Michael Orth

3.8k total citations
36 papers, 1.6k citations indexed

About

Michael Orth is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Neurology. According to data from OpenAlex, Michael Orth has authored 36 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Cellular and Molecular Neuroscience, 20 papers in Molecular Biology and 17 papers in Neurology. Recurrent topics in Michael Orth's work include Genetic Neurodegenerative Diseases (24 papers), Neurological disorders and treatments (16 papers) and Mitochondrial Function and Pathology (14 papers). Michael Orth is often cited by papers focused on Genetic Neurodegenerative Diseases (24 papers), Neurological disorders and treatments (16 papers) and Mitochondrial Function and Pathology (14 papers). Michael Orth collaborates with scholars based in Germany, United Kingdom and United States. Michael Orth's co-authors include Anthony H.V. Schapira, G. Bernhard Landwehrmeyer, Sarah J. Tabrizi, Carsten Saft, James F. Gusella, Jong‐Min Lee, Peter Holmans, Marcy E. MacDonald, Lesley Jones and Hugh Rickards and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Neurology and The American Journal of Human Genetics.

In The Last Decade

Michael Orth

36 papers receiving 1.5k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Michael Orth Germany 23 972 857 632 160 143 36 1.6k
Jenny Sassone Italy 25 991 1.0× 1.0k 1.2× 554 0.9× 253 1.6× 117 0.8× 46 1.7k
Patrícia de Carvalho Aguiar Brazil 18 573 0.6× 397 0.5× 871 1.4× 90 0.6× 133 0.9× 51 1.4k
Albert C. Ludolph Germany 17 634 0.7× 538 0.6× 780 1.2× 232 1.4× 199 1.4× 23 1.4k
Hung‐Li Wang Taiwan 30 1.3k 1.3× 1.4k 1.6× 498 0.8× 368 2.3× 229 1.6× 63 2.2k
Alessandro Ferraris Italy 21 593 0.6× 422 0.5× 595 0.9× 150 0.9× 184 1.3× 46 1.4k
Ilaria Barone Italy 19 560 0.6× 481 0.6× 271 0.4× 145 0.9× 118 0.8× 28 1.1k
Johanne Egge Rinholm Norway 12 512 0.5× 763 0.9× 134 0.2× 222 1.4× 258 1.8× 17 1.3k
Patricia L. Kramer United States 20 1.3k 1.4× 483 0.6× 1.6k 2.5× 220 1.4× 264 1.8× 31 2.3k
Giovanna Levandis Italy 21 510 0.5× 261 0.3× 643 1.0× 196 1.2× 217 1.5× 28 1.2k
Jonathan E. Kurz United States 16 754 0.8× 462 0.5× 151 0.2× 133 0.8× 120 0.8× 26 1.2k

Countries citing papers authored by Michael Orth

Since Specialization
Citations

This map shows the geographic impact of Michael Orth's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Michael Orth with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Michael Orth more than expected).

Fields of papers citing papers by Michael Orth

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Michael Orth. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Michael Orth. The network helps show where Michael Orth may publish in the future.

