed citations. Recurring topics across this work include T-cell and B-cell Immunology (2 papers), Immunotherapy and Immune Responses (2 papers), Cell death mechanisms and regulation (1 paper), Myasthenia Gravis and Thymoma (1 paper), Viral-associated cancers and disorders (1 paper), CAR-T cell therapy research (1 paper), Antifungal resistance and susceptibility (1 paper) and Monoclonal and Polyclonal Antibodies Research (1 paper). The work is most often cited by research in Immunology (395 citations), Cancer Research (87 citations) and Molecular Biology (393 citations). Michele M. Johnson has collaborated with scholars based in United States, Switzerland and Slovenia. Frequent co-authors include Michael J. Lenardo, Richard M. Siegel, David A. Zacharias, David H. Lynch, John K. Frederiksen, Francis Ka-Ming Chan, Roger Y. Tsien, Jennifer M. Puck, Jack Bleesing and Margaret R. Brown. Their work appears in journals such as Science, Blood and The Journal of Immunology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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