Francis Ka-Ming Chan
- Molecular Biology top 0.5%
- Immunology top 0.2%
- Epidemiology top 2%
- Oncology top 2%
- Cancer Research top 1%
- Co-authors
- Kenta MoriwakiDavid MoquinMichael J. LenardoSreerupa ChallaRichard M. SiegelMelissa GuildfordThomas J. McQuadeTathagat Dutta Ray
- Topics
- Cell death mechanisms and regulation (44 papers)Inflammasome and immune disorders (19 papers)interferon and immune responses (18 papers)
- Partner nations
- United StatesBrazilChina
In The Last Decade
Francis Ka-Ming Chan
68 papers receiving 10.9k citations
Hit Papers
Peers
Comparison fields: 5 of 140
- Molecular Biology 7.5k
- Immunology 5.3k
- Epidemiology 1.5k
- Oncology 1.5k
- Cancer Research 1.4k
Countries citing papers authored by Francis Ka-Ming Chan
This map shows the geographic impact of Francis Ka-Ming Chan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Francis Ka-Ming Chan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Francis Ka-Ming Chan more than expected).
Fields of papers citing papers by Francis Ka-Ming Chan
This network shows the impact of papers produced by Francis Ka-Ming Chan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Francis Ka-Ming Chan. The network helps show where Francis Ka-Ming Chan may publish in the future.
Co-authorship network of co-authors of Francis Ka-Ming Chan
This figure shows the co-authorship network connecting the top 25 collaborators of Francis Ka-Ming Chan. A scholar is included among the top collaborators of Francis Ka-Ming Chan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Francis Ka-Ming Chan. Francis Ka-Ming Chan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 11 | |
| 2 | 74 | |
| 3 | 29 | |
| 4 | 150 | |
| 5 | 166 | |
| 6 | 46 | |
| 7 | 55 | |
| 8 | 129 | |
| 9 | 288 | |
| 10 | 263 | |
| 11 | 78 | |
| 12 | 8 | |
| 13 | 75 | |
| 14 | 121 | |
| 15 | 1 | |
| 16 | Phosphorylation-Driven Assembly of the RIP1-RIP3 Complex Regulates Programmed Necrosis and Virus-Induced Inflammationbreakdown → | 1915 |
| 17 | 14 | |
| 18 | 9 | |
| 19 | 10 | |
| 20 | 124 |
About Francis Ka-Ming Chan
Francis Ka-Ming Chan is a scholar working on Immunology, Cancer Research and Biophysics, having authored 69 papers that have together received 11.1k indexed citations. Recurring topics across this work include Cell death mechanisms and regulation (44 papers), Inflammasome and immune disorders (19 papers) and interferon and immune responses (18 papers). The work is most often cited by research in Immunology (5.3k citations), Molecular Biology (7.5k citations) and Cancer Research (1.4k citations). Francis Ka-Ming Chan has collaborated with scholars based in United States, Brazil and China. Frequent co-authors include Kenta Moriwaki, David Moquin, Michael J. Lenardo, Sreerupa Challa, Richard M. Siegel, Melissa Guildford, Thomas J. McQuade, Tathagat Dutta Ray, Ryan M. Genga and Lixin Zheng. Their work appears in journals such as Nature, Science and Cell.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.