Co-authorship network of co-authors of Michael Orth

This figure shows the co-authorship network connecting the top 25 collaborators of Michael Orth. A scholar is included among the top collaborators of Michael Orth based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Michael Orth. Michael Orth is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Wang, Yiqin, Xiaoxian Guo, Kaixiong Ye, Michael Orth, & Zhenglong Gu. (2021). Accelerated expansion of pathogenic mitochondrial DNA heteroplasmies in Huntington’s disease. Proceedings of the National Academy of Sciences. 118(30). 30 indexed citations
2.
Chao, Michael J., Kyung‐Hee Kim, Diane Lucente, et al.. (2018). Population-specific genetic modification of Huntington's disease in Venezuela. PLoS Genetics. 14(5). e1007274–e1007274. 34 indexed citations
3.
Shin, Aram, Tammy Gillis, Jayalakshmi Srinidhi Mysore, et al.. (2016). The HTT CAG-Expansion Mutation Determines Age at Death but Not Disease Duration in Huntington Disease. The American Journal of Human Genetics. 98(2). 287–298. 103 indexed citations
4.
Kojer, Kerstin, et al.. (2016). Mitochondrial cristae remodelling is associated with disrupted OPA1 oligomerisation in the Huntington's disease R6/2 fragment model. Experimental Neurology. 288. 167–175. 27 indexed citations
5.
Demestre, Maria, Michael Orth, Karl J. Föhr, et al.. (2015). Formation and characterisation of neuromuscular junctions between hiPSC derived motoneurons and myotubes. Stem Cell Research. 15(2). 328–336. 72 indexed citations
6.
Duijn, Erik van, David Craufurd, Anna A.M. Hubers, et al.. (2014). Neuropsychiatric symptoms in a European Huntington's disease cohort (REGISTRY). Journal of Neurology Neurosurgery & Psychiatry. 85(12). 1411–1418. 205 indexed citations
7.
Saft, Carsten, et al.. (2013). Neues zur Huntington-Krankheit. Aktuelle Neurologie. 40(7). 377–392. 1 indexed citations
8.
Orth, Michael, et al.. (2012). L04 Assessing dysphagia in Huntington's disease using Fiberoptic Endoscopic Evaluation of Swallowing (FEES). Journal of Neurology Neurosurgery & Psychiatry. 83(Suppl 1). A44.2–A44. 2 indexed citations
9.
Wolf, Robert Christian, Fabio Sambataro, Nenad Vasić, et al.. (2012). Default-mode network changes in preclinical Huntington's disease. Experimental Neurology. 237(1). 191–198. 57 indexed citations
10.
Orth, Michael & Carsten Schwenke. (2011). Age-at-onset in Huntington disease. PLoS Currents. 3. RRN1258–RRN1258. 22 indexed citations
11.
López-Sendón, José Luis, Ana Royuela, Michael Orth, et al.. (2011). What is the impact of education on Huntington's disease?. Movement Disorders. 26(8). 1489–1495. 33 indexed citations
12.
Wolf, Robert Christian, Georg Grön, Fabio Sambataro, et al.. (2011). Brain activation and functional connectivity in premanifest Huntington's disease during states of intrinsic and phasic alertness. Human Brain Mapping. 33(9). 2161–2173. 43 indexed citations
13.
Wild, Edward J., Anna Magnusson, Nayana Lahiri, et al.. (2011). Abnormal peripheral chemokine profile in Huntington’s disease. PLoS Currents. 3. RRN1231–RRN1231. 94 indexed citations
14.
Orth, Michael, Sven Schippling, Sabine Schneider, et al.. (2009). Abnormal motor cortex plasticity in premanifest and very early manifest Huntington disease. Journal of Neurology Neurosurgery & Psychiatry. 81(3). 267–270. 72 indexed citations
15.
Amann, Benedikt L., et al.. (2009). Treatment-Refractory Schizoaffective Disorder in a Patient with Dyke-Davidoff-Masson Syndrome. CNS Spectrums. 14(1). 36–40. 19 indexed citations
16.
Stern, Jeremy S., Michael Orth, & Mary M. Robertson. (2009). Gilles de la Tourette syndrome in pregnancy: a retrospective series. Obstetric Medicine. 2(3). 128–129. 6 indexed citations
17.
Orth, Michael. (2004). G209A mutant alpha synuclein expression specifically enhances dopamine induced oxidative damage. Neurochemistry International. 45(5). 669–676. 25 indexed citations
18.
Orth, Michael, Sarah J. Tabrizi, Anthony H.V. Schapira, & Jonathan M. Cooper. (2003). α-Synuclein expression in HEK293 cells enhances the mitochondrial sensitivity to rotenone. Neuroscience Letters. 351(1). 29–32. 41 indexed citations
19.
Orth, Michael & Rustam R. Mundegar. (2002). Effect of acid maltase deficiency on the endosomal/lysosomal system and glucose transporter 4. Neuromuscular Disorders. 13(1). 49–54. 10 indexed citations
20.
Orth, Michael & Anthony H.V. Schapira. (2001). Mitochondria and degenerative disorders. American Journal of Medical Genetics. 106(1). 27–36. 228 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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